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급성(急性) 신부전(腎不全)이 동반(同伴)된 근양사(筋壤死)에 있어서의 $^{99m}Tc$-MDP 골주사(骨走査)
문희범,한진석,김삼용,조보연,이정상,고창순,조경삼,Moon, H.B.,Han, J.S.,Kim, S.Y.,Cho, B.Y.,Lee, J.S.,Koh, C.S.,Cho, K.S. 대한핵의학회 1980 핵의학 분자영상 Vol.14 No.2
We studied four patients with muscle necrosis associated with acute renal failure to evaluate the diagnostic value of the bone scan in this disease. The illness followed carbon monoxide poisoning in two patients, acute physical exertion in one and contaminated intramuscular injection in the other. Whole-body rectilinear bone scans using technetium 99m-methyldiphosphonate were done. In all patients, increased muscle labelling at the regions of suspected muscle injury was showed, and in one, it was after normalization of serum muscle enzyme levels. In one patient, the bone scan was rechecked 8 months later and showed no residual abnormality. Above all, the site and precise extent of muscle injury could be detected and the degree of muscle labelling seemed to correlate with the severy of muscle injury. These findings suggest that isotope scanning may be useful in the diagnosis of patients with acute muscle necrosis.
그레이브스병 환자의 Suppressor Induction Factor ( SIF ) 의 활성도
양인명(I . M . Yang),윤휘중(H . J . Yoon),조경삼(K . S . Cho),김진우(J . W . Kim),김광원(K . W . Kim),최영길(Y . K . Choi) 대한내과학회 1986 대한내과학회지 Vol.31 No.1
N/A Autologous mixed lymphocyte culture reaction (AMLCR) has been known to represent the generation of suppressor T cell without direct evidence that the proliferating cell is suppressor T cell itself. Our previous study had demonstrated that the proliferative response of T cell during AMLCR in the patients with Graves' disease is not different from that in normal control, The recent studies showed that suppressor induction factor(SIF) which induce suppressor T cell is produced during the AMLCR. We observed the SIF activity in the patients with Graves disease by measuring the suppression of normal allogeneic MLCR by the normal T cell incubated with the supernatant of AVLCR, and also observed the proliferative response of T cell incubated with the supernatant of AMLCR proliferate to determine whether the suppressor T cell proliferate or not during AMLCR. T cells incubated with the supernatant of AMLCR suppressed the allogeneic MLCR prominently in normal controls(55±11%), and the supernatant of patients' AMLC induced the suppressor T cells which successfully suppressed the allogeneic MLCR(50±9%). T cells incubated with the supernatant of AMLCR did not proliferate. These data indicate that the proliferating cells during AMLCR are the self-antigen recognizing T cells which secrets SIF, a kind of lymphokines, to induce the suppressor T cells, It is suggested that the process of self antigen recognization is normal in Graves' disease.