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Final report on force key comparison CCM.F-K2.a and CCM.F-K2.b (50 kN and 100 kN)
Vincke, William,Zhimin, Zhang,Pusa, Aimo,Averlant, Philippe,Kumme, Rolf,Germak, Alessandro,Ueda, Kazunaga,Park, Yon-Kyu,Torres, Jorge,Burke, Ben,Langmead, Fredrik,Fank, Sinan,Knott, Andy,Bartel, Tom Springer-Verlag 2012 METROLOGIA -BERLIN- Vol.49 No.-
<P>This report describes CIPM key comparison CCM.F-K2, a comparison between the deadweight force standard machines of fourteen National Measurement Institutes, at generated forces of 50 kN and 100 kN, in the period from 2004 to 2007. Two different measurement schemes were employed, one for machines capable of generating both 50 kN and 100 kN and the other using the single force of 50 kN, for machines of a lower maximum capacity than 100 kN. Multiple transducers were used and the force–time profile was strictly controlled, to minimize effects of creep. Analysis of the results took careful account of the drift of the transducers' sensitivities throughout the comparison period, as this was one of the major uncertainty contributions. The final results suggest that the nominal 50 kN forces generated at four of the fourteen laboratories (and the 100 kN forces at two of them) may be statistically significantly different from the same nominal forces generated at the other laboratories.</P><P>Main text.To reach the main text of this paper, click on Final Report. Note that this text is that which appears in Appendix B of the BIPM key comparison database kcdb.bipm.org/.</P><P>The final report has been peer-reviewed and approved for publication by the CCM, according to the provisions of the CIPM Mutual Recognition Arrangement (CIPM MRA).</P>
Raluca, Balica Amalia,Cimpean, Anca Maria,Cioca, Andreea,Cretu, Octavian,Mederle, Ovidiu,Ciolofan, Alexandru,Gaje, Pusa,Raica, Marius Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.11
Background: Colorectal carcinoma (CRC) is one of the major causes of cancer death worldwide. Data from the literature indicate differences between the proliferation rate of endothelial cells relative to the morphology growth type, possibly due to origin of specimens (autopsy material, surgery fragments) or quantification methods. Vascular endothelial growth factor (VEGF) is a factor that stimulates the proliferation of endothelial cells. It is expressed in more than 90% of cases of metastatic CRC. Aim: The aim of this study was to evaluate the endothelial cell proliferation and VEGF expression in primary tumors and corresponding liver metastases. Materials and Methods: Our study included 24 recent biopsies of primary tumors and corresponding liver metastases of CRC cases. CD34/Ki67 double immunostaining and RNA scope assay for VEGF were performed. Results: In the primary tumors analysis of VEGFmRNA expression indicated no significant correlation with differentiation grade, proliferative and non-proliferative vessels in the intratumoral and peritumoral areas. In contrast, in the corresponding liver metastases, VEGFmRNA expression significantly correlated with the total number of non-proliferative vessels and total number of vessels. CD34/Ki67 double immunostaining in the cases with poorly differentiated carcinoma indicated a high number of proliferating endothelial cells in the peritumoral area and a low number in the intratumoral area for the primary tumor. Moderately differentiated carcinomas of colon showed no proliferating endothelial cells in the intratumoral area in half of the cases included in the study, for both, primary tumor and liver metastasis. In well differentiated CRCs, in primary tumors, a high proliferation rate of endothelial cells in the intratumoral area and a lower proliferation rate in the peritumoral area were found. A low value was found in corresponding liver metastasis. Conclusions: The absence of proliferative endothelial cells in half of the cases for the primary tumors and liver metastases in moderately differentiated carcinoma suggest a vascular mimicry phenomenon. The mismatch between the total number of vessels and endothelial proliferation in primary tumors indicate that a functional vascular network is already formed or the existence of some mechanisms influenced by other angiogenic factors.