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강성귀(Sung Kyew Kang),박성광(Sung Kwang Park),김원(Won Kim) 대한신장학회 2000 Kidney Research and Clinical Practice Vol.19 No.3
The renal cortical necrosis occurs in approximately 2% of adult patients with acute renal failure. The causes of renal cortical necrosis were usually associated with obstetrical problems. However, the distinctive changes occurred over the past 15 years in the etiology with a high incidence of non-obstetric causes than obstetric ones. We experienced a rare case of diffuse bilateral renal cortical necrosis in 16- year-old man who had a history of glue sniffing. To our knowledge, this is the first report in our country. A 16-year-old man was admitted with vague flank pain and oliguria. There was history of frequent adhesive glue sniffing. Laboratory data were as follows : BUN 77mg/dL, creatinine, 9.3mg/dL, protein 3+, RBC many/HPF, WBC 1-4/HPF in urinalysis, HBsAg(-), Anti-HBs(+), C₃10.5mg/dL, C₄7.4mg/ dL), IgG 1,865mg/mL, IgA 512mg/mL. The kindey size was normal in ultrasonography. Renal cortex was diffusely not enhanced in MIJ Tl weighted image. Diffuse bilateral renal cortical necrosis was diag- nosed by renal biopsy. In this case, adhesive glue sniffing may be a cause of acute cortieal necrosis.
당뇨병 신증 환자에서 초음파로 관찰된 경동맥 두께와 Angiotensin Converting Enzyme 유전자와 Apolipoprotein E 유전자 다형성에 대한 연구
강성귀(Sung Kyew Kang),박성광(Sung Kwang Park),김원(Won Kim),김달식(Dal Sik Kim),박태선(Tae Sun Park),백홍선(Hong Sun Baek) 대한신장학회 2000 Kidney Research and Clinical Practice Vol.19 No.3
N/A Backgroud: We evaluated the distribution of the polymorphisms of apolipoprotein E and angiotensin converting enzyme gene in patients with diabetic nephropathy and also evaluated possible association between the apolipoprotein E carriers and angioten-sin converting enzyme genotypes and intima-media thickness of the common carotid artery. Methods: Study participants were 92 patients with diabetic nephropathy(50 men and 42 women). Hb(A1C), albuminuria, and lipid status were assessed by standard laboratory techniques ; the apolipopro-tein E carriers were assessed by modified amplifi- cation refractory mutation system and the angioten-sin converting enzyme genotypes were assessed by polymerase chain reaction. The intima-media thick- ness was measured by high-resolution ultrasonog-raphy. Results : The apolipoprotein E frequencies of patients were E2 8%, E3 76%, and E4 16%. The intima-media thickness varied by apo E groups. E2 group has less common carotid intima-media thickness than E3 and E4 groups(p<0.05). The angiotensin converting enzyme genotypes were distributed as follows ; 35% II, 49% ID, 16% DD. The intima-media thickness value did not differ among patients with various angiotensin converting enzyme genotypes. Multiple logistic regression analysis showed that age and apolipoprotein E genotypes were determinants for the intima-media thickness. Conclusion: Our results suggested that apolipo- protein E polymorphism is associated with carotid artery intima-media thickness in diabetic nephro-pathy. But, we could not find an association between carotid artery intima-media thickness and angioten-sin converting enzyme gene polymorphism in dia- betic nephropathy.
강성귀(Sung Kyew Kang),김원(Won Kim),김인희(In Hee Kim) 대한신장학회 2001 Kidney Research and Clinical Practice Vol.20 No.4
Mannitol is an osmotic diuretic as a useful agent in the treatment of a variety of clnical conditions. In persons with normal renal function, the mannitol is almost excreted by kidney. Although various studies of the effectiveness of mannitol for the cerebral edema have been reported, but there are still few re- ports on acid-base disorder. This study is based on acid-base and electrolyte changes after the intravenous infusion of hypertonic mannitol for the purpose of preventing cerebral edema. Mannitol were intravenously infused with 300-900 mL for 90-minutes (group A), 1,200-2,600 mL for 24-hours(group B) and 3,200-4,900 mL for over 24-hours(group C) each. Each blood sample was drawn for gas analysis, and electrolytes through arterial line before and after mannitol infusion. In group A, blood pH is increased significantly from baseline level 7.43±4 0.07 to 7.46±0.04, and plasma HCO3 25.3±2.1 mEq/L to 28.9±2.9 mEq/ L each, but plasma K is decreased significantly from baseline level 4.3±0.6 mEq/L to 3.70.8 mEq/L In group B, blood pH is increased significantly from baseline level 7.420.02 to 7.47±0.06, and plasma HCO3 25.2±1.8 mEq/L to 29.1±2.9 mEq/L each, but plasma K is decreased significantly from baseline level 4.2±0.3 mEq/L to 3.8±0.5 mEq/L. In group C, blood pH is increased significantly from baseline level 7.41±0.01 to 7.52±0.04, and plasma HCO3 24.9±1.2 mEq/L to 27.7±2.5 mEq/L each, but plasma K is decreased significantly from baseline level 4.20.1 mEq/L to 3.9±0.2 mEq/L These results clinically used intravenous infusion of mannitol could induce metabolic alkalosis associated with hypokalemia, regardless of its dosage.
강성귀(Sung Kyew Kang),박성광(Sung Kwang Park),김원(Won Kim),이정민(Jung Min Lee),마명신(Myeong Sin Ma) 대한신장학회 2000 Kidney Research and Clinical Practice Vol.19 No.1
Huge hepatic cysts are rarely encountered in autosomal dominant polycystic kidney disease. Huge hepatic cyst bothers the patient either by compression or complications such as intracystic infections or hemorrhages. We report, here, alcohol instillation to treat a huge hepatic cyst causing compression symptom with improvement in size & symptom. A 49-year-old female patient presented with a 4-week history of indigestion and epigastric discomfort. Physical examination was remarkable for tender hepatomegaly. Computerized tomography revealed huge hepatic and kidney cysts. Ultrasonographic guided percutaneous drainage and four times alcohol sclerotherapy of the huge heptic cyst was successful. Relief of the symptoms was maintained after 18 days. This case shows that alcohol sclerotherapy can be a effective nonsurgical method of treating symptomatic huge hepatic cysts.
오강열(Kang Yeal Oh),강성귀(Sung Kyew Kang) 대한내과학회 1987 대한내과학회지 Vol.32 No.4
N/A This study was undertaken to investigate whether cell-mediated immunity are affected by renal diseases. Thirty six patients with lipoid nephrosis and chronic proliferative glomerulonephritis, and 16 controlled subjects were studied by summation of delayed-cutaneous hypersensitivity response to 8 test antigens such as Tetamas, Diphtheria, Group C-streptococcus, Tuberculin, Glycerim, Candida albicans, Tricophyton mentaprophytes and protes mirabilis. The total average diameter of all positive reaction in controlled subjects was 15.34±2.03 mm. Of the 16 subjects, negatvie reaction was observed in 3 (18.8%). In patients with lipoid nephrosis, during relapsing phase the total average diameter of all positive reactions was 5.38±2.48 mm. being significantly lower than that of normal subjects. Negative reaction was observed in 9 of 10 patients (90%). The total average diameter of all positive reactions in patients with lipoid nephrosis in the remission phase was 13.09±1.64 mm showing no significant difference in total average diameters between control and remission phase group. Negative reaction was observed in 1 of 9 patients (11.1%). In chronic proliferative glomerulonephritis patients with the nephrotic syndrome, the total average diameter was 10.25±2.20 mm, showing no significant difference with control group, Negative reaction was observed in 3 of 10 patients (30%). The total average diameters of all positive reactions in patients with chronic proliferative glomrulonephritis without nephrotic syndrome was 12. 93±3.48 mm, showing non significant difference in total average diameters between control and patients group. Negative reaction was observed in 2 of 7 patients (28.56%) This study suggests lipoid nephrosis impairs cell mediated immunity, and the impairement of delayed hypersensitivity reaction seems to be related to clinical stages of nephrosis.