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Nan Xu,Wei-Min Liu,Ming Zhang,Xiaobo Wang 한국공업화학회 2017 Journal of Industrial and Engineering Chemistry Vol.46 No.-
The high-temperature tribological performance of lithium grease is enhanced by addition of Lewis acidborate ester, which could interact with Lewis base RCO2of lithium 12-hydroxystearate (LHS, constitutethe thickenerfiber network of lithium grease) to form a Lewis acid–base complex. Important detailsabout the Lewis acid–base complex and its reinforcing effect on the tribological performance areelucidated by means of SRV oscillating friction and wear tester (SRV), Fourier transformation infraredspectroscopy (FTIR), TGA–DSC, and rheological methods. The experimental results strongly suggest thatthere is a Lewis base–acid interaction between the boron atom of borate ester or boric acid and theoxygen atom of RCO2. Lewis acid centers can serve as second-level linking points to reinforce thestrength of the thickenerfiber network and further improve the colloidal stability of lubricating grease. Compared with pure LHS, the Lewis acid–base complex displays higher thermal stability, allowinglithium grease to be applied to higher temperature. Because of the enhancement of thermal stability andcolloidal stability,film-forming property under high temperature can be greatly improved, resulting in>15% friction and >95% wear reductions.
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Nan Xu,Pengcheng Kan,Xiuhua Yao,Ping Yang,Jiwei Wang,Lei Xiang,Yu Zhu 한국미생물학회 2018 The journal of microbiology Vol.56 No.11
Acute ischaemic stroke (AIS) seriously affects patient quality of life. We explored the role of the intestinal microbiota on oxidative stress and autophagy in stroke, and Astragaloside IV (AS-IV) reversed the changes induced by intestinal microbiota. We determined the characteristics of the intestinal microbiota of AIS and transient ischaemic attack (TIA) patients by 16S sequencing and found that the structure and diversity of the intestinal microbiota in patients with AIS and TIA were significantly different from those in healthy subjects. Specifically, the abundance of genus Bifidobacterium, Megamonas, Blautia, Holdemanella, and Clostridium, content of homocysteine and triglyceride was increased significantly, thus it may be as a potential mechanism of AIS and TIA. Furthermore, germ-free mice were infused intracolonically with fecal supernatants of TIA and AIS with/without feed AS-IV for 12 weeks, and we found that the feces of AIS up-regulated the autophagy markers Beclin-1, light chain 3 (LC3)-II and autophagy-related gene (Atg)12, and the expression of reactive oxygen species (ROS) and NADPH oxidase 2/4 (NOX2/4), malondialdehyde (MDA), however, the expression of total antioxidant capacity (T-AOC) and activity of superoxide dismutase (SOD) and glutathione (GSH) was down-regulated in brain tissue, the content of homocysteine and free fatty acids (FFA) in serum of the mice. Meanwhile, AS-IV could reverse the above phenomenon, however, it does not affect the motor function of mice. AS-IV reversed these changes and it may be a potential drug for AIS therapeutics.
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Nan Xu,Yuzhui Xu,Boshuo Zhang,Qining Song,Jianhua Zhao,Yefeng Bao 대한금속·재료학회 2023 METALS AND MATERIALS International Vol.29 No.11
Double-pass friction-stir processing (FSP) with different probe lengths and coolants was conducted on a 3 mm thick Cu–30%Zn alloy plate, and a heterogeneous lamella structure (HLS) was produced along the plate thickness direction. TheHLS can be divided into three layers with their volume ratio was approximately 1:1:1, i.e., ultra-fine grain layer, fine grainlayer, and coarsened grain layer which produced by cold source assisted FSP, conventional FSP, and annealing treatment,respectively. The average hardness in ultra-fine grain layer was about 123 HV, which was significantly higher than those ofcoarsened grain layer (94 HV) and fine grain layer (105 HV). In each layer, the hardness slightly decreased with distanceincreased from the surface, but the hardness value decreased sharply in the transition zones of different layers. The HLSCu–30%Zn alloy showed yield strength of 220 MPa, ultimate tensile strength of 462 MPa, and fracture elongation of 30%. The strength-ductility synergy was attributed to the back-stress strengthening between the different layers, which induceda remarkable strain hardening at the initial stage of plastic deformation. This study provided a possible way for preparingHLS copper alloys with relatively good mechanical properties, and the HLS copper alloys have an application potential inthe manufacture of bearing, propeller, and condenser.
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Yao, Xuerui,Jiang, Hao,NanXu, Yong,Piao, Xuanjing,Gao, Qingshan,Kim, Nam-Hyung Butterworths, etc 2019 Theriogenology Vol. No.
<P><B>Abstract</B></P> <P>Kaempferol (3,4′,5,7-tetrahydroxyflavone, KAE) is one of the most commonly occurring dietary flavonols. The biological and pharmacological effects of kaempferol depend upon whether it acts as an antioxidant, anti-inflammatory, or anticancer agent. The present study explored the influence of KAE supplementation on <I>in vitro</I> damage to porcine oocytes and its underlying mechanisms. Different concentrations of KAE (0.05, 0.1, 0.5, 1 μM) were added to porcine zygote medium 5 during <I>in vitro</I> culture. The results showed that supplementation with 0.1 μM KAE significantly increased the blastocyst formation rate. Blastocyst formation and quality were significantly increased in the 200 μM H<SUB>2</SUB>O<SUB>2</SUB> treatment group following addition of 0.1 μM KAE. KAE prevented the H<SUB>2</SUB>O<SUB>2</SUB>-induced compromise of mitochondrial membrane potential and reactive oxygen species generation. Furthermore, the extent of autophagy and DNA damage in the blastocysts was attenuated by supplementation with KAE in the H<SUB>2</SUB>O<SUB>2</SUB> induced oxidative injury group compared to that observed in controls. These results suggest that KAE has beneficial effects on the development of porcine parthenotes by attenuating oxidative stress and increasing mitochondrial function.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Kaempferol has beneficial effects on the development of porcine early stage embryos. </LI> <LI> Kaempferol prevented the H<SUB>2</SUB>O<SUB>2</SUB>-induced production of ROS. </LI> <LI> Kaempferol prevented mitochondrial dysfunction induced by H<SUB>2</SUB>O<SUB>2</SUB> in porcine embryos. </LI> <LI> Kaempferol prevented H<SUB>2</SUB>O<SUB>2</SUB>-induced autophagy and DNA damage. </LI> </UL> </P>
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