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SEARCH FOR ASTROPHYSICAL NEUTRINO POINT SOURCES AT SUPER-KAMIOKANDE
Thrane, E.,Abe, K.,Hayato, Y.,Iida, T.,Ikeda, M.,Kameda, J.,Kobayashi, K.,Koshio, Y.,Miura, M.,Moriyama, S.,Nakahata, M.,Nakayama, S.,Obayashi, Y.,Ogawa, H.,Sekiya, H.,Shiozawa, M.,Suzuki, Y.,Takeda, IOP Publishing 2009 The Astrophysical journal Vol.704 No.1
<P>It has been hypothesized that large fluxes of neutrinos may be created in astrophysical 'cosmic accelerators.' The primary background for a search for astrophysical neutrinos comes from atmospheric neutrinos, which do not exhibit the pointlike directional clustering that characterizes a distant astrophysical signal. We perform a search for neutrino point sources using the upward-going muon data from three phases of operation (SK-I, SK-II, and SK-III) spanning 2623 days of live time taken from 1996 April 1 to 2007 August 11. The search looks for signals from suspected galactic and extragalactic sources, transient sources, and uncataloged sources. While we find interesting signatures from two objects-RX J1713.7-3946 (97.5% CL) and GRB 991004D (95.3% CL)-these signatures lack compelling statistical significance given trial factors. We set limits on the flux and fluence of neutrino point sources above energies of 1.6 GeV.</P>
Spiral spin structures and origin of the magnetoelectric coupling inYMn2O5
Kim, J.-H.,Lee, S.-H.,Park, S. I.,Kenzelmann, M.,Harris, A. B.,Schefer, J.,Chung, J.-H.,Majkrzak, C. F.,Takeda, M.,Wakimoto, S.,Park, S. Y.,Cheong, S-W.,Matsuda, M.,Kimura, H.,Noda, Y.,Kakurai, K. American Physical Society 2008 Physical review. B, Condensed matter and materials Vol.78 No.24
Search for dinucleon decay into pions at Super-Kamiokande
Gustafson, J.,Abe, K.,Haga, Y.,Hayato, Y.,Ikeda, M.,Iyogi, K.,Kameda, J.,Kishimoto, Y.,Miura, M.,Moriyama, S.,Nakahata, M.,Nakajima, T.,Nakano, Y.,Nakayama, S.,Orii, A.,Sekiya, H.,Shiozawa, M.,Takeda, American Physical Society 2015 PHYSICAL REVIEW D - Vol.91 No.7
Kim, D Y,Ryu, D H,Hong, J B,Park, J-G,Kwon, Y S,Jung, M A,Jung, M H,Takeda, N,Ishikawa, M,Kimura, S IOP Pub 2004 Journal of Physics, Condensed Matter Vol.16 No.46
<P>We report on the magnetic susceptibility, specific heat and electrical resistivity of the heavy fermion compounds CeNiGe<SUB>2−<I>x</I></SUB>Si<SUB><I>x</I></SUB> (<img ALIGN='MIDDLE' SRC='http://ej.iop.org/images/0953-8984/16/46/018/cm185233ieqn1.gif' ALT='0\leq x \leq 1 '/>). Compounds with <I>x</I><1 show antiferromagnetic order, which with increasing <I>x</I> shifts toward lower temperature owing to increased exchange coupling between the localized 4f magnetic moments and conduction electrons. Eventually, the magnetic order almost becomes absent, for <I>x</I> = 1. An anomaly observed in the specific heat is well interpreted by the Kondo model for a degenerate impurity spin <I>J</I> = 1/2 in the Coqblin–Schrieffer limit. A coherence peak indicative of the formation of a Kondo lattice is found in the electrical resistivity, whose features are consistent with the results for the specific heat. Interestingly, there is a significant deviation from Fermi-liquid behaviour at the critical concentration <I>x</I> = 1. This deviation is attributed to a quantum phase transition in a model with two-dimensional antiferromagnetic fluctuations.</P>
Regulation of Wound Healing and Fibrosis by Hypoxia and Hypoxia-Inducible Factor-1
Robin J. Ruthenborg,반재준,Anum Wazir,Norihiko Takeda,Jung-whan Kim 한국분자세포생물학회 2014 Molecules and cells Vol.37 No.9
Wound healing is a complex multi-step process that requires spatial and temporal orchestration of cellular and non-cellular components. Hypoxia is one of the prominent microenvironmental factors in tissue injury and wound healing. Hypoxic responses, mainly mediated by a master transcription factor of oxygen homeostasis, hypoxia-inducible factor-1 (HIF-1), have been shown to be critically involved in virtually all processes of wound healing and remodeling. Yet, mechanisms underlying hypoxic regulation of wound healing are still poorly understood. Better understanding of how the wound healing process is regulated by the hypoxic microen¬vironment and HIF-1 signaling pathway will provide insight into the development of a novel therapeutic strategy for impaired wound healing conditions such as diabetic wound and fibrosis. In this review, we will discuss recent studies illuminating the roles of HIF-1 in physiologic and pathologic wound repair and further, the therapeutic potentials of HIF-1 stabilization or inhibition.