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웹 서비스 기반 보안 토큰을 이용한 통합 인증 모델 설계
강철수(Chul-Su Kang),이상훈(Sang-Hun Lee),전문석(Moon-Seog Jun) 한국정보과학회 2004 한국정보과학회 학술발표논문집 Vol.31 No.2Ⅰ
웹 서비스는 최근 가장 주목받고 있는 기술 중 하나이다. 오늘날 웹 서비스는 간단한 데이터 공유에서 부터 대규모의 인터넷 판매 및 통화 교환, 애플리케이션 통합 시나리오의 범주까지 다양한 분야에서 전개 되고 있다. 또한 웹 서비스는 모바일, 디바이스, 그리드 시나리오 등에도 적용되고 있다. 현재 웹서비스는 이기종간에 구축된 서비스를 통합하고 호환시키는데 많은 중점을 두고 있다. 여기서 반드시 필요한 부분이 이기종간의 인증 부분이다. 서로 다른 시스템에 접근하기 위해서 사용자는 여러 번의 인증절차를 거쳐야 한다. 본고에서는 사용자의 인증절차를 통합하고, 접근 권한에 대한 제어를 위해 WS-Security의 표준안에 포함된 보안 토큰을 이용한 통합 인증 모델을 제시하고자 한다.
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소량의 Glycopyrrolate 와 Propantheline 이 심박동에 미치는 영향
임웅모,강철수,박형덕 대한마취과학회 1983 Korean Journal of Anesthesiology Vol.16 No.4
Glycopyrrolate and propantheline, being synthetic Quaternary ammonium compounds that cannot cross the blood-brain barrier, will not have the vagal center stimulation. The author administered each small dose of glycopyrrolate and propantenline intravenously to normal human volunteers, and compared its effect on the heart rate. The results were as follows ; 1) Glrcopyrrolate(0. 1 mg) produced a slight decrease on the heart rate, 2) Propsntheline (0.5 mg) produced no detectable change on the heart rate. 3) Glrcopyrrolate(0.1 mg) 10 min. after pretreatment with propsntheline(0.5 mg) produced a significant increase on the heart rate. 4) Propantheline(0.5 ng) 10 minutes after pretreatment with glycopyrrolate(0.1 mg) produced more significant increase on the heart rate than glrcopyrrolate after propantheline. 5) From the above results, it is suggested that the depressive effect of sympat.hetic gangl. ion by propantheline may be less than by glycopyrrolate.
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소량의 Glycopyrrolate(Robinul)이 마취 및 혼수환자의 심박수에 미치는 영향
김영호,임웅모,강철수 대한마취과학회 1982 Korean Journal of Anesthesiology Vol.15 No.4
Glycopyrrolate (Robinul) is a potent anticholinergic drug. Being a quantenary ammonium compound, it does not cross the blood-brain barrier and thus does not have the central effects. It has been shown that glycopyrrolate has a pharmacologic properties similar to that of atropine, belladonna alkaloid, but it has lesser effect on the heart rate than that of atropine. The authors administered a small does of glycopyrrolate intravenously to 18 people whowere awake, 21 comatous cases and 17 halothane anesthetized cases. The effect on the heart rate in these 3 groups was compared. The results are as follows; 1) In the awake state, glycopyrrolate (0.1 mg) produced a slight decrease in the heart rate. 2) In the comatose state, glycopyrrolate (0.1 mg) produced a slight increase in the heart rate. 3) In the halothane anesthetized state, glycopyrrolate (0.1 mg) produced a significant increase in the heart rate. 4) In the comatose state, the absence of bradycardia after a small dose of glycopyrrolate is similar to a small dose of atropine. Therefore we suggest that bradycardia after glycopyrrolate and atropine is caused by sympathetic ganglion block.
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Halothane-N2O-O2 마취중 Clonidine 이 Sodium Nitroprusside 의 혈역학 및 폐내 Shunt 에 미치는 영향
박찬진,정성수,임웅모,강철수 대한마취과학회 1989 Korean Journal of Anesthesiology Vol.22 No.2
Supplementation of antihypertensive action of sodium nitroprusside (SNP) is almost standard practice and should obviate the need for potentially toxic doses to control blood pressure. Clonidine, an antihypertensive agent known to reduce sympathetic outfiow via alpha2-adrenergic receptor stimulation, has been shown to decrease MAC of halogenated agent, and to reduce the amount of SNP required to reduce the desired hypotension. To determine the effects of clonidine on the hemodynamics and intrapulmonary shunting during SNP infusion, clonidine and/or SNP were administered to 22 patients anesthetized with halothane-N2, O-O2 (FIO2:0.5). In one group of 11 patients, clonidine 1.5μg/kg was injected intravenously. In another group of 11 patients, clonidine 1.5 μg/kg was injected intravenously 30 minutes after starting the SNP infusion (3 μg/kg/min). The results were as follows. 1) Clonidine alone produced a small decrease in MAP (10%) and CI (8%) but other hemodynamic values remained unaltered. 2) Arterial oxygen tion and intrapulmonary shunting was not changed by clonidine. 3) Heart rate (15%) was increased , but MAP (29%), MPAP (24%), PCWP (25%), CVP (32% ), SVR (29%)and PVR(24%) were decreased significantly, and CI, SVI remained unchanged during SNP hypotension. 4) SNP caused a significant increase in intrapulmonary shunt fraction from 8.62% to 10.58% and a decrease in PaO₂. 5) In group of clonidine under SNP infusion, conidine did not significantly affect the hemodynamic response to SNP except for 15% decrease in BP. 6) Clonidine caused no significant change on gas exchange effect of SNP. These results indicate that clonidine did not significantly affect the hemodynamics and intrapul-monary shunting during SNP hypotension. Therefore, clonidine could be used as a valuable adjuvant for reducing the amount of SNP and decreasing MAC of halothane.
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