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In situ monitoring of pore structure of magnesium oxysulfate cement paste: Effect of MgSO4/H2O ratio
Shengwen Tang,Changrong Wei,Rongjin Cai,Jiasheng Huang,E. Chen,Junhui Yuan 한국공업화학회 2020 Journal of Industrial and Engineering Chemistry Vol.83 No.-
This work systematically presented the pore structure evolution of different magnesium oxysulfate(MOS) cement pastes by non-contact impedance measurement (NCIM), fractal theory and othermicrostructural experiments. According to the evolution of electrical impedance response, threehydration stages (dissolution, acceleration and hardened stages) can be identified in the pastes. Furthermore, based on the results obtained from NCIM and fractal theory, the porosity, incremental porevolume, pore tortuosity, average pore diameter and maximal pore diameter of MOS cement pastes areinvestigated. The influence of MgSO4/H2O ratio on pore structure of MOS cement paste is primarilyexplained. It can be inferred from predicted and experimental results that NCIM is a good method toanalyze the microstructure evolution of MOS cement pastes in-situ.
Zhao Yongjie,Shang Shengwen,Song Ya,Li Tianyue,Han Mingliang,Qin Yuexuan,Wei Meili,Xi Jun,Tang Bikui 한국미생물학회 2022 The journal of microbiology Vol.60 No.11
Mycobacterium tuberculosis (M. tuberculosis) is a highly pathogenic intracellular pathogen that causes tuberculosis (TB), the leading cause of mortality from single infections. Redox homeostasis plays a very important role in the resistance of M. tuberculosis to antibiotic damage and various environmental stresses. The antioxidant sulforaphane (SFN) has been reported to exhibit anticancer activity and inhibit the growth of a variety of bacteria and fungi. Nonetheless, it remains unclear whether SFN exhibits anti-mycobacterial activity. Our results showed that the SFN against M. tuberculosis H37Ra exhibited bactericidal activity in a time and dose-dependent manner. The anti-tubercular activity of SFN was significantly correlated with bacterial reactive oxygen species (ROS) levels. In addition, SFN promoted the bactericidal effect of macrophages on intracellular bacteria in a dose-dependent manner, mediated by increasing intracellular mitochondrial ROS levels and decreasing cytoplasmic ROS levels. Taken together, our data revealed the previously unrecognized antimicrobial functions of SFN. Future studies focusing on the mechanism of SFN in macrophages against M. tuberculosis are essential for developing new host-directed therapeutic approaches against TB.