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앱시스산 (酸) 에 의해 기공이 (氣孔) 닫히는 신호전달과정에서 G-단백질의 역할
이준상(Joon Sang Lee),이한주(Han Ju Yi),이수민(Su Min Lee),이영숙(Young Sook Lee) 한국식물학회 1994 Journal of Plant Biology Vol.37 No.4
Activation of phospholipase C (PLC) and consequent elevation of inositol 1,4,5-trisphosphate (IP_3) level have been reported during ABA-induced closure of stomata. However, it is not known yet whether G-protein regulates the PLC activity during the process. We tested if G-protein is involved in ABA-induced stomatal closure, using pertussis toxin (PT_χ), which inactivates its substrate G-proteins, and cholera toxin, which activates its substrate G-proteins. PT_χ inhibited ABA-induced stomatal closure both in intact epidermal peels from abaxial surface of Commelina communis L. and in partially digested epidermal tissue of Vicia faba L., which contained only guard cells and no other epidermal cells. Cholera toxin did not show any of the effect. Moreover, PT_χ completely blocked ABA-induced elevation of IP_3 level in partially digested epidermal tissue of V. faba L.. These results indicate that PT_χ-sensitive, PLC-linked G-protein is involved in signal transduction process of ABA in guard cells.