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Infuence of Diferent XLPE Cable Defects on the Initiation of Electric Trees
Fei Qi,Dai Wan,Xi OuYang,Qian Jiang,Miao Zhao,Hengyi Zhou 대한전기학회 2019 Journal of Electrical Engineering & Technology Vol.14 No.6
Defects in the insulation layer of cross-linked polyethylene (XLPE) cable are the leading cause of electrical tree. The mechanism that how the diferent insulation defects infuence on the starting of electrical tree should be explored, and it will be utilized to improve cable insulation in the production and installation. In the paper, frstly the spike-air gap defect as the main defect characteristics of cable fault has been determined by analyzing the actual cable defect type and infuence. Then, the needle-electrode short cable test system was built and the representative defect model was constructed, in order to simulate the infuence of the spike-air gap defect of diferent sizes in the outer semiconductor layer on the initial voltage of electrical tree. Thirdly, the Physical model was created to analyze the infuence of each defect parameters. The research showed that the electrical tree of XLPE cable was mainly caused by defects in the semiconductor layer. The maximum electric feld distribution can be formulized by the maximum feld strength Mason model. According to the formulation and the test results, the main parameters, including the spur curvature, the micro-pore pressure and the initial energy of generating electric tree, have a positive promotion on the initial voltage of the electrical tree and can hinder the tree developing. Therefore, utilizing the interface treatment method between the insulation layer and the semiconductor layer, the insulation heat treatment process and the flling of the inert gas are to improve the parameters and further resist electrical tree aging in the production and installation.
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Lin Shuang-Zhe,Wu Wei-Jie,Cheng Yu-Qing,Zhang Jian-Bin,Jiang Dai-Xi,Ren Tian-Yi,Ding Wen-Jin,Liu Mingxi,Chen Yuan-Wen,Fan Jian-Gao 생화학분자생물학회 2023 Experimental and molecular medicine Vol.55 No.-
Macrophages are immune cells crucial for host defense and homeostasis maintenance, and their dysregulation is involved in multiple pathological conditions, such as liver fibrosis. The transcriptional regulation in macrophage is indispensable for fine-tuning of macrophage functions, but the details have not been fully elucidated. Prolyl endopeptidase (PREP) is a dipeptidyl peptidase with both proteolytic and non-proteolytic functions. In this study, we found that Prep knockout significantly contributed to transcriptomic alterations in quiescent and M1/M2-polarized bone marrow-derived macrophages (BMDMs), as well as aggravated fibrosis in an experimental nonalcoholic steatohepatitis (NASH) model. Mechanistically, PREP predominantly localized to the macrophage nuclei and functioned as a transcriptional coregulator. Using CUT&Tag and co-immunoprecipitation, we found that PREP was mainly distributed in active cis-regulatory genomic regions and physically interacted with the transcription factor PU.1. Among PREP-regulated downstream genes, genes encoding profibrotic cathepsin B and D were overexpressed in BMDMs and fibrotic liver tissue. Our results indicate that PREP in macrophages functions as a transcriptional coregulator that finely tunes macrophage functions, and plays a protective role against liver fibrosis pathogenesis.
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