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Wei Li,Qian Liu,Guoyu Zhang,Xuedong Cheng,Yingfeng Wang,Zhenzhen Xu 한국섬유공학회 2022 Fibers and polymers Vol.23 No.8
This work aimed to evaluate the paste stability and desizability of new starch grafted copolymer [poly(sodium allylsulfonate)-g-starch-g-poly(ethyl acrylate)] (PSAS-g-starch-g-PEA) for providing an important supporting role in sizingapplication. The PSAS-g-starch-g-PEA samples were prepared by grafting reaction of acid-thinned starch (AHS) with SASand subsequent with EA in water phase. The PSAS-g-starch-g-PEA granules were characterized by Fourier transforminfrared spectroscopy and scanning electron microscopy. Also, paste stability was assessed and desizability was investigatedin terms of desizing efficiency, swelling ability of the film and time interval for the break of starch film in water. Comparedwith control AHS, the PSAS-g-starch-g-PEA had higher paste stability, desizing efficiency and swelling ability, and lowertime interval required, concluding that the PSAS and PEA branches could promote the paste stability and desizability ofstarch. Increasing the number of both branches facilitated the paste stability and desizability. PSAS-g-starch-g-PEA displayedpotential value in textile sizing field.
Shan Guang,Gu Juan,Zhou Daoping,Li Lingxun,Cheng Wei,Wang Yueping,Tang Tian,Wang Xuedong 생화학분자생물학회 2020 Experimental and molecular medicine Vol.52 No.-
Therapeutic failure in prostate cancer (PC) is believed to result from its unusually invasive and metastatic nature. Cancer-associated fibroblasts (CAFs) are essential in the tumor microenvironment. We intended to study the role of CAF-derived exosomes in the context of PC and the potential regulatory mechanism associated with miR-423-5p and GREM2. CAF-derived exosomes decreased the chemosensitivity of parental PC cells and enhanced the drug resistance of drug-resistant cells. PC-associated fibroblast-derived exosomes carrying miR-423-5p increased the resistance of PC to taxane by inhibiting GREM2 through the TGF-β pathway. Inhibition of the TGF-β pathway partially reversed the increased drug resistance in PC cells induced by CAF-derived exosomes. Inhibition of miR-423-5p enhanced the drug sensitivity of PC cells in vivo. We showed that CAF-secreted exosomal miR-423-5p promoted chemotherapy resistance in PC by targeting GREM2 through the TGF-β pathway. This study may allow the development of novel approaches for PC.