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Room-temperature power factor of CuAlO2 composite tablets enhanced by MWCNTs
Ma Lingxiao,Dong Chenhao,Li Wenquan,Su Qing,Zhou Jinyuan,Xie Erqing,Lan Wei 한국물리학회 2022 Current Applied Physics Vol.33 No.-
CuAlO2 with high theoretical thermoelectric performance has potential applications in thermal energy conversion. Herein, multi-wall carbon nanotubes (MWCNTs)/CuAlO2 composite tablets are prepared by using different amounts of MWCNTs and solid paraffin binder, where MWCNTs served as a conductive agent and rendered three orders of magnitude increase in electrical conductivity. Seebeck coefficient of the composites was reduced with increasing MWCNTs content. Consequently, an optimal room-temperature thermoelectric power factor (PF) of 1.31 μW m 1K 2 has been rendered by MWCNTs/CuAlO2 composite tablet with 1 wt % MWCNTs. Moreover, PF value increased with increasing temperature after a slight decrease at 333 K, which can be ascribed to the modulation of electrical conductivity. Current work provides an effective strategy to improve thermoelectric performance of CuAlO2 materials.
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Chen Hao,Zhang Dong-Ming,Zhang Zhi-Ping,Li Ming-Zhang,Wu Hai-Feng 한국유전학회 2021 Genes & Genomics Vol.43 No.12
Background Mitochondrial unfolded protein response plays an important role in the occurrence and development of breast cancer. However, the role of mitochondrial unfolded protein response (UPRmt) in the sensitivity of breast cancer to cisplatin chemotherapy has not yet been cleared. Objectives The purpose of this study is to explore the role of mitochondrial unfolded protein response in breast cancer sensitivity to cisplatin. Methods In this study, qRT-PCR, Western blotting, Immunofuorescence, CCK-8, Colony formation, Transwell assay and TUNEL staining assay were used to confrm the role of UPRmt in breast cancer cells treated with cisplatin. Results Cisplatin increased the levels of UPRmt including CLPP, HSP60, LONP1 in MCF7 and MDA-MB-231 cells. UPRmt inducer Nicotinamide ribose (NR) could promote the proliferation and invasion of breast cancer cells treated with cisplatin. Importantly, SIRT3 was discovered to increase UPRmt in breast cancer cells and silencing of SIRT3 could inhibit the efect of NR in breast cancer. Conclusions UPRmt regulated by SIRT3 could protect breast cancer cell from cisplatin. Controlling SIRT3-induced UPR may be a potential therapeutic target to increase the sensitivity of breast cancer chemotherapy.
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