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클래스 불균형 데이터에 적합한 기계 학습 기반 침입 탐지 시스템
정윤경(Yun-Gyung Cheong),박기남(Kinam Park),김현주(Hyunjoo Kim),김종현(Jonghyun Kim),현상원(Sangwon Hyun) 한국정보보호학회 2017 정보보호학회논문지 Vol.27 No.6
본 논문에서는 정상과 이상 트래픽이 불균형적으로 발생하는 상황에서 기계 학습 기반의 효과적인 침입 탐지 시스템에 관한 연구 결과를 소개한다. 훈련 데이터의 패턴을 학습하여 정상/이상 패킷을 탐지하는 기계 학습 기반의 IDS에서는 훈련 데이터의 클래스 불균형 정도에 따라 탐지 성능이 현저히 차이가 날 수 있으나, IDS 개발 시 이러한 문제에 대한 고려는 부족한 실정이다. 클래스 불균형 데이터가 발생하는 환경에서도 우수한 탐지 성능을 제공하는 기계 학습 알고리즘을 선정하기 위하여, 본 논문에서는 Kyoto 2006+ 데이터셋을 이용하여 정상 대 침입 클래스 비율이 서로 다른 클래스 불균형 훈련 데이터를 구축하고 다양한 기계 학습 알고리즘의 인식 성능을 분석하였다. 실험 결과, 대부분의 지도 학습 알고리즘이 좋은 성능을 보인 가운데, Random Forest 알고리즘이 다양한 실험환경에서 최고의 성능을 보였다. This paper aims to develop an IDS (Intrusion Detection System) that takes into account class imbalanced datasets. For this, we first built a set of training data sets from the Kyoto 2006+ dataset in which the amounts of normal data and abnormal (intrusion) data are not balanced. Then, we have run a number of tests to evaluate the effectiveness of machine learning techniques for detecting intrusions. Our evaluation results demonstrated that the Random Forest algorithm achieved the best performances.
항Ro(SS-A) 및 항La(SS-B)항체 양성인 전신성 경피증 환자에서 태어난 환아로 선천성 완전 심장차단을 가졌던 1례
정광호,남경재,이윤우,박용원 인제대학교 1994 仁濟醫學 Vol.15 No.4
선천성 완전 심장차단의 중요한 원인 중의 하나는 모체의 항 Ro(SSA) 항체가 태반을 통과하는 것이다. 26세의 항 Ro(SSA) 항체 및 항 La(SS-B) 항체가 양성인 전신성경피증 산모로부터 태어난 18개월된 여아에게서 선천성 완전 심장 차단을 관찰하였기에 보고하는 바이다. One of the important causes of congenital heart block is the transplacental transmission of maternal ants-Ro(SSA) antibody. An 18 month old girl was brought to our hospital because of dyspnea from which she had been suffered after birth. A complete heart block, atrial septal defect(secundum type) and patent ductus arteriosus were found. Closure of defects was done and permanent pace maker installed. She died of pneumonia and congestive heart failure at the age of 30 months. Her mother had this baby at the age of 26 years old and had suffered from systemic sclerosis since her early twenties. The laboratory studies on her revealed as follows. ANA 1 : 2650, anti-nRNP Ab (-), anti-Scl 70 Ab (-), anti-centromere Ab (-), rheumatoid factor (-), anti-Ro(SSA) Ab (+), anti-La(SSB) Ab (+), interstitial pulmonary fibrosis (DLCO : 50% of predicted value).
Kim, Seung Yun,Lee, Hyoung Jin,Park, Eujin,Ahn, Yo Han,Ha, Il-Soo,Cheong, Hae Il,Kang, Hee Gyung Korean Society of Pediatric Nephrology 2015 Childhood kidney diseases Vol.19 No.2
Rituximab (RTX), a monoclonal antibody against the B-cell marker CD20, is commonly used as a treatment for antibody-mediated diseases or B-lymphocyte-mediated diseases. Destruction of B cells may reverse the disease course in many conditions; however, patients who are treated with RTX cannot respond appropriately to de novo infection due to lack of B lymphocytes. Here, we report one such case. A 7-year-old renal allograft recipient presented with severe anemia due to parvovirus infection after RTX treatment. The patient had focal segmental glomerulosclerosis and had received cadaveric kidney transplantation 6 months previously. She was treated with high-dose steroid for acute rejection and RTX for Epstein Barr Virus infection 3 months previously. At presentation, her hemoglobin level was 5.4 g/dL and leukocyte and platelet counts were normal. She had microcytic normochromic anemia and high viral load of parvovirus B19(70,578 copies/mL). Intravenous immunoglobulin ($200mg/kg{\cdot}d$) treatment controlled the progression of anemia and parvovirus infection. De novo parvovirus infection during the B lymphocyte-depletion period may have precipitated the severe anemia in this case. Close monitoring of infection is required after RTX therapy.
A case of pseudohypoaldosteronism type 1 with a mutation in the mineralocorticoid receptor gene
Lee, Se-Eun,Jung, Yun-Hye,Han, Kyoung-Hee,Lee, Hyun-Kyung,Kang, Hee-Gyung,Ha, Il-Soo,Choi, Yong,Cheong, Hae-Il The Korean Pediatric Society 2011 Clinical and Experimental Pediatrics (CEP) Vol.54 No.2
Pseudohypoaldosteronism type 1 (PHA1) is a rare form of mineralocorticoid resistance characterized in newborns by salt wasting with dehydration, hyperkalemia and failure to thrive. This disease is heterogeneous in etiology and includes autosomal dominant PHA1 owing to mutations of the NR3C2 gene encoding the mineralocorticoid receptor, autosomal recessive PHA1 due to mutations of the epithelial sodium channel (ENaC) gene, and secondary PHA1 associated with urinary tract diseases. Amongst these diseases, autosomal dominant PHA1 shows has manifestations restricted to renal tubules including a mild salt loss during infancy and that shows a gradual improvement with advancing age. Here, we report a neonatal case of PHA1 with a NR3C2 gene mutation (a heterozygous c.2146_2147insG in exon 5), in which the patient showed failure to thrive, hyponatremia, hyperkalemia, and elevated plasma renin and aldosterone levels. This is the first case of pseudohypoaldosteronism type 1 confirmed by genetic analysis in Korea.