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에 대한 혈청 IgG 역가로 위 - 십이지장질환을 감별진단할 수 있는가 ?
최지용(Ji Yong Choi),김학양(Hak Yang Kim),윤덕형(Duck Hyoung Yoon),노승혁(Seung Hyuk Rho),김수진(Soo Jin Kim),이자영(Ja Young Lee),백광호(Gwang Ho Baik),김억(Auk Kim),이진헌(Jin Heon Lee),김용범(Yong Bum Kim),김우중(Woo Joong Kim),유 대한소화기학회 2002 대한소화기학회지 Vol.39 No.3
Background/Aims: Among the diagnostic methods to detect Helicobacter pylori (H. pylori) infection, serological test is a noninvasive, safe, and inexpensive method in clinical practice. This study was performed to determine whether the titer of anti-H. pylori IgG antibody was correlated with endoscopic findings of gastroduodenal diseases, gastric ulcer stage, and age. Methods: Two hundred and eighteen patients who were H. pylori positive by serological method and received gastroduodenal endoscopy were enrolled. All patients had the titers of anti-H. pylori IgG antibody >300 U/mL. We compared the antibody titer according to gastroduodenal diseases, peptic ulcer stage, and age group. Results: There was no statistically significant difference between serum anti-H. pylori IgG antibody titer according to gastroduodenal diseases (chronic gastritis, 999.8±120 U/mL; gastric ulcer, 1043±155.6 U/mL; duodenal ulcer, 1018.5±121.9 U/mL; gastric cancer, 1351±266 U/mL; p=0.504). No significant difference in the anti-H. pylori IgG antibody titer was observed according to peptic ulcer stage (p=0.065) and age (p=0.791). Conclusions: The serum anti-H. pylori IgG antibody titer is not valuable in differentiating the gastroduodenal diseases. (Korean J Gastroenterol 2002;39:167-172)
배영덕 ( Young Deok Bae ),김수진 ( Soo Jin Kim ),남택만 ( Teck Man Nam ),조한수 ( Han Su Cho ),노승혁 ( Seung Hyuk Rho ),윤덕형 ( Duck Hyoung Yoon ),윤종우 ( Jong Woo Yoon ),이삼열,남은숙,오국환 ( Kook Hwan Oh ),채동완 ( Dong Wan Cha 대한신장학회 2002 Kidney Research and Clinical Practice Vol.21 No.6
Immunosuppressive medications after renal allograft transplantation have impacted the course of acute and chronic rejection; however, they have no defined effects on the prevention of recurrent and Glomerulonephritis (GN) in an allograft kidney. Authors experienced a case of rapidly progressive glomerulonephritis (RPGN). The 35-year-old female patient developed a rapid deterioration of renal function 4 years after renal transplantation. The allograft biopsy showed crescentic glomerulonephritis evolving from membranoproliferative glomerulonephritis (MPGN) type Ⅰ. She was given pulse steroid and oral cyclophosphamide therapy immediately after the renal biopsy. Graft function stabilized and proteinuria decreased even though graft function did not recover to pre-treatment level and low grade proteinuria persisted.