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鄭世珍 ( Jung¸ Sejin ) 중국어문학회 2021 中國語文學誌 Vol.- No.76
‘Shipu(食譜)’ describes the system and history, various ingredients and recipes of the food. ‘Shipu’ has been compiled in earnest from the Song Dynasty. In this paper, 『Shanjiaqinggong(山家淸供)』, the most important source of information of the Southern Song Dynasty, was studied. In addition, a basic exploration of the book was conducted to examine the old recipes and food culture of China. 『Shanjiaqinggong』 was written by Lin Hong(林洪) lived in the Southern Song Dynasty and various editions of this book remain. 『Shanjiaqinggong』 has the following characteristics: First, this book has characteristics and value as a culinary manuscripts. This is because there are the diversity of food ingredients, the specificity and variety of cooking methods, the connection between meals and curing, traces of the Southern Song Dynasty vegetarian culture, and traces of the Southern Song Dynasty royal cuisine. Second, it connects food with literature by quoting literary works related to food. Third, it reflects local people and food culture of the Fujian(福建). In short, 『Shanjiaqinggong』 is a culinary manuscripts that must be studied for the study of old food and a material that should be used for the study of social culture in the Southern Song Dynasty.
양회염운사(兩淮鹽運使) 노견증(盧見曾)의 문화 활동 연구
鄭世珍 ( Jung¸ Sejin ) 한국중국산문학회 2020 중국산문연구집간 Vol.10 No.-
Yangzhou(揚州) in the 18th century was a rich and mature cultural space. The salt merchant invested unselfishly in social and cultural fields, making Yangzhou a cultural center in Jiangnan(江南). During Qianlong, Lu Jianceng(1690-1768) was a competent official. When he was working in Yangzhou, he planned the cultural activities: first, he renovated Yangzhou's famous places. Second, he organized poetry societies with a large number of people to encourage literary creation. Third, he linked with money-needed scholars and salt merchants who had a huge collection of books to encourage academic activities. In Yangzhou, he planned a cultural event represented by poetic creation and academic activities, and gathered a large number of people, by holding a large-scale Red Bridge Poetry Society(紅橋修禊詩會) attended by more than 7,000 people, as well as by serving as a medium for linking scholars and salt merchants. He became the leader of Yangzhou's cultural space. Although it is a cultural activity involving many levels of interests, it is undeniable that Yangzhou became a cultural center in the 18th century because of Lu Jianceng's character.
Jung, In-Hyuk,Choi, Jae-Hoon,Jin, Jing,Jeong, Se-Jin,Jeon, Sejin,Lim, Chaeji,Lee, Mi-Ran,Yoo, Ji-Young,Sonn, Seong-Keun,Kim, Young Ho,Choi, Beom Kyu,Kwon, Byoung S.,Seoh, Ju-Young,Lee, Cheol Whan,Kim, The Federation of American Societies for Experimen 2014 The FASEB Journal Vol.28 No.11
<P>CD137 (4-1BB), a member of the tumor necrosis factor receptor superfamily, has been reported to be expressed in atherosclerotic plaques, and to promote lesion formation. However, the role of CD137 in mediating atherosclerotic plaque stability and the possible underlying molecular and cellular mechanisms are poorly understood. Here, apolipoprotein E-deficient (<I>ApoE</I><SUP>−/−</SUP>) and CD137-deficient <I>ApoE</I><SUP>−/−</SUP> (<I>ApoE</I><SUP>−/−</SUP>CD137<SUP>−/−</SUP>) mice fed a chow diet for 66 wk were used. CD137 induces plaque instability, which is characterized by increased plaque necrosis, decreased collagen content, decreased vascular smooth muscle cell (VSMC) content, and increased macrophage infiltration. CD137 also increases the infiltration of effector T (T<SUB>eff</SUB>) cells into plaque lesion sites, resulting in increased interferon-γ (IFN-γ) expression. Interestingly, T<SUB>eff</SUB>-cell-derived IFN-γ inhibits collagen synthesis in atherosclerotic plaques. Furthermore, CD137 activation increases the apoptosis of VSMCs, possibly by decreasing the antiapoptotic regulator, Bcl-2, and subsequently up-regulating cleaved caspase-3. In macrophages, activation of CD137 signaling boosted the oxidized low density lipoprotein-induced expression of matrix metalloproteinase 9 <I>via</I> the p38 mitogen-activated protein kinase and extracellular signal-regulated kinase1/2 signaling pathways. In summary, activation of CD137 signaling decreases the stability of advanced atherosclerotic plaques <I>via</I> its combined effects on T<SUB>eff</SUB> cells, VSMCs, and macrophages.—Jung, I.-H., Choi, J.-H., Jin, J., Jeong, S.-J., Jeon, S., Lim, C., Lee, M.-R., Yoo, J.-Y., Sonn, S.-K., Kim, Y. H., Choi, B. K., Kwon, B. S., Seoh, J.-Y., Lee, C. W., Kim, D.-Y., Oh, G. T. CD137-inducing factors from T cells and macrophages accelerate the destabilization of atherosclerotic plaques in hyperlipidemic mice.</P>