http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
오늘 본 자료
On the Capacity Gain from Full Duplex Communications in a Large Scale Wireless Network
Wang, Xudong,Huang, Huaiyu,Hwang, Taewon IEEE Computer Society 2016 IEEE TRANSACTIONS ON MOBILE COMPUTING Vol.15 No.9
<P>Compared to half duplex communications, full duplex communications can significantly improve link capacity. However, in a large scale wireless network such as a wireless mesh network, the capacity gain from full duplex communications has not been fully investigated. To this end, a metric of network capacity called transmission capacity is studied in this paper for a full duplex wireless network. It captures the maximum transmission throughput in a unit area, subject to a certain outage probability. The key challenge of deriving transmission capacity is to characterize the aggregate interference of the typical link in a full duplex wireless network, which is completely different from that in a half duplex wireless network. In this paper, stochastic geometry is employed to model the network topology as a Thomas cluster point process and then the aggregate interference is characterized as a shot-noise process. Based on these models, the transmission capacity is derived. Analytical results show that under the same network density the distribution of aggregate interference in a full duplex wireless network is more dispersed than that in a half duplex wireless network. Comparisons of transmission capacity between a full duplex network and a half duplex network reveal that the capacity gain from full duplex communications is limited due to severe aggregate interference. This result implies that self-interference cancellation alone cannot ensure scalable full duplex wireless networking.</P>
Huaiyu Wen,Yao Xiao,Yi Han,Yuqian Zhao,Shan Wang 한국정밀공학회 2023 International Journal of Precision Engineering and Vol.10 No.2
Wind turbine gear is an essential part of a wind turbine, and the induction hardening is an important treatment method for its surface strengthening. However, when scanning induction heating, the end position is the weak part of the process. In this paper, the dynamic evolution process of the end temperature field was studied during the scanning induction heating of wind turbine gear. It was found that a significant hysteresis emerge in the temperature field of the lateral tooth profile before the scanning heating reaches the steady state stage. The reason lies in the poor heat transfer caused by the gear end structure, and the combined effect of induced eddy current heating in the region. The hysteresis distance of the temperature field at the end, is positively correlated with the inductor scanning speed within a certain range. The material parameters of gear are affected by the change of temperature, which in turn affects the heating process. The induced magnetic field on the two tooth flanks has an obvious forward movement phenomenon under the combined action of the gear temperature and the magnetic flux concentrator. The steady-state temperature field forms a loop on both flanks. Increasing the movement speed within a certain range, a gap would appear at the lower boundary of the ring. The evolution process of the end temperature of the gear scanning induction heating is clarified in this paper, which would provide a research foundation for improving the end heating quality and precise control.
Zhiwei Zhang,Mengyu Fan,Qianqiao Wang,Huaiyu Li,Chunying Zhu,Youguang Ma,Taotao Fu 한국공업화학회 2022 Journal of Industrial and Engineering Chemistry Vol.110 No.-
This study focuses on the influence of the resultant force caused by the density difference of the twophases on the droplet generation process in a step-emulsification microfluidic device. Threedimensionalvisualization is used to observe the characteristics of droplet generation. According to thedifference in droplet generation form, it can be divided into drip generation mode and suspended generationmode. In the suspended generation mode, the droplets are more inclined to the formation process ofthe expansion of the ellipsoid, whose size in the horizontal direction is larger than the vertical direction;while in the drip generation mode, the shape of the droplet is opposite. The control stage of the dropletgeneration under various operating conditions is determined. As the viscosity of the dispersed phaseincreases, the control stage of the suspended generation mode is the necking stage, and the control stageof the drip generation mode changes from the first stage of three-dimensional expansion to the neckingstage. The model of the droplet generation is proposed by combining mechanical analysis and experimentaltesting data, and the droplet size prediction formula is thereby obtained.
TRIM56 Suppresses Multiple Myeloma Progression by Activating TLR3/TRIF Signaling
Ying Chen,Jing Zhao,Dengzhe Li,Jinxia Hao,Pengcheng He,Huaiyu Wang,Mei Zhang 연세대학교의과대학 2018 Yonsei medical journal Vol.59 No.1
Purpose: Tripartite-motif-containing protein 56 (TRIM56) has been found to exhibit a broad antiviral activity, depending upon E3 ligaseactivity. Here, we attempted to evaluate the function of TRIM56 in multiple myeloma (MM) and its underlying molecular basis. Materials and Methods: TRIM56 expression at the mRNA and protein level was measured by qRT PCR and western blot analysis. 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) and flow cytometry analysis was performed to investigate the effect of TRIM56 on MM cell proliferation and apoptosis. The concentrations of interferon (IFN)-β, interleukin (IL)-6, and tumor necrosis factor-α in MM cell culture supernatants were detected with respective commercial ELISA kits. Western blot was employedto determine the effect of TRIM56 on toll-like receptor 3 (TLR3)/toll-IL-1 receptor (TIR) domain-containing adaptor inducingIFN-β (TRIF) signaling pathway. Results: TRIM56 expression was prominently decreased in MM cells. Poly (dA:dT)-induced TRIM56 overexpression in U266 cells suppressed proliferation, induced apoptosis, and enhanced inflammatory cytokine production, while TRIM56 knockdown improvedgrowth, diminished apoptosis, and inhibited inflammatory cytokine secretion in RPMI8226 cells. Moreover, TRIM56 knockdownblocked TLR3 signaling pathway. Furthermore, poly (I:C), a TLR3 agonist, markedly abolished TRIM56 depletion-induced increase of proliferation, decrease of apoptosis, and reduction of inflammatory factor in MM cells. Conclusion: TRIM56 may act as a tumor suppressor in MM through activation of TLR3/TRIF signaling pathway, contributing to a better understanding of the molecular mechanism of TRIM56 involvement in MM pathogenesis and providing a promising therapystrategy for patients with MM.