http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Fenglan Sun,Mingyan Tuo,Jürgen Kurths,Wei Zhu 제어·로봇·시스템학회 2020 International Journal of Control, Automation, and Vol.18 No.8
This paper studies the finite-time consensus of leader-following multi-agent systems with multiple time delays over time-varying topology. The finite-time consensus protocol based on the agents’ states and the communication topology is designed. By adopting the algebraic graph theory, Lyapunov stability theory and pinning control method, some sufficient conditions for the finite-time consensus are given. It is proved that the system can reach consensus in a finite time both over the connected and disconnected topology. Moreover, the upper bound of the convergence time is given. Several simulations are presented to verify the effectiveness of the adopted method.
Kim, Hyung-Goo,Ahn, Jang-Won,Kurth, Ingo,Ullmann, Reinhard,Kim, Hyun-Taek,Kulharya, Anita,Ha, Kyung-Soo,Itokawa, Yasuhide,Meliciani, Irene,Wenzel, Wolfgang,Lee, Deresa,Rosenberger, Georg,Ozata, Metin Elsevier 2010 American journal of human genetics Vol.87 No.4
<P>By defining the chromosomal breakpoint of a balanced t(10;12) translocation from a subject with Kallmann syndrome and scanning genes in its vicinity in unrelated hypogonadal subjects, we have identified <I>WDR11</I> as a gene involved in human puberty. We found six patients with a total of five different heterozygous <I>WDR11</I> missense mutations, including three alterations (A435T, R448Q, and H690Q) in WD domains important for β propeller formation and protein-protein interaction. In addition, we discovered that WDR11 interacts with EMX1, a homeodomain transcription factor involved in the development of olfactory neurons, and that missense alterations reduce or abolish this interaction. Our findings suggest that impaired pubertal development in these patients results from a deficiency of productive WDR11 protein interaction.</P>
Yongzhen Guo,Yang Luo,Weiping Wang,Xiong Luo,Chao Ge,Jürgen Kurths,Manman Yuan,Yang Gao 제어·로봇·시스템학회 2020 International Journal of Control, Automation, and Vol.18 No.2
This paper focuses on the dynamical characteristics of complex-valued memristor-based BAM neural network (CVMBAMNN) with leakage time-varying delay. With two different controllers, we have obtained fixedtime and finite-time synchronization criteria respectively in complex domain for our special model, which few work has studied before. Since fixed-time synchronous system can improve communication security, we designed a scheme for RGB image encryption and decryption. In order to satisfy the requirement of much lower error in image secure communication, our approach can get the error of fixed-time synchronization to about 1×10−13. Due to our highly consistent system, we do get good encryption and decryption effect with encryption and decryption scheme. Finally, numerical simulations are included to demonstrate the correctness of our theoretical results.
Kim, H.G.,Kim, H.T.,Leach, Natalia T.,Lan, F.,Ullmann, R.,Silahtaroglu, A.,Kurth, I.,Nowka, A.,Seong, I.,Shen, Y.,Talkowski, Michael E.,Ruderfer, D.,Lee, J.H.,Glotzbach, C.,Ha, K.,Kjaergaard, S.,Levin University of Chicago Press [etc.] 2012 American journal of human genetics Vol.91 No.1
Potocki-Shaffer syndrome (PSS) is a contiguous gene disorder due to the interstitial deletion of band p11.2 of chromosome 11 and is characterized by multiple exostoses, parietal foramina, intellectual disability (ID), and craniofacial anomalies (CFAs). Despite the identification of individual genes responsible for multiple exostoses and parietal foramina in PSS, the identity of the gene(s) associated with the ID and CFA phenotypes has remained elusive. Through characterization of independent subjects with balanced translocations and supportive comparative deletion mapping of PSS subjects, we have uncovered evidence that the ID and CFA phenotypes are both caused by haploinsufficiency of a single gene, PHF21A, at 11p11.2. PHF21A encodes a plant homeodomain finger protein whose murine and zebrafish orthologs are both expressed in a manner consistent with a function in neurofacial and craniofacial development, and suppression of the latter led to both craniofacial abnormalities and neuronal apoptosis. Along with lysine-specific demethylase 1 (LSD1), PHF21A, also known as BHC80, is a component of the BRAF-histone deacetylase complex that represses target-gene transcription. In lymphoblastoid cell lines from two translocation subjects in whom PHF21A was directly disrupted by the respective breakpoints, we observed derepression of the neuronal gene SCN3A and reduced LSD1 occupancy at the SCN3A promoter, supporting a direct functional consequence of PHF21A haploinsufficiency on transcriptional regulation. Our finding that disruption of PHF21A by translocations in the PSS region is associated with ID adds to the growing list of ID-associated genes that emphasize the critical role of transcriptional regulation and chromatin remodeling in normal brain development and cognitive function.
Simultaneous Pi2 observations by the Van Allen Probes inside and outside the plasmasphere
Ghamry, E.,Kim, K.‐,H.,Kwon, H.‐,J.,Lee, D.‐,H.,Park, J.‐,S.,Choi, J.,Hyun, K.,Kurth, W. S.,Kletzing, C.,Wygant, J. R.,Huang, J. American Geophysical Union 2015 JOURNAL OF GEOPHYSICAL RESEARCH. SPACE PHYSICS Vol.120 No.6
Migraine Mutations Increase Stroke Vulnerability by Facilitating Ischemic Depolarizations
Eikermann-Haerter, Katharina,Hyun Lee, Jeong,Yuzawa, Izumi,Liu, Christina H.,Zhou, Zhipeng,Kyoung Shin, Hwa,Zheng, Yi,Qin, Tao,Kurth, Tobias,Waeber, Christian,Ferrari, Michel D.,van den Maagdenberg, A Ovid Technologies Wolters Kluwer -American Heart A 2012 CIRCULATION - Vol.125 No.2