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Review : Apoptotic Cell Death Following Traumatic Injury to the Central Nervous System
(Joe E . Springer) 생화학분자생물학회 2002 BMB Reports Vol.35 No.1
Apoptotic cell death is a fundamental and highly regulated biological process in which a cell is instructed to actively participate in its own demise. This process of cellular suicide is activated by developmental and environmental cues and normally plays an essential role in eliminating superfluous, damaged, and senescent cells of many tissue types. In recent years, a number of experimental studies have provided evidence of widespread neuronal and glial apoptosis following injury to the central nervous system (CNS). These studies indicate that injury-induced apoptosis can be detected from hours to days following injury and may contribute to neurological dysfunction. Given these findings, understanding the biochemical signaling events controlling apoptosis is a first step towards developing therapeutic agents that target this cell death process. This review will focus on molecular cell death pathways that are responsible for generating the apoptotic phenotype. It will also summarize what is currently known about the apoptotic signals that are activated in the injured CNS, and what potential strategies might be pursued to reduce this cell death process as a means to promote functional recovery.
Apoptotic Cell Death Following Traumatic Injury to the Central Nervous System
Springer, Joe E. Korean Society for Biochemistry and Molecular Biol 2002 Journal of biochemistry and molecular biology Vol.35 No.1
Apoptotic cell death is a fundamental and highly regulated biological process in which a cell is instructed to actively participate in its own demise. This process of cellular suicide is activated by developmental and environmental cues and normally plays an essential role in eliminating superfluous, damaged, and senescent cells of many tissue types. In recent years, a number of experimental studies have provided evidence of widespread neuronal and glial apoptosis following injury to the central nervous system (CNS). These studies indicate that injury-induced apoptosis can be detected from hours to days following injury and may contribute to neurological dysfunction. Given these findings, understanding the biochemical signaling events controlling apoptosis is a first step towards developing therapeutic agents that target this cell death process. This review will focus on molecular cell death pathways that are responsible for generating the apoptotic phenotype. It will also summarize what is currently known about the apoptotic signals that are activated in the injured CNS, and what potential strategies might be pursued to reduce this cell death process as a means to promote functional recovery.
Antibacterial mechanism of ZnO nanoparticles under dark conditions
Joe, A.,Park, S.H.,Shim, K.D.,Kim, D.J.,Jhee, K.H.,Lee, H.W.,Heo, C.H.,Kim, H.M.,Jang, E.S. Korean Society of Industrial and Engineering Chemi 2017 Journal of industrial and engineering chemistry Vol.45 No.-
<P>To evaluate the antibacterial activity of zinc oxide (ZnO) in the absence of a light source, we examined three different types of nanoparticles that vary in size and the number of oxygen defect sites. Colony forming units (CFU) and various microscopic investigations revealed that the antibacterial activity of ZnO nanoparticles under dark conditions was not related to reactive oxygen species (ROS) generation and nanoparticles transfection, but depended on the ZnO attachment to bacterial cell walls and increasing concentrations of Zn2+ ions in the bacterial cytoplasm due to local dissolution of the attached ZnO. (C) 2016 The Korean Society of Industrial and Engineering Chemistry. Published by Elsevier B.V. All rights reserved.</P>
무손실 스너버 적용 소프트 스위칭 Forward 컨버터
김은수(E S. Kim),김태진(K Y. Joe),최해영(T J. Kim),조기연(H Y. Choi),김윤호(Y H. Kim) 전력전자학회 1997 전력전자학술대회 논문집 Vol.1997 No.-
To achieve high efficiency in high power and high frequency applications, reduction of switching losses and noise is very important. In this paper, an improved soft switching forward converter is proposed. The proposed converter is constructed by using non-dissipative snubbers in parallel with the main switch and output diode of the conventional forward converter. Due to the use of the non-dissipative snubbers in the primary and secondary, the proposed converter achieves zero-voltage and zero-current switching for all switching devices without switching losses and output diode recovery losses. The complete operating principles, theoritical analysis, experimental results will be presented.