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RISS 인기검색어
- 검색키워드
- 저자 : Ren Xiaorui (검색결과 2 건)
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Xiaorui Ren,Huanhuan Li,Ke Liu,Hongyi Lu,Jingshuai Yang,Ronghuan He 한국섬유공학회 2018 Fibers and polymers Vol.19 No.12
Poly(vinylpyrrolidone) (PVP), as the low-cost and commercial material, exhibits superior phosphoric acid doping capability due to the presence of heterocycle and carbonyl groups in the repeat unit. However, it can’t be used as the high temperature polymer electrolyte membrane (HT-PEM) alone because of its significant hydrophilicity and poor mechanical stability. In the present work, polyethersulfone (PES), polysulfone (PSU), polyetherketone-cardo (PEK-c), polyvinylidene fluoride (PVDF) and poly(vinylidene fluoride-co-hexafluoropropylene) (PHFP), five kinds of engineering thermoplastics with excellent mechanical properties and chemical inertness, are chosen to prepare a series of PVP blend membranes by the polymer blending method in order to enhance the dimensional and mechanical stabilities of PVP based membranes. The influence of structures of enhanced polymers on properties of HT-PEMs was investigated systematically. PVP blend membranes with aromatic polymers (i.e. PES, PSU and PEK-c) exhibited decreased volume swellings, increased acid doping contents, superior conductivities and improved mechanical strengths, which determined that they are more suitable for electrolytes of fuel cell applications comparing with PVP/PVDF and PVP/PHFP membranes blended with aliphatic polymers.
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Guo Junjie,Liang Tianzeng,Chen Huifu,Li Xiangen,Ren Xiaorui,Wang Xiuying,Xiao Kan,Zhao Jiangchao,Zhu Huiling,Liu Yulan 아세아·태평양축산학회 2022 Animal Bioscience Vol.35 No.8
Objective: The purpose of this study was to evaluate the protection of glutamate (GLU) against the impairment in intestinal barrier function induced by lipopolysaccharide (LPS) stress in weaned pigs. Methods: Twenty-four weaned pigs were divided into four treatments containing: i) nonchallenged control, ii) LPS-challenged control, iii) LPS+1.0% GLU, and iv) LPS+2.0% GLU. On day 28, pigs were treated with LPS or saline. Blood samples were collected at 0, 2, and 4 h post-injection. After blood samples collection at 4 h, all pigs were slaughtered, and spleen, mesenteric lymph nodes, liver and intestinal samples were obtained. Results: Dietary GLU supplementation inhibited the LPS-induced oxidative stress in pigs, as demonstrated by reduced malondialdehyde level and increased glutathione level in jejunum. Diets supplemented with GLU enhanced villus height, villus height/crypt depth and claudin-1 expression, attenuated intestinal histology and ultrastructure impairment induced by LPS. Moreover, GLU supplementation reversed intestinal intraepithelial lymphocyte number decrease and mast cell number increase induced by LPS stress. GLU reduced serum cortisol concentration at 4 h after LPS stress and downregulated the mRNA expression of intestinal corticotropin-releasing factor signal (corticotrophin-releasing factor [CRF], CRF receptor 1 [CRFR1], glucocorticoid receptor, tryptase, nerve growth factor, tyrosine kinase receptor A), and prevented mast cell activation. GLU upregulated the mRNA expression of intestinal transforming growth factor β. Conclusion: These findings indicate that GLU attenuates LPS-induced intestinal mucosal barrier injury, which is associated with modulating CRF signaling pathway. Objective: The purpose of this study was to evaluate the protection of glutamate (GLU) against the impairment in intestinal barrier function induced by lipopolysaccharide (LPS) stress in weaned pigs.Methods: Twenty-four weaned pigs were divided into four treatments containing: i) non-challenged control, ii) LPS-challenged control, iii) LPS+1.0% GLU, and iv) LPS+2.0% GLU. On day 28, pigs were treated with LPS or saline. Blood samples were collected at 0, 2, and 4 h post-injection. After blood samples collection at 4 h, all pigs were slaughtered, and spleen, mesenteric lymph nodes, liver and intestinal samples were obtained.Results: Dietary GLU supplementation inhibited the LPS-induced oxidative stress in pigs, as demonstrated by reduced malondialdehyde level and increased glutathione level in jejunum. Diets supplemented with GLU enhanced villus height, villus height/crypt depth and claudin-1 expression, attenuated intestinal histology and ultrastructure impairment induced by LPS. Moreover, GLU supplementation reversed intestinal intraepithelial lymphocyte number decrease and mast cell number increase induced by LPS stress. GLU reduced serum cortisol concentration at 4 h after LPS stress and downregulated the mRNA expression of intestinal corticotropin-releasing factor signal (corticotrophin-releasing factor [<i>CRF</i>], CRF receptor 1 [<i>CRFR1</i>], glucocorticoid receptor, tryptase, nerve growth factor, tyrosine kinase receptor A), and prevented mast cell activation. GLU upregulated the mRNA expression of intestinal transforming growth factor β.Conclusion: These findings indicate that GLU attenuates LPS-induced intestinal mucosal barrier injury, which is associated with modulating CRF signaling pathway.
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