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- 저자 : Tavernier, Jan (검색결과 4 건)
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Chloé Tavernier,Sohail Ahmed,Katherine Albro Houpt,Seong Chan Yeon 대한수의학회 2020 Journal of Veterinary Science Vol.21 No.1
Cat vocalizes to communicate with another and express their internal states. The vocal repertoire of the cat is wide and up to 21 different vocalizations have been described in the literatures. But it is more than probable that the repertoire contains more types of vocalizations. An ethogram was created in this paper describing the actual known vocalisations of the domestic cat based on an auditory classification. However, the audiogram allows also a visual classification which can increase the accuracy of vocalization differentiation. The classification can be risky as it is sometimes unclear if different types of vocalizations are produced in different environments or if a unique type of vocalization is used with variation in the acoustic parameters. As an example, isolation calls produced by kittens differ depending on the context. The environment has an important impact on the vocal behaviour and thus feral cats and pet cats vocalize differently. Pet cats are thus able to create an efficient communication with humans thanks to the flexibility of vocalisation behaviours. This review allowed us to create a simple model of the cat vocal repertory.
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Agile Management and Interoperability Testing of SDN/NFV-Enriched 5G Core Networks
최태상,김태연,Wouter Tavernier,Aki Korvala,Jussi Pajunpää 한국전자통신연구원 2018 ETRI Journal Vol.40 No.1
In the fifth generation (5G) era, the radio internet protocol capacity is expected to reach 20 Gb/s per sector, and ultralarge content traffic will travel across a faster wireless/wireline access network and packet core network. Moreover, the massive and mission-critical Internet of Things is the main differentiator of 5G services. These types of real-time and large-bandwidth-consuming services require a radio latency of less than 1 ms and an end-to-end latency of less than a few milliseconds. By distributing 5G core nodes closer to cell sites, the backhaul traffic volume and latency can be significantly reduced by having mobile devices download content immediately from a closer content server. In this paper, we propose a novel solution based on software-defined network and network function virtualization technologies in order to achieve agile management of 5G core network functionalities with a proof-of-concept implementation targeted for the PyeongChang Winter Olympics and describe the results of interoperability testing experiences between two core networks.
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De Bosscher, Karolien,Beck, Ilse M.,Dejager, Lien,Bougarne, Nadia,Gaigneaux, Anthoula,Chateauvieux, Sé,bastien,Ratman, Dariusz,Bracke, Marc,Tavernier, Jan,Vanden Berghe, Wim,Libert, Claude,Diede Springer Basel 2014 Cellular and molecular life sciences Vol.71 No.1
<P>Glucocorticoids (GCs) block inflammation via interference of the liganded glucocorticoid receptor (GR) with the activity of pro-inflammatory transcription factors NF-κB and AP-1, a mechanism known as transrepression. This mechanism is believed to involve the activity of GR monomers. Here, we explored how the GR monomer-favoring Compound A (CpdA) affects AP-1 activation and activity. Our results demonstrate that non-steroidal CpdA, unlike classic steroidal GCs, blocks NF-κB- but not AP-1-driven gene expression. CpdA rather sustains AP-1-driven gene expression, a result which could mechanistically be explained by the failure of CpdA to block upstream JNK kinase activation and concomitantly also phosphorylation of c-Jun. In concordance and in contrast to DEX, CpdA maintained the expression of the activated AP-1 target gene <I>c</I>-<I>jun</I>, as well as the production of the c-Jun protein. As for the underlying mechanism, GR is a necessary intermediate in the CpdA-mediated gene expression of AP-1-regulated genes, but seems to be superfluous to CpdA-mediated JNK phosphorylation prolongation. The latter phenomenon concurs with the inability of CpdA to stimulate DUSP1 gene expression. ChIP analysis demonstrates that DEX-activated GR, but not CpdA-activated GR, is recruited to AP-1-driven promoters. Furthermore, in mice we observed that CpdA instigates a strong enhancement of TNF-induced AP-1-driven gene expression. Finally, we demonstrate that this phenomenon coincides with an increased sensitivity towards TNF lethality, and implicate again a role for JNK2. In conclusion, our data support the hypothesis that a ligand-induced differential conformation of GR yields a different transcription factor cross-talk profile.</P><P><B>Electronic supplementary material</B></P><P>The online version of this article (doi:10.1007/s00018-013-1367-4) contains supplementary material, which is available to authorized users.</P>
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