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신경섬유종증에 동반된 가성동맥류 파열로 발생한 자연 혈흉
김순종 ( Kim Sun Jong ),정훈 ( Jeong Hun ),이성순 ( Lee Seong Sun ),임채만 ( Im Chae Man ),이상도 ( Lee Sang Do ),고윤석 ( Go Yun Seog ),김우성 ( Kim U Seong ),김동순 ( Kim Dong Sun ),김원동 ( Kim Won Dong ),심태선 ( Sim Tae Seon 대한결핵 및 호흡기학회 2001 Tuberculosis and Respiratory Diseases Vol.50 No.1
김순종 ( Sun Jong Kim ),이응준 ( Eung Jun Lee ),이태훈 ( Tae Hoon Lee ),유광하 ( Kwang Ha Yoo ),이계영 ( Kye Young Lee ) 대한결핵 및 호흡기학회 2006 Tuberculosis and Respiratory Diseases Vol.61 No.1
Pulmonary aspergillosis presents as the following three different types depending on the immune status of the host: invasive aspergillosis, allergic bronchopulmonary aspergillosis (ABPA), and aspergilloma. Aspergilloma develops as a result of an aspergillus growth inside a pre-existing lung cavity. However, endobronchial aspergilloma without a lung parenchymal lesion is quite rare. We encountered a case of endobronchial aspergilloma that developed in a healthy 75 year-old woman that led to necrotizing pneumonia of the right lower lobe. The chief complaints were fever, cough and yellowish sputum. The chest film revealed haziness with cavity-like shadows on the right lower lobe, and the chest CT scan showed endobronchial calcified density in the basal bronchus of the right lower lobe with peribronchial lymph node enlargement. Bronchoscopy revealed an obstruction of the basal orifice of the right lower lobe by blackish stone-like material, and the aspergilloma was confirmed by the bronchoscopic biopsy. The pneumonia improved after bronchoscopic removal of this lesion. We report this case along with a review of the relevant literature. (Tuberc Respir Dis 2006; 61: 60-64)
만성 폐쇄성 폐질환 환자의 중증도 분류시 FEV<sub>1</sub>과 PEFR의 연관성
신상열,윤재호,김순종,유광하,Shin, Sang Youl,Ho, Yoon Jae,Kim, Sun Jong,Yoo, Kwang Ha 대한결핵및호흡기학회 2005 Tuberculosis and Respiratory Diseases Vol.58 No.5
연구배경 : COPD환자에서 질환의 중증도, 치료 반응정도, 급성악화등을 평가하는데 $FEV_1$과 PEFR이 중요한 측정지표로 사용되고 있다. 하지만 COPD환자에서 PEFR과 $FEV_1$의 일치성에 대해서는 잘 알려져 있지 않아 PEFR 측정이 중증도 분류 검사로 사용이 가능한지는 모르는 상태이다. 방 법 : 2003년 9월부터 2004년 8월까지 건국대학교 병원호흡기 내과 외래에서 진료받은 COPD환자 125명을 대상으로 $FEV_1$과 PEFR을 측정하여 그 결과를 통계, 분석하였다. 결 과 : $FEV_1$ 예측치의 평균은 $56.98{\pm}18.21$이었고 PEFR 예측치의 평균은 $70{\pm}27.60$로 PEFR 예측치가 $FEV_1$ 예측치보다 13%정도 높게 측정 되었다. 두 검사 사이에는 유의한 상관관계가 있었다. COPD환자들의 나이와 PEFR 과는 유의한 상관관계가 없었다. 주관적 증상인 호흡 곤란과 PEFR 과는 유의한 상관관계가 있었다. 결 론 : COPD 환자들에서 PEFR 을 이용한 중증도 분류시 $FEV_1$에 비해 경한 쪽으로 분류되는 성향이 있으므로 증상이 심한 경우 중증도 분류 해석에 주의를 요해야 하겠다. COPD 환자들에서 중증도 분류가 확정된 경우 추적 관찰은 PEFR 값으로 $FEV_1$을 대체하는 것이 가능할 것으로 생각된다. Background : Measurement of the $FEV_1$ and PEFR in COPD patients is a significant indicator of the disease severity, the response to treatment and the acute exacerbation. However, it is not known if PEFR can be used to determine the severity of COPD because the agreement between PEFR and $FEV_1$ in COPD patients is not well known. Methods : From September, 2003 to August, 2004, 125 out patients with COPD who were treated at the pulmonary clinic in KonKuk University Hospital were enrolled in this study. The $FEV_1$ and PEFR of each patient were measured and all the data was analyzed using SPSS. Results : The average predicted $FEV_1$ % and PEFR % was $56.98{\pm}18.21%$ and $70{\pm}27.60%$, respectively. There was linear correlation between the predicted $FEV_1$ % and predicted PEFR %. There was no correlation between age of the COPD patients and the predicted PEFR %. There was correlation between dyspnea, which is a subjective symptom of the patients, and the predicted PEFR %. Conclusion : In COPD patients, the classification of the severity by PEFR tends to underestimate the state of the disease compared with the classification of the severity by the $FEV_1$. Therefore, the classification of the severity by PEFR should be interpreted carefully in patients with severe symptoms. Once the classification of the severity has made, the follow-up examination may use the PEFR instead of the $FEV_1$.
나주옥(Joo Ock Na),김순종(Soon Jong Kim),심태선(Tae Sun Shim),임채만(Chae Man Lim),이상도(Sang Do Lee),김우성(Woo Sung Kim),김동순(Dong Soon Kim),김원동(Won Dong Kim),고윤석(Youn Suck Koh) 대한내과학회 2002 대한내과학회지 Vol.62 No.3
Background: Diffuse alveolar hemorrhage (DAH) is an uncommon pulmonary disease. It could be occurred by diver se causes with the different response to the treatment. However, the clinical features of DAH have not been well known in Korea. Methods: Twenty cases identified as DAH between March of 1990 and July of 2000 at a university affiliated hospital were retrospectively reviewed. Results: The median age was 45yr (range: 18 ~73yr) with 11 females. Diagnosis was made by clinical and radiologic findings including hemoptysis, newly developed anemia, and diffuse bilateral lung opacities with the continuous bloody lavage fluid or bloody bronchial aspirate on bronchoscopy. The mean APACHE III score was 50.8 (±26.7) points. Hemoptysis prior to admission was observed in 8 (40%) patients. Diffuse crackles were heard on the bilateral lung fields in 18 cases. On chest radiographs, diffuse ground glass appearance and/or confluent air space consolidation with ill defined irregular margin were mainly observed. In patients with DAH the hemoglobin level fell a mean of 2.0 (±0.8) g/dL. Fourteen (70%) patient s received mechanical ventilation due to the respiratory failure. Six patients (30%) were dead during admission. Among these patients, 4 patients were dead due to refractory respiratory failure with continuous DAH. The bleeding tendency such as disseminated intravascular coagulation or low platelet count was higher in dead patient s than the survivors (p =0.018). The survivor s of DAH with noninfectious causes showed better responses to immunosuppressive drugs with/without plasmapheresis compared to nonsurvivors (p =0.003). Conclusion: DAH was pr one to develop acute respiratory failure and needed mechanical ventilation frequently. However, DAH of non-infectious causes showing a good response to the immunosuppresive therapy would have a better prognosis. In addition, DAH would have a worse prognosis in cases of combined bleeding tendency. (Korean J Med 62:258-267, 2002)
비소세포폐암에서 PNA-Mediated PCR Clamping을 이용한 EGFR 돌연변이 분석법
이계영 ( Kye Young Lee ),김희정 ( Hee Joung Kim ),김순종 ( Sun Jong Kim ),유광하 ( Gwang Ha Yoo ),김원동 ( Won Dong Kim ),오서영 ( Seo Young Oh ),김완섭 ( Wan Seop Kim ) 대한결핵 및 호흡기학회 2010 Tuberculosis and Respiratory Diseases Vol.69 No.4
Background: Recent studies have demonstrated that the epidermal growth factor receptor (EGFR) genotype is the most important predictive marker to EGFR-tyrosine kinase inhibitors (TKIs) and first-line gefitinib treatment will be approved in the near future for use in non-small cell lung cancer (NSCLC) patients with the EGFR mutation. Direct sequencing is known to be the standard for detecting EGFR mutations; however, it has limited sensitivity. Peptide nucleic acids (PNA)-mediated PCR clamping method is a newly introduced method for analyzing EGFR mutations with increased sensitivity and stability. Methods: A total of 71 NSCLC patients were analyzed for EGFR mutations using the PNA-mediated PCR clamping technique. Sixty-nine patients were analyzed for clinicopathologic correlation with EGFR genotype; 2 patients with indeterminate results were excluded. In order to determine EGFR-TKI drug response, 57 patients (42 gefitinib, 15 erlotinib) were included in the analysis. Results: The EGFR mutation rate was 47.8%. Being female, a non-smoker, and having adenocarcinoma were favorable clinicopathologic factors, as expected. However, more than a few smokers (33.3%), male (28.1%), and patients with non-adenocarcinoma (28.6%) had the EGFR mutation. Having a combination of favorable clinicopathologic factors did not increase the EGFR mutation rate significantly. Drug response to EGFR-TKIs showed significant differences depending on the EGFR genotype; ORR was 14.3% for wild type vs 69.0% for mutant type; DCR is 28.6% for wild type vs 96.6% for mutant type. The median EGFR-TKI treatment duration is 7.6 months for mutant type group and 1.4 months for wild type group. Conclusion: EGFR genotype determined using the PNA-mediated PCR clamping method is significantly correlated with the clinical EGFR-TKI responses and PNA-mediated PCR.