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유희용 ( Hee Yong Yoo ),정철호 ( Chul Ho Chung ),노미오 ( Mi Oh Roh ),박형규 ( Hyeong Kyu Park ),변동원 ( Dong Won Byon ),서교일 ( Kyo Il Suh ),유명희 ( Myung Hi Yoo ) 대한내과학회 2009 대한내과학회지 Vol.77 No.6
Thyroid tuberculosis is rare, and primary tuberculosis is extremely rare. In most cases, thyroid tuberculosis presents as a thyroid nodule, thyroiditis, thyroid abscess, or thyroid cancer, and is often diagnosed after thyroidectomy. We experienced a case of thyroid tuberculosis that presented as a palpable thyroid nodule in a 32-year-old female patient. The patient had been previously diagnosed with cervical tuberculous lymphadenitis and had been treated successfully 6 years ago. Fine needle aspiration of the thyroid gland showed a small number of neutrophils and macrophages in a necrotic background, and many acid-fast bacilli were found with AFB staining. The patient began anti-tuberculosis therapy as an outpatient and is currently being followed regularly. Here, we report a case of thyroid tuberculosis diagnosed after the apparent cure of cervical lymph node tuberculosis. Additionally, a review of the literature is included. (Korean J Med 77:771-774, 2009)
갑상선 기능항진증에서 인슐린 분비 및 적혈구 인슐린 수용체에 관한 연구
나우균(Woo Kyun Na),김극배(Guk Bae Kim),유명희(Myung Hi Yoo),윤석중(Seuk Joong Yeon) 대한내과학회 1987 대한내과학회지 Vol.33 No.3
N/A Impaired glucose tolerance is well recognized in hyperthyroidism. To investigate the mechanism of glucose intolerance in hyperthyroidism, we performed IV GTT and erythrocyte insulin receptor assay in 7 normal control, 16 thyrotoxic patients and 6 euthyroid patients after antithyroid medication. The glucose decay constant K was decreaaed (K<1.0) in 31% (5/16) of thyrotoxic patients. Insulin secretion at the 1st phase during IV GTT was inereased in thyrotoxic patients compared with normal control (Area under the curve, 761±196 vs 322±49 μU/ml min p<0.05), but 2nd phase insulin secretion showed no significant difference. 125 I-insulin max. specific binding percent of the erythrocyte showed no significant difference in throtoxic patients compared with normal control (p>0.05). Thyrotoxic patients were divided into two groups: group I (n=11) who showed impaired glucose tolerance (K>1.0) and group II (n=5) who showed impaired glucose tolerance (K < 1.0) during IV GTT. 1st phase insulin secretion was increased in group I compared with group II patients (AUC 941±251 vs 310±132 μU/ml min, p<0. 05), while 2nd phase insulin release showed no significant difference. 125 I-insulin max. specific binding percent of erythrocytes showed no significant difference in group I compared with group II (9.80±0.75/o vs 10.40±0.77%, p>0.05). In 5 euthyroid patients on antithyroid medication, insulin secretion & erythrocyte insulin receptor assay showed no significant change after treatment. In conclusion, Ist phase insulin secretion was increased and erythrocyte insulin receptor assay was normal in thyrotoxic patients, suggesting that pheripheral postreceptor defect or hepatic insulin resistance may be responsible for the glucose intolerance of hyperthyroidism.
당뇨병환자에서 동맥경화증의 병인에 관여하는 내피세포 및 혈소판의 기능에 관한 연구
우종욱(Chong Wook Woo),김현수(Hyun Soo Kim),홍대식(Dae Sik Hong),박희숙(Hee Sook Park),유명희(Myung Hi Yoo),김극배(Guk Bae Kim) 대한내과학회 1989 대한내과학회지 Vol.37 No.1
N/A Atherosclerosis is well known as a major complication in diabetes. To investigate the role of platelet function, Factor VIII: vWF and prostacyclin in the pathogenesis of atherosclerosis in diabetes, we performed a platelet function test and measured PGF 1 which is a metabolite of prostacylin, Factor VIII: vWF and thromboxande B2 in 15 patients with diabetes and 10 normal controls. The platelet function test showed that ADP-induced platelet aggregation was significantly enhanced in diabetic patients with retinopathy (80.6±11. 45% vs 42.6±8.71%, p<0.05) compared with normal controls, but collagen induced platelet aggregation or platelet adhesiveness showed no significant difference between diabetic patients and normal controls. The plasma Factor VIII R: Ag level in diabetic patients as not signficantly different from normal controls while plasma Factor VIII: vWF in diabetic patients was significantly increased compared with normal controls (51.33±5.33% vs 27.50±6.71%, p<0.05) The prostaglandin F1α level was signficantly decrease in diabetic patients (2.01±0.32 pg/tube vs 3. 17±0.48pg/tube, p<0.05), while the plasma thromboxane B2 level showed no signficant difference between diabetic patients and normal controls. Our data showed that the increase plasma Factor VIII: vWF level and decrease PGF1α level as well as abnormal platelet function may contribute to the mechanism of atherosclerosis in diabetic patients.