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崔京求,李王休 全北大學校 1980 論文集 Vol.22 No.-
The present study was carried out to clarify the inducing factors of seed dormancy of 12 newly improved rice varieties by checking the effect of hull and germination inhibitors on seed germination of 30 day-old rice seeds. The obtained results were as follows; 1. Hulled or half-hulled rice seeds showed remarkably high percentage of germination and rapid germination speed. Therefore, the dormancy of immature rice seed seemed to be mainly due to its hull. 2. Soaking seeds in water at 30℃ had an effect on breaking dormancy and the effect tended to be decreased in inverse proportion to the degree of seed dormancy. 3. Through bioassay with lettuce and rice seed germination, the existence of germination inhibitors which leached into water from rice seeds was recognized. In general, high dormant rice varieties tended to have less water soluble inhibitors than low ones. 4. The inhibitors in rice seeds were detected in the zones of Rf 0.1-0.3 and Rf 0.8-1.0 even though the patterns of paper-chromatogram on inhibitors in seeds differed with varieties. 5. The amount of germination inhibitors in seeds was closely but not completely related with the degree of seed dormancy. Therefore, it may be reasonable to evaluate the degree of immature rice seed dormancy with the amount of inhibitors in the seed and that of leaching inhibitors from it. 6. In some varieties such as Iri 332, Milyang 23, and Milyand 29, the seed dormancy was also due to the physiological immature of embryo as well as the effect of hull and inhibitors. 7. Considering the above conditions, the tested 12 varieties may be grouped as follows; a. Low dormant varieties: Tongil, Josaengtongil, Suweon 264, Tongilchal, Milyang 21. b. Medium dormant varieties: Iri 326, Iri 327. c. High dormant varieties: Iri 332, Yushin, Milyang 23, Milyang 29, Milyang 30.
Lee, Yong-Hoon,Lee, Wang-Hyu,Lee, Du-Ku,Shim, Hyeong-Kwon The Korean Society of Plant Pathology 2001 Plant Pathology Journal Vol.17 No.3
The plant growth-promoting Pseudomonas strains, WR8-3 (Pseudomonas fluorescens), WR9-11 (Pseudomonas sp.) and WR9-16 (P.putida), which induced resistance systematically in watermelon to gummy stem rot were investigated on their induced systemic resistance(ISR)-related characteristics. The pyoverdine production was repressed in the standard succinate medium by increasing the concentration of $\textrm{FeCL}_3$. But the iron-binding ability on chrome azurol S agar media (CAS) was observed only in the strains, WR8-3 and WR9-16. When the two strains were mutated, the resulting iron-binding siderophore-negative mutants, WR8-3m and WR 9-16m, failed to promote the growth of watermelon and to induce resistance. The strains, WR8-3 and WR 9-16, slightly inhibited the growth of Didymella bryoniae at a low concentration of $\textrm{FeCL}_3$ on Kong's medium B, but not to exert control dffect. The strain WR9-11 showed antagonism in the concentration of $\textrm{FeCL}_3$ from 0 to $1,000\mu\textrm{M}$. When the crude lipoplysaccharide of each strain was treated in the rhizosphere of watermelon, mean lesion area was similar to that of the untreated control. The strains, WR9-11 and WR9-16 produced some level of hydrogen cyanide (HCN). Salicylic acid production was not detected in all of the strains.
Lee, Kui Jae,Park, Jong Chul,Lee, Young Hoon,Lee, Doo Ku,Choi, Min Kyung,Lee, Wang Hyu 전북대학교 농업과학기술연구소 2002 農大論文集 Vol.33 No.1
Barley yellow mosaic virus (BaYMV) and barley mild mosaic virus (BaMMV) bymoviruses, both transmitted in soil by the root-infecting fungus Polymyxa graminis, are responsible for the economicalally importan yellow mosaic disease of winter barley in East Asia and Europe (Huth et al., 1984; Huth and Adams, 1990; Kashiwazaki et al., 1998). They have a bipartite genome comprising two 3'-polyadenylated ssRNA molecules of 7.6kb(RNA 1) and 3.5-3.7kb (RNA2)(Huth et al. 1984; Kashiwazaki et al. 1989; Kashiwazaki and Ogawa; 1989). Key words: barley yellow mosaicirus, Bumovirus, nucleotide sequence, RNA 1, capsid protein.
Lee, Yong-Hoon,Lee, Wang-Hyu,Shim, Hyeong-Kwon,Lee, Du-Ku The Korean Society of Plant Pathology 2000 Plant Pathology Journal Vol.16 No.6
The selected five plant growth-promoting rhizobacteria (PGPR) strains, WR8-3 (Pseudomonas fluorescens), WR8-6 (P. putida), WR9-9 (P. fluorescens), WR9-11 (Pseudomonas sp.), and WR9-16 (P. putida) isolated in the rhizosphere of watermelon plants were tested on their growth promotion and control effect against gummy stem rot of watermelon. Strains, WR8-3 and WR9-16 significantly increased stem length of watermelon, and there was a little increase in leaf area, fresh weight and root length when strains, WR8-3, WR9-9 and WR9-16 were treated. Generally, seed treatment was better for plant growth promotion than the soil drench, but there was no significant difference. Seed treatment and soil drench of each bacterial strain also significantly reduced the mean lesion area (MLA) by gummy stem rot, but there was no significant difference between the two treatments. At initial inoculum densities of each strain ranging from 10$^6\;to\;10^{15}$ cfu/g seed, approximately the same level of disease resistance was induced. But resistance induction was not induced at the initial inoculum density of 10$^3$ cfu/g seed. Resistance was induced by treating the strains, WR9-9, WR9-11 and WR9-16, on all of four watermelon varieties tested, and there was no significant difference in the decrease of gummy stem rot among varieties. Populations of the strains treated initially at log 9-10 cfu/g seed, followed with a rapid decrease from planting day to 1 week after planting, but the population density was maintained above log 5.0 cfu/g soil until 4 weeks after planting. Generally no or very weak in vitro antagonism was observed at the strains treated excepting WR9-11. Rifampicin-resistant bacteria which had been inoculated were not detected in the stems or leaves, which suggesting that the bacterium and the pathogens remained spatially separated during the experiment. This is the first report of rsistance induction in watermelon to gummy stem rot by PGPR strains.