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노인의 정보화 기기 이용 불편감이 삶의 만족도에 미치는 영향: 사회적 존중감과 차별 경험의 이중매개효과
송시영 한국노인복지학회 2023 노인복지연구 Vol.78 No.4
본 연구는 노인의 정보화 기기 이용 불편감이 삶의 만족도에 미치는 영향을 사회적 존중감과 차별 경험이 매개하는지 살펴보고자 하였다. 이를 위해 2020년 노인실태조사에 참여한 만 65세 이상 노인 8,732명을 대상으로 SPSS 23.0과 PROCESS macro를 활용하여 이중매개효과를 검증하였다. 주요 분석 결과는 다음과 같다. 첫째, 노인의 정보화 기기 이용 불편감이 삶의 만족도에 부적인 직접 효과를 보였다. 둘째, 사회적 존중감은 노인의 정보화 기기 이용 불편감과 삶의 만족도 간의 관계를 매개하는 것으로 나타났다. 셋째, 차별 경험은 노인의 정보화 기기 이용 불편감과 삶의 만족도 간의 관계를 매개하는 것으로 나타났다. 넷째, 사회적 존중감과 차별 경험은 노인의 정보화 기기 이용 불편감과 삶의 만족도 간의 관계를 이중매개하는 것으로 나타났다. 본 연구의 결과를 통해 사회적 존중감과 차별 경험의 메커니즘을 확인하였으며, 정보화 시대에서 삶의 만족도를 증진하기 위해 디지털 역량 강화를 통한 사회적 존중감 증가 및 차별경험 경감을 위한 실천적 노력의 필요성을 제시하였다. This study attempted to examine whether perceived social respect and discrimination experiences mediate the effect of the older adults’ discomfort in using digital devices on life satisfaction. To this end, the multi-mediated effect was verified using SPSS 23.0 and PROCESS macro in 8, 732 older adults aged 65 or older who participated in the Survey on Korean older adults’ status in 2020. The main analysis results are as follows. First, the older adults’ discomfort in using digital devices showed a positive direct effect on life satisfaction. Second, perceived social respect was found to mediate the relationship between the older adults’ inconvenience of using digital devices and life satisfaction. Third, the experience of discrimination was found to mediate the relationship between the older adults’ inconvenience of using digital devices and life satisfaction. Fourth, the effect of perceived social respect and discriminatory experience was found to mediate the relationship between the older adults’ inconvenience of using digital devices and life satisfaction. Through the results of this study, the mechanism of perceived social respect and discrimination experience was confirmed, and practical efforts were presented to increase social respect and reduce discrimination experience by strengthening digital capabilities to improve life satisfaction in the information age.
중노년기의 수면잠재기(sleep onset latency)와 인지기능 간의 연관성 연구
송시영,강현서,김율리,이현정,이은 한국노년학회 2019 한국노년학회 학술발표논문집 Vol.2019 No.2
Ⅰ. 연구배경 및 목적 Ⅱ. 연구방법 Ⅲ. 연구 결과 Ⅳ. 논의
송시영 대한소화기학회 2008 대한소화기학회지 Vol.51 No.2
Pancreatic cancer is currently the major leading cause of cancer-related deaths in the Western countries with an overall 5-year survival rate of less than 3%. The key aim of investigation is to identify the cellular population in which some of the earliest molecular events occur, presumably the ultimate target for carcinogenic insult. Advances in pathological classification and genetics have improved our descriptive understanding of this disease. However, important aspects of pancreatic cancer biology remain poorly understood. Factors associated with the increased risk of pancreatic cancer include smoking, chronic pancreatitis, diabetes, prior gastric surgery, and exposure to radiation or chemicals. A number of syndromes have been identified with the increased incidence of pancreatic cancer, including familial atypical multiple-mole melanoma syndrome, hereditary nonpolyposis colorectal cancer, and hereditary pancreatitis, etc. Recently, there have been growing evidences that stem cell biology could provide new insights into the understanding of cancer biology. Three postulates regarding the relationship between stem and tumor cells have been proposed. First, the similarities in the mechanims that regulate self-renewal of normal stem cells and cancer cells. Second, the possibility that tumor cells might arise from normal stem cells and third, the notion that tumors might contain ‘cancer stem cells' - rare cells with indefinite proliferative potential which drive the formation and growth of tumors. New insights for the cancer stem cells and their possible markers in pancreatic cancer have been suggested recently. Further observations of molecular and cellular events in the early stage of pancreatic carcinogenesis may have important implications regarding the cellular lineage responsible for pancreatic ductal metaplasia and neoplasia, and provide further support for the presence of stem cell capabilities within mature pancreatic epithelium. (Korean J Gastroenterol 2008;51:71-83)