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천공성 급성 담낭염에 의한 복막염으로 복강경적 담낭절제 수술 후 다시 복막투석으로 복귀한 증례 1예
민진수 ( Jin Soo Min ),장세나 ( Se Na Jang ),김형욱 ( Hyung Wook Kim ),신영신 ( Shin Young Shin ),원용성 ( Yong Sung Won ),진동찬 ( Dong Chan Jin ) 대한신장학회 2010 Kidney Research and Clinical Practice Vol.29 No.1
We experienced a 59 year-old female diabetic CAPD patient with severe peritonitis due to perforated acute cholecystitis. Because of heart failure due to old myocardial infarction and cerebral infarction she had been treated with CAPD for 5 years in bed-ridden state. Initial presentation was dark brown colored peritoneal dialysate effluent (changed greenish bile color later) and septic shock. We diagnosed perforated acute cholecystitis by computerized tomography three days after improvement of sepsis. She was received laparoscopic cholecystectomy and continuous venovenous hemodiafiltration for two weeks and returned to peritoneal dialysis without complication. Secondary CAPD peritonitis with cholecystitis or bowel disease should be carefully considered in patients with specific dialysate color, which could be cured with laparoscopic surgery, and then patients can be returned to CAPD again without complication.
결핵성 심막염으로 항결핵약을 복용하던 중 발생한 혈구 탐식증후군
노진희 ( Jin Hee No ),강지영 ( Ji Young Kang ),이보희 ( Bo Hee Lee ),김윤지 ( Yun Ji Kim ),이정은 ( Jung Eun Lee ),민진수 ( Jin Soo Min ),강민규 ( Min Kyu Kang ),김경희 ( Kyung Hee Kim ),윤형규 ( Hyoung Kyu Yoon ),송정섭 ( Jeong S 대한결핵 및 호흡기학회 2008 Tuberculosis and Respiratory Diseases Vol.65 No.6
A 63-year old woman was admitted to our hospital for an evaluation of thrombocytopenia. She had been diagnosed with tuberculous pericarditis three months earlier in a local clinic and treated with anti-tuberculosis medication. Two months later, thrombocytopenia developed. The medication was subsequently stopped because it was suspected that the anti-tuberculosis medication, particularly rifampin, might have caused the severe platelet reduction. However, the thrombocytopenia was more aggravated. A bone marrow biopsy was performed, which showed moderate amounts of histiocytes with active hemophagocytosis. This finding strongly suggested that the critical thrombocytopenia had been caused by hemophagocytic syndrome, not by the side effects of the anti-tuberculosis medication. Furthermore, the development of hemophagocytosis might have been due to an uncontrolled tuberculosis infection and its associated aberrant immunity. Therefore, she was started with both standard anti-tuberculosis medication and chemotherapy using etoposide plus steroid. One month after the initiation of treatment, the thrombocytopenia had gradually improved and she was discharged in a tolerable condition. At the third month of the follow-up, her platelet level and ferritin, the activity marker of hemophagocytic syndrome, was within the normal range. (Tuberc Respir Dis 2008;65:522-526)
이재호 ( Jae ho Lee ),박성환 ( Sung Hwan Park ),박경수 ( Kyung Su Park ),김호연 ( Ho Youn Kim ),최우형 ( Woo Hyung Choi ),전병연 ( Byoung Yeon Jun ),민진수 ( Jin Soo Min ),김세원 ( Sei Won Kim ),정연오 ( Yeon Oh Jeong ),지은혜 ( E 대한류마티스학회 2010 대한류마티스학회지 Vol.17 No.4
Ankylosing spondylitis (AS) is occasionally accompanied by hematological malignancies such as myelodysplastic syndrome, acute myelogenous leukemia, or multiple myeloma. Chronic myelogenous leukemia (CML) is a myeloproliferative disorder associated with Philadelphia chromosome and is usually treated with imatinib, which inhibits tyrosine kinases. Although there have been reports of CML cases accompanied by several rheumatic diseases such as rheumatoid arthritis, Behcet`s disease, systemic sclerosis, or undifferentiated spondylopathy, no studies have reported a case of CML with AS. We experienced a 50-year-old male patient who presented with buttock and low back pain and was diagnosed with both AS and CML. Magnetic resonance imaging showed sacroiliitis along with abnormal marrow infiltration, and a bone marrow biopsy confirmed the CML diagnosis. He was treated with imatinib, which was effective for the CML but not for the AS. This is the first case report of AS accompanied by CML.