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반응성 밀링에 의해 제조된 Cr<sub>2</sub>O<sub>3</sub> 분산강화형 Cu 합금의 미세조직과 입자조대화
박은범 ( Eun-bum Park ),황승준 ( Seung-joon Hwang ) 한국열처리공학회 2018 熱處理工學會誌 Vol.31 No.4
Copper powder dispersed with 4 vol.% of Cr<sub>2</sub>O<sub>3</sub> was successfully produced by a simple milling at 210 K with a mixture of Cu<sub>2</sub>O, Cu and Cr elemental powders, followed by Hot Pressing (HP) at 1123 K and 50 MPa for 2h to consolidate the milled powder. The microstructure of the HPed material was characterized by standard metallographic techniques such as XRD (X-ray Diffraction), TEM and STEM-EDS. The results of STEMEDS analysis showed that the HPed materials comprised a mixture of nanocrystalline Cu matrix and Cr<sub>2</sub>O<sub>3</sub> dispersoid with a homogeneous bimodal size distribution. The mechanical properties of the HPed materials were characterized by micro Vickers hardness test at room temperature. The thermodynamic considerations on the heat of formation, the incubation time to ignite MSR (Mechanically induced Self-sustaining Reaction), and the adiabatic temperature for the heat of displacement reaction between the oxide-metal are made for the delayed for mation of Cr<sub>2</sub>O<sub>3</sub> dispersoid in terms of MSR suppression. The results of TEM observation and hardness test indicated that the relatively large dispersoids in the HPed materials are attributed to the significant coarsening for the high temperature consolidation; this leads to the low Vickers hardness value. Based on the thermodynamic calculation for the operating processes with a limited number of parameters, the formation kinetics and coarsening of the Cr<sub>2</sub>O<sub>3</sub> dispersoid are discussed. (Received June 14, 2018; Revised June 25, 2018; Accepted July 5, 2018)
Ticlopidine에 의해 유발된 담즙정체간염과 진정적혈구계무형성증 1예
이지연 ( Ji Yeon Lee ),박은범 ( Eun Bum Park ),안재홍 ( Jae Hong Ahn ),서상준 ( Sang Jun Suh ),정영걸 ( Young Kul Jung ),김지훈 ( Ji Hoon Kim ),신봉경 ( Bong Kyung Shin ),양진혁 ( Jin Hyuk Yang ),연종은 ( Jong Eun Yeon ),변관수 ( 대한간학회 2008 Clinical and Molecular Hepatology(대한간학회지) Vol.14 No.1
말기신부전 환자에서 투석치료의 시작이 염증상태 및 면역반응에 미치는 영향에 관한 연구
이재원 ( Jae Won Lee ),김혜원 ( Hye Won Kim ),박은범 ( Eun Bum Park ),부창수 ( Chang Su Boo ),고강지 ( Gang Jee Ko ),조상경 ( Sang Kyung Jo ),조원용 ( Won Yong Cho ),김형규 ( Hyoung Kyu Kim ) 대한신장학회 2007 Kidney Research and Clinical Practice Vol.26 No.5
Purpose : inflammation is a common feature in chronic kidney disease patients, and it could contribute to long-term morbidity and mortality related with malnutrition and atherosclerosis. In this study, we aimed to investigate the effect of initiating dialysis on inflammatory state, nutritional parameter, and immune response in end-stage renal disease (ESRD) patients. Methods : 57 ESRD patients who initiated hemodialysis (HD, n=31) or continuous ambulatory peritoneal dialysis (CAPD, n=26) were enrolled. Pro-inflammatory cytokine, tumor necrosis factor (TNF)-α, and anti-inflammatory cytokines, interleukin (IL)-10 and adiponectin were measured before and 3 months after initiation of dialysis. Inflammatory marker, highly sensitive C-reactive protein (hs-CRP), and nutritional parameter, albumin, were also checked. Lipopolysaccharide (LPS)-stimulated production of TNF-α and IL-10 were measured for the evaluation of immune response by external stimuli. Results : As uremia was reduced by initiating dialysis, serum level of TNF-α was decreased and adiponectin was increased. These changes were accompanied by the decrease of hs-CRP and the increase of serum albumin. LPS-stimulated cytokines production was increased after initiating dialysis. There differences in these parameters comparing HD and CAPD patients except more increase of serum adiponectin level in CAPD patients. Conclusion : Our study demonstrated that initiation of dialysis results in decrease of inflammation, improvement of nutritional status, and restoration of proper immune responsiveness in ESRD patients. These results suggest that correction of uremic milieu through dialysis has beneficial effects. Therefore, initiation of dialysis might have the advantage of improving inflammatory and nutritional status, and correcting immune dysfunction in ESRD patients.