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Zhiwei Gao,Desheng Li,Zongqiang Liu,Lezhi Ye 한국자동차공학회 2022 International journal of automotive technology Vol.23 No.5
The retarder is widely used to ensure braking safety in heavy vehicles due to its noncontact braking characteristics. Currently, retarders are generally mounted on tractors, but semi-trailers have greater inertia and are prone to accidents. Therefore, the semi-trailer’s brakes have to work frequently to maintain stability during downhill braking, which causes the brakes to generate high temperatures or even failure. Based on the above problems, a novel internal pump-hydraulic retarder axle (IP-HRA) is proposed, which integrates the pump and hydraulic retarder (HR) into the support axle. The prototype of the IP-HRA was made and a bench test was conducted. In the experiment, the single-side braking power of the IP-HRA reaches 300 kW and its torque can be controlled by adjusting the outlet pressure. A co-simulation model of articulated vehicles is established to predict the braking state by computer solutions. Based on the experimental data, the methods of IP-HRA braking semi-trailer and traditional HR braking tractor are compared. The results of co-simulation show that the IP-HRA is more conducive to the braking stability of the articulated vehicle than the traditional HR when only retarder is used. Finally, the braking time and braking deceleration of IP-HRA are verified by road experiments.
Wenguang Guo,Desheng Li,Lezhi Ye,Zhiwei Gao,Kai Zhang 한국자동차공학회 2019 International journal of automotive technology Vol.20 No.5
To overcome the large power consumption, the braking torque heat recession, and installation difficulties for trailers of eddy current retarder (ECR), a novel self-excited, liquid-cooled, and bridge integrated retarder (SLB-EMR) is proposed in this paper. The structure and work principle of the SLB-EMR are described particularly. Based on the magnetic equivalent circuit (MEC) method, an analytical model of the eddy current braking torque considering magnetic flux leakage and end effect is established. The power generation and braking performance of the SLB-EMR are predicted by the finite element analysis (FEA). We carried out tests for the eddy current braking torque, the heat-fade of braking torque, the no-load loss torque, and natural characteristics of the SLB-EMR respectively. The test results showed that the eddy current braking torque reached 2592 N·m at 1000 r/min. The braking torque declined by 15.5 % after the braking 12 min continuously. The analytical model of eddy current braking torque, and FEA model of the generator and eddy current brake were verified by the test. Compared with the ECR, the SLB-EMR had no-power consumption and low head-fade.
Yang Sheng,Xie JiaJun,Pan ZhiJie,Guan HongMei,Tu YueSheng,Ye YuanJian,Huang ShouBin,Fu ShiQiang,Li KangXian,Huang ZhiWei,Li XiaoQi,Shi ZhanJun,Li Le,Zhang Yang 생화학분자생물학회 2024 Experimental and molecular medicine Vol.56 No.-
The meniscus is vital for maintaining knee homeostasis and function. Meniscal calcification is one of the earliest radiological indicators of knee osteoarthritis (KOA), and meniscal calcification is associated with alterations in biomechanical properties. Meniscal calcification originates from a biochemical process similar to vascular calcification. Advanced glycation end products (AGEs) and their receptors (RAGEs) reportedly play critical roles in vascular calcification. Herein, we investigated whether targeting AGE-RAGE is a potential treatment for meniscal calcification. In our study, we demonstrated that AGE-RAGE promotes the osteogenesis of meniscal cells and exacerbates meniscal calcification. Mechanistically, AGE-RAGE activates mTOR and simultaneously promotes ATF4 accumulation, thereby facilitating the ATF4-mTOR positive feedback loop that enhances the osteogenic capacity of meniscal cells. In this regard, mTOR inhibits ATF4 degradation by reducing its ubiquitination, while ATF4 activates mTOR by increasing arginine uptake. Our findings substantiate the unique role of AGE-RAGE in the meniscus and reveal the role of the ATF4-mTOR positive feedback loop during the osteogenesis of meniscal cells; these results provide potential therapeutic targets for KOA.