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연구보고 : 천식 환자의 기관지 조직에서 Eotaxin mRNA 발현에 관한 연구
인광호 ( Kwang Ho In ),조재연 ( Jae Yun Cho ),강세용 ( Sae Yong Kang ),이상엽 ( Sang Youb Lee ),심재정 ( Jae Jeong Shim ),강경호 ( Kyung Ho Kang ),유세화 ( Se Hwa Yoo ),나영순 ( Young Soon Na ),김한겸 ( Han Gyum Kim ) 대한결핵 및 호흡기학회 1998 Tuberculosis and Respiratory Diseases Vol.45 No.4
폐결핵 환자에서의 발열의 빈도 및 화학치료에 대한 반응
인광호(Kwang Ho In),조재연(Jae Yon Cho),강경호(Kyung Ho Kang),유세화(Se Hwa Yoo) 대한내과학회 1991 대한내과학회지 Vol.40 No.2
N/A Fever is often used clinically to judge the severity of pulmonary tuberculosis and to evaluate its response to therapy. Sometimes fever persists for a long time even after starting chemotherapy. Records of 101 hospitalized patients with pulmonary tuberculosis were reviewed to investigate the incidence of fever, fever response to modern chemotherapy and analyzed possible factors affecting fever response in pulmonary tuberculosis. The results were as follows: 1) sixty-eight patients among 101 (69%) were febrile on admission. 2) There were no significant differences between febrile and afebrile groups in the extent of tuberculous lesion, presence of symptoms, and sputum smear positivity of acid-fast bacilli. 3) In febrile patients, the duration of fever after initiation of chemotherapy was short (mean, 5 days; range, 1 to 74 days). 4) In febrile patients there were no significant differences between early responders (<1 week) and the late responders (>1 week) in the extent of lesion, presence of symptoms, and smear positivity of acid-fast bacilli. In conclusion, the incidence of fever in pulmonary tuberculosis is high among admitted patients and fever subside after a few days of treatment in most cases, but sometimes it may persist much longer time.
기관내삽관과 기관절개술을 시행한 환자에서의 폐흡인에 관한 연구
인광호(Kwang Ho In),안태훈(Tae Hoon Ahn),유진목(Zin Moc Yu),강경호(Kyung Ho Kang),유세화(Se Hwa Yoo) 대한내과학회 1988 대한내과학회지 Vol.35 No.3
N/A Aspiration is a well recognized source of pulmonary infection. In most clinical situations, however, the actual incidence of aspiration has not been well documented, We investigated the incidence of pulmonary aspiration and the factors affecting aspiration in patients with intubation and tracheostomy. Aspiration was evaluated by applying Evans blue dye to the posterior tongue just after each formula tube feeding for 24 hours. Bluish discoloration and elevated glucose concentration of the material suctioned from the artificial airway were regarded as evidence of aspiration. The results obtained were as follows: 1) The incidence of pulmonary aspiration in patients with intubation or tracheostomy was high (69%), 2) Unconscious patients tended to aspirate frequently, but the presence or absence of ventilatory support, tracheostomy or intubation did not play a significant role in the development of aspiration. 3) There was only a weak correlation between the Evans blue dye test and the glucose oxidase reagent strip method in detecting pulnonary aspiration. In conclusion, the incidence of pulmonary aspiration in patients with intubation and tracheostomy is very high. These findings suggest that even in the case of intubation and tracheostomy, careful observation of pulmonary aspiration is necessary
실험견의 급성 저 산소성 폐고혈압중에서 내피세포 기원 확장인자 ( EDRF ) 의 역할
김인옥(In Ohk Kim),인광호(Kwang Ho In),조재연(Jae Yun Cho),이진구(Jin Goo Lee),심재정(Jae Jung Shim),강경호(Kyung Ho Kang),유세화(Se Hwa Yoo) 대한내과학회 1994 대한내과학회지 Vol.47 No.6
N/A Objectives : Hypoxic pulmonary vasoconstriction has been well documented in the pulmonary cirulation. However the mechanism and the modulating factors of the hypoxic pulmonary vasoconstriction remain unclarified. With the recent evidences that the endothelium-de- rived relaxing factor (EDRF) is an important mediator of vascular tone, there have been increasing interests in defining the role of the EDRF as a potential mediator of hypoxic pulmonary vasoconstriction. But the results of studies on the role of EDRF during hypoxic pulmonary vasoconstriction have been inconsistent, and the studies in the intact animals are very rare. To investigate the role of EDRF during pulmonary hypertension induced by hypoxia, we measured changes of hemodynamic parameters after adding NO synthesis inhibitor and its substrate. Methods : Five dogs were anesthesized with thiopental sodium. After intubation with an endotracheal tube, the dogs were mechanically ventilated with a Harvard volume-cycled animal ventilator. Venous and arterial catheters were placed in the limb vein and femoral artery. Swan-Ganz catheter was inserted via right internal jugular vein for measuring pulmonary arterial pressure and pulmonary capillary wedge pressure, and cardiac output. We measured the changes of hemodynamic parameters in normoxia and hypoxia and the effects of L-NNA (30 mg/kg) and L-arginine (200 mg/kg). Results : 1) Hypoxia caused a significant increase in pulmonary arterial pressure. 2) The infusion of L-NNA in normoxic state did not change any of the hemodynamic variables. 3) With the infusion of L-NNA, the increase in pulmonary arterial pressure produced by hypoxia were significantly augmented. 4) With the infusion of L-arginine, hypoxic pulmonary hypertension augmented by L-NNA was significantly reversed, Conclusion : L-NNA did not affect the pulmonary arterial pressure in normoxic state, but augmented the pulmonary hypertension during hypoxia and L-arginine reversed the hypoxic pulmonary hypertension almost to control level. It is suggested that diminished EDRF formation contributes greatly to the pulmonary hypertension induced by hypoxia.