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Paik, In-Hwan,Jin, Seon-Deok,Jeon, Byung-Seon,Yu, Jae-Pyoung,Park, Chi-Young,Paek, Woon-Kee national science museum of korea 2010 Journal of Korean nature Vol.3 No.4
The Hangang (Han River) Ecological Learning Center is a river ecosystem park which was completed in December of 2002. The river ecosystem park was created based on the understanding of the problems of standard park-formed river which centers around convenient facilities instead of on the wish to accentuate the innate unique characteristics of a river. Therefore, the study was conducted in order to analyze the changes and habitation usage of the avifauna in the Hangang Ecological Learning Center to provide basic information for the improvement of bird species diversity in an ecosystem park. The study was conducted in July and October of both 2003 and 2005 to study the avian colonies of the same time period, and the habitat usage of four different areas were examined. Main dominant species includes species which preferred waterside and grassland, and 2005 showed higher levels of species, bird count and number of migratory birds. Furthermore, among the four regions used as habitats, the confluence region showed the highest level of increase, and this is predicted to have caused by the shallow depth of the confluence and the stability of the waterside plant colonies.
백서 적혈구에서 glutathione이 paraquat 독성에 미치는 영향
김명철,박재윤,채기영,천영욱,박평심,차종희 朝鮮大學校 附設 醫學硏究所 1991 The Medical Journal of Chosun University Vol.16 No.2
The participation of superoxide in initiating tissue damage from administration of a xenobiotics is best illustrated by paraquat intoxication, in which it is known that one electron reduction of paraquat leads to the formation of radicals which react with molecular oxygen to give superoxide. In the present study, the effects of paraquat on the level of glutathione and activities of superoxide dismutase(SOD), catalase, glutathione peroxidase, glutathione reductase and glutathione stransferase were investigated in rat liver and erythrocytes. The contents of glutathione in liver and blood were sinificantly decreased by paraquat administration, but the activities of enzymes were unaltered. Incubation of the buffered aerobic mixture of erythrocyte in the presence of 5mM glucose, Paraquat and/or N-ethylmaleimide(NEM) resulted in the formation of lipid peroxide, the activities of various enzyme and the levels of glutathione were determined. The malondialdehyde(MDA) contents as a indicator of lipid peroxidation was decreased and the levels of total glutathione were not changed but the levels of oxidized glutathione(GSSG) were in creased in paraquat treated erythrocytes. The activites of ezyme were decreased in paraquat treated erythrocytes but in reduced glutathione(GSH) treated erythrocyte, the enzymes activities were less decreased by paraquat. These results suggest that paraquat toxicity was probably somewhat reduced by GSH, but paraquat-induced injury were not increased by GSH depletion in erythrocytes.
( Pyoung-jae Park ),( Tae Wan Lim ),( Sae Byeol Choi ),( Wan Bae Kim ),( Sang Yong Choi ) 대한간학회 2016 춘·추계 학술대회 (KASL) Vol.2016 No.1
Purpose: Associating liver partition and portal vein ligation for staged hepatectomy (ALPPS) has recently been developed for patients with predicted insufficient future liver remnant volumes to induce more rapid hepatic hypertrophy and increase resectability. It has been usually performed for metastatic liver cancer from colorectal cancer, but few reports about ALPPS for hepatocellular carcinoma, especially in the liver cirrhosis combined portal hypertension were published. Especially, any treatment options of huge HCC under liver cirrhosis with portal hypertension were not proper. We reported a successful case of ALPPS for huge HCC combined with liver cirrhosis and portal hypertension. Methods: A 58-year-old female patient was admitted for abdominal pain for 3 months. She had a history of chronic hepatitis B, but it was not treated. On abdominal CT, about 20cm sized huge heterogeneously enhancing mass was identified and replaced to the right hepatic lobe. Right glisson and right hepatic vein were compressed and it invaded to middle hepatic vein and segment 4. Nodularity of liver surface, moderate splenomegaly and enlarged varices were identified. AFP level was severely increased to 158389 ng/mL and PIVKA-II level was over 100,000 mAU/mL. ICG 15(%) was checked to 48.2%. It was suggested to severe liver cirrhosis and inoperable state. The future remnant liver volume (LLS+S1) on CT volumetry was 306 mL (291+15). Severe post-hepatectomy liver failure was strongly expected and so ALPPS was planned. Results: During 1st stage operation, the partition between left lateral section and S4, right anterior portal vein ligation was performed. The partition plane was covered with Proceed mesh. The reason of right anterior portal vein ligation was that cental hepatectomy was preferred to right tri-sectionectomy if right tri-sectionectomy would make post-hepatectomy liver failure even though ALPPS was performed. Total bilirubin level was increased to 2.22 mg/dL but CT volume of left lateral section was increased to 387 mL at postoperative 12 days. 2nd stage operation was performed at 14 postoperative days. During 2nd stage operation, anatomical central hepatectomy was performed without sacrifice of the right posterior section with right posterior glisson and right hepatic vein. Total bilirubin level was increased to 5.13 mg/dL on new postoperative 1 day, but it was recovered to normal range on postoperative 12 days. CT remnant liver volume (SLLS+S1+RPS) was 589mL (395+16+178) at postoperative 8 days although biloma was identified at resection area. She recovered at postoperative 1 month. Conclusion: Although the validity and oncologic safety of ALPPS were not yet fully investigated, ALPPS for HCC under severe liver cirrhosis with portal hypertension was possible and more studies are needed to further evaluate its effectiveness and oncological outcomes. Figure 1. (A) preoperative CT shows that huge HCC (>20cm) compresses of right glisson; (B) CT between 1st stage and 2nd stage operation shows increased volume of left lateral section and the partition between left lateral section and S4 (blue arrow); (C) CT after 2nd stage operation shows succesful central hepatectomy. Figure 2. (A) Huge HCC underlying severe liver cirrhosis was identified during 1st operation, (B) 1st stage operation: the partition between left lateral section and S4, right anterior portal vein ligation The partition plane was covered with Proceed mesh, (C) 2nd stage operation: the anatomical central hepatectomy.
( Pyoung-jae Park ),( Tae Wan Lim ),( Sae Byeol Choi ),( Wan Bae Kim ),( Sang Yong Choi ) 대한간학회 2016 춘·추계 학술대회 (KASL) Vol.2016 No.1
Introduction: Associating liver partition and portal vein ligation for staged hepatectomy (ALPPS) has recently been developed for patients with predicted insufficient future liver remnant volumes to induce more rapid hepatic hypertrophy and increase resectability. It has been usually performed for metastatic liver cancer from colorectal cancer, but few reports about ALPPS for hepatocellular carcinoma, especially in the liver cirrhosis combined portal hypertension were published. Especially, any treatment options of huge HCC under liver cirrhosis with portal hypertension were not proper. We reported a successful case of ALPPS for huge HCC combined with liver cirrhosis and portal hypertension. Case report: A 58-year-old female patient was admitted for abdominal pain for 3 months. She had a history of chronic hepatitis B, but it was not treated. On abdominal CT, about 20 cm sized huge heterogeneously heterogeneously enhancing mass was identified and replaced to the right hepatic lobe. Right glisson and right hepatic vein were compressed and it invaded to middle hepatic vein and segment 4. Nodularity of liver surface, moderate splenomegaly and enlarged varices were identified. AFP level was severely increased to 158389 ng/mL and PIVKA-II level was over 100,000 mAU/mL. ICG 15(%) was checked to 48.2 %. It was suggested to severe liver cirrhosis and inoperable state. The future remnant liver volume(LLS+S1) on CT volumetry was 306 mL(291+15). Severe post-hepatectomy liver failure was strongly expected and so ALPPS was planned. During 1st stage operation, the partition between left lateral section and S4, right anterior portal vein ligation was performed. The partition plane was covered with Proceed mesh. The reason of right anterior portal vein ligation was that centalhepatectomy was preferred to right tri-sectionectomy if right tri-sectionectomy would make post-hepatectomy liver failure even though ALPPS was performed. Total bilirubin level was increased to 2.22 mg/dL but CT volume of left lateral section was increased to 387 mL at postoperative 12 days. 2nd stage operation was performed at 14 postoperative days. During 2nd stage operation, anatomical central hepatectomy was performed without sacrifice of the right posterior section with right posterior glisson and right hepatic vein. Total bilirubin level was increased to 5.13 mg/dL on new postoperative 1 day, but it was recovered to normal range on postoperative 12 days. CT remnant liver volume( SLLS+S1+RPS) was 589mL(395+16+178) at postoperative 8 days although biloma was identified at resection area. She recovered at postoperative 1 month. Conclusion: Although the validity and oncologic safety of ALPPS were not yet fully investigated, ALPPS for HCC under severe liver cirrhosis with portal hypertension was possible and more studies are needed to further evaluate its effectiveness and oncological outcomes.
( Pyoung Jae Park ),( Shin Hwang ),( Young Il Choi ),( Young Dong Yu ),( Gil Chun Park ),( Sung Won Jung ),( Sam Youl Yoon ),( Gi Won Song ),( Tae Yong Ha ),( Sung Gyu Lee ) 대한간학회 2012 Clinical and Molecular Hepatology(대한간학회지) Vol.18 No.4
Erythropoietic protoporphyria (EPP) is an inherited disorder of the heme metabolic pathway that is characterized by accumulation of protoporphyrin in the blood, erythrocytes, and tissues, and cutaneous manifestations of photosensitivity, all resulting from abnormalities in ferrochelatase (FECH) activity due to mutations in the FECH gene. Protoporphyrin is excreted by the liver, and excess protoporphyrin leads to cholelithiasis with obstructive episodes and chronic liver disease, finally progressing to liver cirrhosis. Patients with end-stage EPP-associated liver disease require liver transplantation. We describe here a 31-year-old male patient with EPP who experienced acute-on-chronic liver failure and underwent deceased-donor liver transplantation. Surgical and postoperative care included specific shielding from exposure to ultraviolet radiation to prevent photosensitivity-associated adverse effects. The patient recovered uneventfully and was doing well 24 months after transplantation. Future prevention and treatment of liver disease are discussed in detail. (Clin Mol Hepatol 2012;18:411-415)