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RISS 인기검색어
- 검색키워드
- 저자 : Salpietro, E. (검색결과 4 건)
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Results of a New Generation of ITER TF Conductor Samples in SULTAN
Bruzzone, P.,Stepanov, B.,Wesche, R.,Salpietro, E.,Vostner, A.,Okuno, K.,Isono, T.,Takahashi, Y.,Hyoung Chan Kim,Keeman Kim,Shikov, A.K.,Sytnikov, V.E. IEEE 2008 IEEE transactions on applied superconductivity Vol.18 No.2
<P>A new generation of ITER TF conductor samples has been assembled and tested in SULTAN in 2007 following a common procedure agreed among the ITER parties. The test results of six SULTAN samples, made of twelve conductor sections manufactured in Europe, Japan, Korea and Russia, are reported here. The conductor layout reflects the ITER TF conductor design, with minor differences for the Nb<SUB>3</SUB>Sn strand characteristics, void fraction and twist pitch. The object of the test is a straight comparison with the ITER requirement of 5.7 K current sharing temperature at 68 kA current and 11.3 T field. A broad range of behavior is observed.</P>
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Recent Developments in United Airways Disease
Giorgio Ciprandi,Davide Caimmi,Michele Miraglia del Giudice,Mario La Rosa,Carmelo Salpietro,Gian Luigi Marseglia 대한천식알레르기학회 2012 Allergy, Asthma & Immunology Research Vol.4 No.4
The nose and lung are both part of the respiratory tract. Often the diseases affecting the nose and/or the bronchi are treated separately. However, in recent years, numerous studies have highlighted the fact that the respiratory system is a single entity and the concept of “united airway disease”has become more and more important. The unity of the respiratory tract is confirmed both from a morphological and from a functional point of view. Nevertheless, this concept is also confirmed for the respiratory immune system, innervation and vascularization interesting all along the tract, from the nose to the bronchioles. When treating rhinitis, it is often necessary to assess the presence of asthma. Patients with sinusitis should be evaluated for a possible concomitant asthma. Conversely, patients with asthma should always be evaluated for possible nasal disease. The medications that treat nasal diseases appear to be useful in improving control of asthma and in reducing bronchial hyperresponsiveness as well. Physicians should always keep these notions in mind, and evaluate and treat respiratory diseases taking into account the unity of the respiratory tract.
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Preparation of the ITER poloidal field conductor insert (PFCI) test
Zanino, R.,Egorov, S.,Kim, K.,Martovetsky, N.,Nunoya, Y.,Okuno, K.,Salpietro, E.,Sborchia, C.,Takahashi, Y.,Weng, P.,Bagnasco, M.,Richard, L.S.,Polak, M.,Formisano, A.,Zapretilina, E.,Shikov, A.,Veder IEEE 2005 IEEE transactions on applied superconductivity Vol.15 No.2
The Poloidal Field Conductor Insert (PFCI) of the International Thermonuclear Experimental Reactor (ITER) has been designed in the EU and is being manufactured at Tesla Engineering, UK, in the frame of a Task Agreement with the ITER International Team. Completion of the PFCI is expected at the beginning of 2005. Then, the coil shall be shipped to JAERI Naka, Japan, and inserted into the bore of the ITER Central Solenoid Model Coil, where it should be tested in 2005 to 2006. The PFCI consists of a NbTi dual-channel conductor, almost identical to the ITER PF1 and PF6 design, ∼45 m long, with a 50 mm thick square stainless steel jacket, wound in a single-layer solenoid. It should carry up to 50 kA in a field of ∼6 T, and it will be cooled by supercritical He at ∼4.5 K and ∼0.6 MPa. An intermediate joint, representative of the ITER PF joints and located at relatively high field, will be an important new item in the test configuration with respect to the previous ITER Insert Coils. The PFCI will be fully instrumented with inductive and resistive heaters, as well as with voltage taps, Hall probes, pick-up coils, temperature sensors, pressure gauges, strain and displacement sensors. The test program will be aimed at DC and pulsed performance assessment of conductor and intermediate joint, AC loss measurement, stability and quench propagation, thermal-hydraulic characterization. Here we give an overview of the preparatory work toward the test, including a review of the coil manufacturing and of the available instrumentation, a discussion of the most likely test program items, and a presentation of the supporting modeling and characterization work performed so far.
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Leonardo Cavone,Caterina Cuppari,Sara Manti,Luisa Grasso,Teresa Arrigo,Luca Calamai,Carmelo Salpietro,Alberto Chiarugi 대한이비인후과학회 2015 Clinical and Experimental Otorhinolaryngology Vol.8 No.2
Objectives. The nuclear protein high mobility group protein box 1 (HMGB1) is a proinflammatory mediator that belongs to the alarmin family of proinflammatory mediators, and it has recently emerged as a key player in different acute and chronic immune disorders. Several lines of evidence demonstrate that HMGB1 is actively released extracellularly from immune cells or passively released from necrotic cells. Because of the ability of HMGB1 to sustain chronic inflammation, we investigated whether the protein is present in nasal fluids of patients with different forms of rhinitis. Methods. HMGB1 levels were evaluated in nasal fluids of healthy subjects or rhinitis patients who were treated or not treated with different treatments. Results. We report that the level of HMGB1 was significantly increased in nasal fluids of patients with allergic rhinitis, patients with NARES (nonallergic rhinitis with eosinophiliac syndrome), as well as patients with polyps. We also found that a formulation containing the HMGB1-binding compound glycyrrhizin (GLT) reduced the HMGB1 content in nasal fluids of rhinitis patients to an extent similar to that with nasal budesonide treatment. We also found that among the cultured human leukocyte populations, eosinophils released higher amounts of HMGB1. Based on the ability of HMGB1 to sustain eosinophil survival and the ability of GLT to inactivate HMGB1, we report that GLT selectively killed cultured eosinophils and had no effect on neutrophils, macrophages, and lymphocytes. Conclusion. Collectively, these data underscore the role of HMGB1 in rhinitis pathogenesis and the therapeutic potential of GLT formulations in treatment of chronic inflammatory disorders of the nasal mucosa.
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