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Shulong Tan,Hua Geng,Geng Yang 전력전자학회 2016 JOURNAL OF POWER ELECTRONICS Vol.16 No.6
This paper presents an impedance-matching-based control scheme for the harmonic resonance damping of multiple grid-connected-converters (GCCs) with LCL filters. As indicated in this paper, harmonic resonance occurs if a GCC possesses an output impedance that is not matched with the rest of the network in some specific frequency bands. It is also revealed that the resonance frequency is associated with the number of GCCs, the grid impedance and even the capacitive loads. By controlling the grid-side current instead of the converter-side current, the critical LCL filter is restricted as an internal component. Thus, the closed-loop output impedance of the GCC within the filter can be configured. The proposed scheme actively regulates the output impedance of the GCC to match the impedance of the external network, based on the detected resonance frequency. As a result, the resonance risk of multiple GCCs can be avoided, which is beneficial for the plug-and-play property of the GCCs in microgrids. Simulation and experimental results validate the effectiveness of the proposed method.
Tan, Shulong,Geng, Hua,Yang, Geng The Korean Institute of Power Electronics 2016 JOURNAL OF POWER ELECTRONICS Vol.16 No.6
This paper presents an impedance-matching-based control scheme for the harmonic resonance damping of multiple grid-connected-converters (GCCs) with LCL filters. As indicated in this paper, harmonic resonance occurs if a GCC possesses an output impedance that is not matched with the rest of the network in some specific frequency bands. It is also revealed that the resonance frequency is associated with the number of GCCs, the grid impedance and even the capacitive loads. By controlling the grid-side current instead of the converter-side current, the critical LCL filter is restricted as an internal component. Thus, the closed-loop output impedance of the GCC within the filter can be configured. The proposed scheme actively regulates the output impedance of the GCC to match the impedance of the external network, based on the detected resonance frequency. As a result, the resonance risk of multiple GCCs can be avoided, which is beneficial for the plug-and-play property of the GCCs in microgrids. Simulation and experimental results validate the effectiveness of the proposed method.
Ma, Yuan,Eun, Jae-Soon,Yang, Shulong,Lee, Kwang-Seung,Lee, Eun-Sil,Kim, Chung-Soo,Oh, Ki-Wan The Korean Society of Ginseng 2010 Journal of Ginseng Research Vol.34 No.1
The present investigation was conducted to evaluate the regulation of sleep architecture by the red ginseng water extract (RGE) in acutely and chronically restraint stressed rats. Adult rats were fitted with sleep.wake recording electrodes. Following post-surgical recovery, rats were extensively habituated for freely moving polygraphic recording conditions. Polygraphic signs of sleep-wake activities were recorded for 24 h after RGE administration and induction of stress and were analyzed to understand the regulation of sleep architecture. Acute stress decreased wakefulness and increased total sleep, non-rapid eye movement (NREM) sleep, and rapid eye movement (REM) sleep in both the daytime and nighttime recording. RGE shortened the daytime NREM and REM sleep, without changing the wakefulness and total sleep. RGE increased nighttime wakefulness, and decreased total, NREM and REM sleep. Chronic stress increased wakefulness and decreased total sleep in the daytime recording, and increased REM and decreased NREM sleep in both the day and night time recording. RGE ameliorated chronic stress and induced alterations of REM and NREM sleep in the day and night time sleep architecture. Acute and chronic stress could also induce alternations in cortex electroencephalogram (EEG) recording during NREM, REM sleep and wakefulness. These findings suggest that RGE may modulate the sleep behavior in acutely and chronically stressed rats and the ameliorating effect of RGE on the sleep architecture may involve in modulation of $\alpha$-, $\theta$- and $\delta$- wave activities of the cortical EEG.
Yuan MA,Jae Soon EUN,Shulong YANG,Kwang Seung LEE,Eun-Sil LEE,Chung-Soo Kim,Ki-Wan OH 고려인삼학회 2010 Journal of Ginseng Research Vol.34 No.1
The present investigation was conducted to evaluate the regulation of sleep architecture by the red ginseng water extract (RGE) in acutely and chronically restraint stressed rats. Adult rats were fitted with sleep?wake recording electrodes. Following post-surgical recovery, rats were extensively habituated for freely moving polygraphic recording conditions. Polygraphic signs of sleep-wake activities were recorded for 24 h after RGE administration and induction of stress and were analyzed to understand the regulation of sleep architecture. Acute stress decreased wakefulness and increased total sleep, non-rapid eye movement (NREM) sleep, and rapid eye movement (REM) sleep in both the daytime and nighttime recording. RGE shortened the daytime NREM and REM sleep, without changing the wakefulness and total sleep. RGE increased nighttime wakefulness, and decreased total, NREM and REM sleep. Chronic stress increased wakefulness and decreased total sleep in the daytime recording, and increased REM and decreased NREM sleep in both the day and night time recording. RGE ameliorated chronic stress and induced alterations of REM and NREM sleep in the day and night time sleep architecture. Acute and chronic stress could also induce alternations in cortex electroencephalogram (EEG) recording during NREM, REM sleep and wakefulness. These findings suggest that RGE may modulate the sleep behavior in acutely and chronically stressed rats and the ameliorating effect of RGE on the sleep architecture may involve in modulation of α-, θ- and δ- wave activities of the cortical EEG.
Sanjoinine A Isolated from Semen Zizyphi Spinosi Protects Against Kainic Acid-Induced Convulsions
Sung-Ryul Yoon,Young-Jun Jo,Shulong Yang,김윤배,남상윤,김형춘,홍진태,오기완 대한약학회 2009 Archives of Pharmacal Research Vol.32 No.11
These experiments were performed to know whether sanjoinine A, a component of the alkaloid fraction of Semen Zizyphi Spinosi, acts as an anti-convulsive agent in the kainic acid (KA)-induced experimental convulsion model and whether these effects are mediated by decreased intracellular calcium. Oral administration of sanjoinine A (4 and 8 mg/kg) increased the survival rate and latency of convulsion onset, and decreased the seizure scores and the weight loss induced by intraperitoneal (i.p.) injection of KA (50 mg/kg) in mice. In addition, sanjoinine A protected against neuronal damage and apoptosis in the hippocampus after KA administration, as analyzed by using immunohistochemistry and TUNEL assay. Sanjoinine A also significantly blocked seizure-form electroencephalogram alterations induced by KA. Moreover, in cultured rat neuronal cells, sanjoinine A inhibited KA-induced cell death, as measured by propidium iodide detection. Sanjoinine A also increased intracellular chloride and inhibited the elevation of intracellular calcium induced by KA. Sanjoinine A, therefore protects against KAinduced convulsions by increasing intracellular chloride and reducing intracellular calcium levels.