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        Mg-doped ZnO layer to enhance electron transporting for PbS quantum dot solar cells

        Xing Meibo,Wei Yuyao,Wang Dandan,Shen Qing,Wang Ruixiang 한국물리학회 2021 Current Applied Physics Vol.21 No.-

        In this work, the effect of Mg doping on the performance of PbS quantum dot (QD) solar cells (QDSCs) is investigated. To elucidate that, PbS QDSCs with pristine ZnO and Mg-doped ZnO (ZMO) as electron transporting layers (ETLs) are fabricated, respectively. The current density-voltage (J-V) measurements are performed. The results show that the cell efficiency of the device with ZMO as an ETL is 9.46%, which increases about 75% compared to that of the pristine ZnO based device (5.41%). Enhanced short current density (Jsc) and fill factor (FF) are observed. It is demonstrated that Mg doping could passivate the surface defects and suppress the carrier recombination in ZnO ETL, thus resulting in larger bandgap and higher Fermi level (EF). The strategy of Mgdoped ZnO ETL provides a promising way for pushing solar cell performance to a high level.

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        The effects of aging for improving wastewater sludge electro-dewatering performances

        Hang Lv,Daoguang Liu,Siqi Xing,Dandan Wu,Fang Wang,Jiakuan Yang,Xu Wu,Wenbiao Zhang,Xiaohu Da 한국공업화학회 2019 Journal of Industrial and Engineering Chemistry Vol.80 No.-

        The effects of sludge aging on improving sludge electro-dewatering (SED) performance were studied,with the sludge properties variation during aging process investigated. Sludge electro-dewateringperformances were explored by constant voltage method (25 V) on various samples. The organic matterin the extracellular polymeric substances (EPS) was gradually consumed and decomposed during sludgeaging process. Under identical electro-dewatering conditions, dewaterability and time-space yield ofaged sludge achieved 49.33% solid content and 223.46 kg m 2 h 1 respectively. Such results indicate thebeneficial effects of sludge aging process in terms of SED performances, which needs further explorationinto fundamental mechanisms.

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        Dexmedetomidine targets miR-146a and participates in the progress of chronic obstructive pulmonary disease in vivo and in vitro

        Li Na,Li Shuangfeng,Wu Yehua,Xiong Lu,Li Tiejun,Xing Dandan,Li Qiuchang,Wu Duozhi 한국유전학회 2021 Genes & Genomics Vol.43 No.12

        Background Chronic obstructive pulmonary disease (COPD) is a chronic lung disease and the third leading cause of death in the world. Dexmedetomidine has been reported to efectively inhibit histamine-induced bronchoconstriction. However, the molecular mechanism of dexmedetomidine in COPD has not been found. Objective To explore the role and mechanism of dexmedetomidine in COPD, and to provide theoretical basis for clinical treatment of COPD. Methods The expression of miR-146a was regulated by mimics or inhibitor and the relative expression of apoptotic proteins p53, Bax and Bcl-2 in human bronchial epithelial 16HBE cells was determined by real-time PCR and Western blot. Dexmedetomidine was treated for 16HBE cells and alveolar epithelial type II cells (AEC2), the cell apoptosis was detected by TUNEL and Hoechst33342 staining. A COPD rat model was established by smoking to test the efects of dexmedetomidine on the progression of COPD. The levels of IL-6, IL-1β and TNF-α in serum were measured by ELISA and the protein concentration of bronchoalveolar lavage fuid (BALF) was also detected in dexmedetomidine treated COPD rat model. Results miR-146a promoted 16HBE cell apoptosis and reduced cell proliferation. Additionally, dexmedetomidine was showed to reduce the 16HBEL cell apoptosis through reducing the expression of miR-146a. Moreover, dexmedetomidine regulated cell apoptosis and cell apoptosis through miR-146a in AEC2 cells. More importantly, dexmedetomidine attenuated the morphology and pathology of COPD rat model. Conclusion Dexmedetomidine reduced 16HBE cells and AEC2 cell apoptosis and attenuated COPD by down-regulating miR-146a.

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