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RISS 인기검색어
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- 저자 : 황장룡(jang Yong Hwang) (검색결과 3 건)
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정준모(Joon Mo Chung),최용환(Yong Hwan Choi),황장룡(jang Yong Hwang),김재왕(Jae Wang Kim),고병구(Byung Goo Ko),곽규식(Kyu Sik Kwack) 대한소화기학회 1985 대한소화기학회지 Vol.17 No.2
N/A Fourty-nine consecutive in patients with cirrhosis and ascites were prospectively investigated for spontaneous bacterial peritonitis. The ascitic fluid was cultured and smeared and was also analyzed for white blood cell count, polymorphonuclear(PML) count and chemical study. Among these patients seven patients(14.3%) were proved as spontaneous bacterial peritonitis. Culture was positive for Eschericha coli, Staphylococcus aureous and Klebsiella pneumoniae. The mean white blood cell count and polymorphonuclear count were 3,791/ mm3 and 3,449/mm3 respectively. In-hospital mortality was 42.9% and causes of death were hepatic failure and hepatorenal syndrome.
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정준모,최용환,황장룡,김재왕,곽규식 대한소화기내시경학회 1985 Clinical Endoscopy Vol.5 No.1
Esophageal ulcers induced by doxycycline is a rare complication. These patients usually complain of sudden onset of symptoms, ie acute substernal or chest pain and odynophagia without prior hietory of esophageal syraptoms. On esophagoscopic examination, there are upper or midesophageal ulcers, which heal after diseontinuation of the drug within 2 weeks. A history of ingestion of the doxycycline,with liquid jost before bedtime can be elicited. The exact eause of the xaucosal ulceration is not clear, but a direct irritant effeet on esophageal mucosa seems most likely. We report 5 cases of esophageal uleeration secondary to the ingestion of doxycydine. Esophagoscopy revealed esophageal ulcers in all patients and the patients hecame asymptomatic following stopping of tbe drugs and taking antacids.
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각종 간질환에서의 혈중 IgM anti-HBc 의 임상적 의의
정준모,최용환,황장룡,김재왕,김교선 대한내과학회 1986 대한내과학회지 Vol.30 No.1
In 130 patients of various liver diseases, in order to assess the clinical significance of serum IgM anti-HBc, we studied the prevalence, serum level and duration of positivity of IgM anti-HBc and its usefulness in diagnosing acute hepatitis B which was negative for HBsAg. The result obtained was as follows, In 23 cases of acute hepatitis B, we found positive reaction of serum IgM anti-HBc in all patients until about 40 days after the onset of the disease, In one patient with acute hepatitis who was Positive for HBsAg, IgM anti-HHc was negative but IgM anti-HA was positive. So we diagnosed this case as HBsAg carrier with acute hepatitis A. IgM anti-HBc was useful to differentiate acute hepatitis R from a HBsAg carriers with acute hepatitis of other causes. The healthy HBsAg carriers and the patients with primary hepatocellular carcinoma-all of them were negative for IgM anti-HBc. In a while, 1 out of 35 patients (3%) with chronic hepatitis B and 3 out of 30 patients (10%) with liver cirrhosis were positive for IgM anti-HBc. In the initial stage of acute hepatitis, B, the titers of serum IgM anti-HBc were higher than those of chronic hepatitis B and liver cirrhosis who were positive for IgM anti-HBc. So the titer of serum IgM anti-HBc was helpful for differentiating acute hepatitis from chronic one. 6 out of 23 patients with acute hepatitis B were aegative for HBsAg but positive for IgM anti-HBc.
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