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증례 / 단백상실성 장병증 후에 발병된 Reflex Sympathetic Dystrophy Syndrome 1예
최원(Won Choi),박원(Won Park),이경주(Kyoung Ju Yi),김범수(Pum Soo Kim),송정수(Jeong Soo Song),장재남(Jae Nam Chang),이돈행(Don Haeng Lee),김형길(Hyung Gil Kim),권계숙(Kye Sook Kwon),조현근(Hueon Geun Cho),신용운(Yong Woon Shin),김영수 대한내과학회 1999 대한내과학회지 Vol.56 No.1
A 16-year old girl was admitted for chronic diarrhea, generalized edema, severe pain of lower extremities, and general weakness. She could not walk because of burning pain of knee and ankle. 99mTc-MDP bone scan demonstrated increased periarticular uptake in both lower extremity with focal increased activity in proximal portion of both tibias.. Abnormal abdominal activity in ascending, transverse, and descending colon is also seen. Bone densitometry showed severe osteoporosis in lower extremity. 99mTc-HSA scan showed abnormal radioactivity in small bowel, and descending colon indicating protein losing enteropathy. Endoscopic biopsy of terminal ileum showed a few dilated lymphatics in the submucosa. After steroid and supplemental therapy, the symptoms and signs of both protein losing enteropathy and RSDS much improved simultaneously. Reflex sympathetic dystrophy syndrome is a complex of symptoms characterized by severe pain, swelling, autonomic vasomotor dysfunction, and impaired mobility of affected extremities. The important causes are trauma, fracture, and inflammation. We assumed that the reflex sympathetic dystrophy syndrome of this girl was due to protein losing enteropathy. And as far as we know there has not been reported case that protein losing enteropathy assumed as a cause of reflex sympathetic dystrophy syndrome.