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월남전에서 치험한 흉부손상 120례에 대한 임상적 고찰
변해공 대한흉부심장혈관외과학회 1974 Journal of Chest Surgery (J Chest Surg) Vol.7 No.1
During the 35 month period from November 1966 to November 1967 and from June 1971 to March 1973 I had experienced 127 cases of non fatal wounds of chest in Viet-Nam. .Among these 127 cases, 62[45.4%] were gun shot wounds, 49[35.8%] were shrapnel wounds and the other were traffic accident. stab wounds and miscellanous. Approximately 21% of gun shot wound were perforating and 79% were penetrating but all cases of shrapnel wounds were penetrating. Of these 127 cases. 90% evacuated to hospital within 6 hours and average time 2.5 hours. The tranfusion requirement of these cases ranged from zero to 36 pints of whole blood with an average of 2.600cc. Initial intrathoracic findings were hemopneumothorax and hemothorax mostly. and the incidence of open thoracotomy was 9.5%[12cases] and closed thoracotomy was 82.8%[104cases], which were contrast to the reports from Korean conflict. I had experienced 24 cases with complication, such as large hematoma in lung parenchyme[8 cases], atelectasis[4 cases], pyothorax [3 cases], pneumonia [3 cases], fibrothorax [3 cases], pleural effusion [2 cases] and wound infection [2 cases]. Mortality rate for entire group was 4.7% but the cases associated with brain injury was 100%, with spinal cord injury was 50%, with large vessel 50%, and abdominal injury was 33.3%, and nobody died solely of thoracic injury.
胎盤組織 核膜의 Carboxylmethylation에 關한 硏究
邊海貢,林圭,黃炳斗,申石澈 충남대학교 의과대학 지역사회의학연구소 1987 충남의대잡지 Vol.14 No.1
Nuclear envelopes were prepared from human placental nuclei by a combination of sonication, digestion with DNase, potassium chloride treatment and ultracentrifugation, and their carboxylmethylation by protein methylase 5 were investigated. 1. The purity of the nuclear envelope was at least over 80% in terms of its contaminating enzyme activities (fumarase, acid phosphatase, 5'-nucleotidase, catalase and NADH: cytochrome C reductase). 2. ^14C-methyl groups from S-adenosyl-L-(methyl-^14C)methionine were incorporated in the nuclear envelope and nuclear pore complex at a rate of 4.8 and 3.8 pmoles/mg protein/min, respectively. 3. The carboxylmethylation of the nuclear envelope and pore complex protein increased proportionally with time. 4. The optimal pH for the carboxylmethylation of the nuclear envelope was around 5. 4. 5. Copper ion (Cu^2+) inhibited the carboxylmethylation of the nuclear envelope by 50%, but the other metal ions, cAMP, polyamines and insulin were without effect. 6. The carboxylmethylation of the nuclei and nuclear envelopes decreased the binding of carboxylmethylated histone to the nuclei and nuclear envelopes. 7. The carboxylmethylated fractions of the nuclei, nuclear envelopes and pore complex protein had molecular weights of 68, 000 and over 120, 000, as determined by scdium dodecyl sulfate/ polyacrylamide gel electrophoresis. 8. Endogenous phosphorylation of the envelope was not obserbed, but exogenous cAMP dependent phosphorylation occured mainly in the nuclear envelope protein fractions of molecular weights 100, 000, 85, 000, 59, 000 and 51, 000, and the phosphorylation was not affected by the carboxyl methylation of the nuclear eovelope protein. The above results suggest that the carboxylmethylation of nuclear envelopes and pore complex proteins may be involved in the transport of macromolecules into the nucleus.
황병두,변해공,임규,이재흔,백문기 충남대학교 의과대학 지역사회의학연구소 1983 충남의대잡지 Vol.10 No.2
Two forms of adenosine deaminase (ADase) having molecular weights of 1,500,000 & 800,000 in the soluble fraction of the early placenta and plasma membrane bound ADase of the placenta were demonstrated respectively. Their properties and activity changes with gestational age have been investigated. ADases exhibit a broad pH optimum from 6 to 9 and heat labile, being completely inactivated by heat treatment at 70℃ for 10 min. Soluble fraction ADases and plasma membrane bound ADase are inhibited about 100% and 80% by s mM Cu^2+. The apparent Michaelis constant of pADase-cytotrophoblast(pADase-C) for adenosine is 110μM, and for deoxyadenosine 21μM, that of pADase-syncytiotrophoblast(pADase-S) is 80μM and 35μM, and that of plasma membrane bound ADase is 31μM and 26μM, respectively. Kinetic analysis of pADase-C and pADase-S in the presence of 10μM Cu^2+ show that the nature of the inhibition to pADase-C and pADase-S are noncompetitive and uncompetitive, respectively. The pADases (pADase-C + pADase-S) activity decreased with gestational age, the activity at term being about one-tenth that of the early placenta, while plasma membrane bound ADase increased with gestational age by about 2-fold. From above results it is discussed that pADase-C is related to cytotrophoblast, pADase-S to syncytiotrophoblast and plasma membrane bound ADase to cellular differentiation of placenta, and suggested that soluble fraction ADases of human placenta is different from plasma membrane bound ADase in their biological roles.