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- 저자 : Xin-Teng Wu (검색결과 3 건)
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Iterative tuning of decoupling controller for 3-DOF precision motion stage
Wei Teng,Qingfeng Gao,Xin Wu,Haihua Mu 대한기계학회 2012 JOURNAL OF MECHANICAL SCIENCE AND TECHNOLOGY Vol.26 No.2
A practical iterative tuning algorithm of a decoupling controller is presented to eliminate intrinsic coupling among 3-DOF precision motion stage. General decoupling control cannot eliminate coupling completely, which will prevent the enhancement of control accuracy in high-precision motion system. The proposed algorithm can be used to tune the parameters of a decoupling controller iteratively through minimizing a quadratic cost function of the tracking error in non-movement direction when the stage moves in one direction. The tuning algorithm addressed for the motion stage needs only measurement signals in an actual motion system rather than a detailed model of the stage. The proposed algorithm is demonstrated by experimental results. It can be applied further in other multi-DOF motion systems.
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Recent Advances of 2D Nanomaterials in the electrode materials of Lithium-Ion Batteries
Xiaobei Zang,Teng Wang,Zhiyuan Han,Lingtong Li,Xin Wu 성균관대학교(자연과학캠퍼스) 성균나노과학기술원 2019 NANO Vol.14 No.2
The upcoming energy crisis and the increasing power requirements of electronic devices have drawn enormous attention to research in the field of energy storage. Owing to compelling electrochemical and mechanical properties, two-dimensional nanomaterials can be used as electrodes on lithium-ion batteries to obtain high capacity and long cycle life. This review summarized the recent advances in the application of 2D nanomaterials on the electrode materials of lithium-ion batteries.
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Cheng Chen,Song Hu,Heng-Jing Hu,Zhi-Xuan Liu,Xin-Teng Wu,Tao Zou,Hua Su 대한심장학회 2024 Korean Circulation Journal Vol.54 No.4
Background and Objectives: Long-term pathological myocardial hypertrophy (MH) seriously affects the normal function of the heart. Dronedarone was reported to attenuate left ventricular hypertrophy of mice. However, the molecular regulatory mechanism of dronedarone in MH is unclear. Methods: Angiotensin II (Ang II) was used to induce cell hypertrophy of H9C2 cells. Transverse aortic constriction (TAC) surgery was performed to establish a rat model of MH. Cell size was evaluated using crystal violet staining and rhodamine phalloidin staining. Reverse transcription quantitative polymerase chain reaction and western blot were performed to detect the mRNA and protein expressions of genes. JASPAR and luciferase activity were conducted to predict and validate interaction between forkhead box O3 (FOXO3) and protein kinase inhibitor alpha (PKIA) promoter. Results: Ang II treatment induced cell hypertrophy and inhibited sirtuin 1 (SIRT1) expression, which were reversed by dronedarone. SIRT1 overexpression or PKIA overexpression enhanced dronedarone-mediated suppression of cell hypertrophy in Ang II-induced H9C2 cells. Mechanistically, SIRT1 elevated FOXO3 expression through SIRT1-mediated deacetylation of FOXO3 and FOXO3 upregulated PKIA expression through interacting with PKIA promoter. Moreover, SIRT1 silencing compromised dronedarone-mediated suppression of cell hypertrophy, while PKIA upregulation abolished the influences of SIRT1 silencing. More importantly, dronedarone improved TAC surgery-induced MH and impairment of cardiac function of rats via affecting SIRT1/FOXO3/PKIA axis. Conclusions: Dronedarone alleviated MH through mediating SIRT1/FOXO3/PKIA axis, which provide more evidences for dronedarone against MH.
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