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      • Prototype Theory and Case Assignment

        ( Yumiko Kinjo ),( Kobe University ) 한국언어정보학회 1995 국제 워크샵 Vol.1995 No.-

        Case assignment is one of the most important issues in theoretical lin-guistics. The goal of this paper is to describe both syntactic and semantic bases for case assignment in terms of PrototY.I.1~theory [1,4}. and to give an explanation for the difference in case assignment between English and Japanese by examining the Instrument Subject construction and deverbal nouns. English allows the structural case assignment under the syntactic prototype, while Japanese does not, because it has a strong requirement for the semantic prototype.

      • SCISCIESCOPUS

        Influence of Ni on the magnetocaloric effect in Nanoperm-type soft-magnetic amorphous alloys

        Podmiljsak, B.,Kim, J.H.,McGuiness, P.J.,Kobe, S. Elsevier Sequoia 2014 JOURNAL OF ALLOYS AND COMPOUNDS Vol.591 No.-

        We have studied the influence of Ni on the magnetocaloric effect (MCE) in Nanoperm-type amorphous materials by investigating a series of Fe<SUB>84-x</SUB>Ni<SUB>x</SUB>Zr<SUB>6</SUB>B<SUB>10</SUB> alloys with x=0, 2, 4 and 6. As expected, the Curie temperature increased with the amount of Ni from 427K for x=0 to 482K for x=6. The maximum magnetic entropy change (ΔS<SUB>M</SUB><SUP>pk</SUP>) for an applied field of 1.4T also increased, reaching a value of 1.52J/Kkg for x=6, which is an increase of 25% compared to the Ni-free alloy. The refrigerant capacity first decreased for x=2 and then increased, reaching a maximum value of 93J/kg (ΔH=1.4T) for x=6. For a 5T field change, the value increased to 407J/kg, which is higher than the 355J/kg achieved with Gd<SUB>5</SUB>Ge<SUB>1.9</SUB>Si<SUB>2</SUB>Fe<SUB>0.1</SUB>. We confirmed the proposed ''master curve'' behavior for second-order magnetic transition (SOMT) alloys for the temperature dependence of ΔS<SUB>M</SUB> for different alloy compositions of the same series, which makes it easier and faster to find proper candidates for a magnetic refrigerator.

      • Conscientiousness Is Not Always a Good Predictor of Performance: The Case of Creativity

        Roni Reiter-Palmon,Jody J. Illies,Lisa M. Kobe-Cross 대한사고개발학회 2009 The International Journal of Creativity & Problem Vol.19 No.2

        Meta-analyses investigating the relationship between Conscientiousness and per formance suggest a positive relationship for a variety of criteria. However, recently it has been argued that Conscientiousness is not always a good predictor of performance, particularly for creative performance. Additionally, it has been suggested that Conscientiousness includes two distinct components, achievement and dependability, which may have different relationships with criterion measures. Two studies were conducted to determine whether the components of Conscientiousness predict creativity better than the full factor. Students in each study completed a measure of the Five Factor Model and a measure of creative performance. In the first study, creative accomplishments were measured and in the second study, creative problem solving was measured. As predicted, both studies revealed a cooperative suppression effect when analyzing the conscientiousness components together such that achievement was positively related and dependability negatively related to creative performance. Also, both studies showed that the overall Conscientiousness factor was not related to creativity.

      • The Nuclear Immune Receptor <i>RPS4</i> Is Required for <i>RRS1<sup>SLH1</sup></i> -Dependent Constitutive Defense Activation in <i>Arabidopsis thaliana</i>

        Sohn, Kee Hoon,Segonzac, Cé,cile,Rallapalli, Ghanasyam,Sarris, Panagiotis F.,Woo, Joo Yong,Williams, Simon J.,Newman, Toby E.,Paek, Kyung Hee,Kobe, Bostjan,Jones, Jonathan D. G. Public Library of Science 2014 PLoS genetics Vol.10 No.10

        <▼1><P>Plant nucleotide-binding leucine-rich repeat (NB-LRR) disease resistance (R) proteins recognize specific “avirulent” pathogen effectors and activate immune responses. NB-LRR proteins structurally and functionally resemble mammalian Nod-like receptors (NLRs). How NB-LRR and NLR proteins activate defense is poorly understood. The divergently transcribed Arabidopsis <I>R</I> genes, <I>RPS4</I> (resistance to <I>Pseudomonas syringae</I> 4) and <I>RRS1</I> (resistance to <I>Ralstonia solanacearum</I> 1), function together to confer recognition of <I>Pseudomonas</I> AvrRps4 and <I>Ralstonia</I> PopP2. <I>RRS1</I> is the only known recessive NB-LRR <I>R</I> gene and encodes a WRKY DNA binding domain, prompting suggestions that it acts downstream of RPS4 for transcriptional activation of defense genes. We define here the early RRS1-dependent transcriptional changes upon delivery of PopP2 <I>via Pseudomonas</I> type III secretion. The Arabidopsis <I>slh1</I> (<I>sensitive to low humidity 1</I>) mutant encodes an RRS1 allele (RRS1<SUP>SLH1</SUP>) with a single amino acid (leucine) insertion in the WRKY DNA-binding domain. Its poor growth due to constitutive defense activation is rescued at higher temperature. Transcription profiling data indicate that RRS1<SUP>SLH1</SUP>-mediated defense activation overlaps substantially with AvrRps4- and PopP2-regulated responses. To better understand the genetic basis of RPS4/RRS1-dependent immunity, we performed a genetic screen to identify <I><U>su</U>ppressor of</I><U>s</U>l<U>h</U>1 <I><U>i</U>mmunity</I> (<I>sushi</I>) mutants. We show that many <I>sushi</I> mutants carry mutations in <I>RPS4</I>, suggesting that RPS4 acts downstream or in a complex with RRS1. Interestingly, several mutations were identified in a domain C-terminal to the RPS4 LRR domain. Using an <I>Agrobacterium</I>-mediated transient assay system, we demonstrate that the P-loop motif of RPS4 but not of RRS1<SUP>SLH1</SUP> is required for RRS1<SUP>SLH1</SUP> function. We also recapitulate the dominant suppression of RRS1<SUP>SLH1</SUP> defense activation by wild type RRS1 and show this suppression requires an intact RRS1 P-loop. These analyses of RRS1<SUP>SLH1</SUP> shed new light on mechanisms by which NB-LRR protein pairs activate defense signaling, or are held inactive in the absence of a pathogen effector.</P></▼1><▼2><P><B>Author Summary</B></P><P>How plant NB-LRR resistance proteins and the related mammalian Nod-like receptors (NLRs) activate defense is poorly understood. Plant and animal immune receptors can function in pairs. Two Arabidopsis nuclear immune receptors, RPS4 and RRS1, confer recognition of the unrelated bacterial effectors, AvrRps4 and PopP2, and activate defense. Using delivery of PopP2 into Arabidopsis leaf cells <I>via Pseudomonas</I> type III secretion, we define early transcriptional changes upon RPS4/RRS1-dependent PopP2 recognition. We show an auto-active allele of RRS1, RRS1<SUP>SLH1</SUP>, triggers transcriptional reprogramming of defense genes that are also reprogrammed by AvrRps4 or PopP2 in an RPS4/RRS1-dependent manner. To discover genetic requirements for RRS1<SUP>SLH1</SUP> auto-activation, we conducted a suppressor screen. Many <I>suppressor of</I> slh1 <I>immunity</I> (<I>sushi</I>) mutants that are impaired in RRS1<SUP>SLH1</SUP>-mediated auto-activation carry loss-of-function mutations in RPS4. This suggests that RPS4 functions as a signaling component together with or downstream of RRS1-activated immunity, in contrast to earlier hypotheses, significantly advancing our understanding of how immune receptors activate defense in plants.</P></▼2>

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