http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
On the Influence of Prosody in Processing Relative Clauses in Japanese
Satoru Muraoka,Toshio Matsuura,Tsutomu Sakamoto 서울대학교 인지과학연구소 2006 Journal of Cognitive Science Vol.7 No.2
The present study investigates the effects of prosodic information when processing relative clauses in Japanese. We focused on Accent Reduction and Initial Lowering as prosodic cues, and conducted two decision-making experiments with auditory stimuli to verify how the above cues affect the processing of relative clauses. Results from the experiments indicated that both Accent Reduction and Initial Lowering influence syntactic processing in Japanese. In addition, the results show that not only accented words but also unaccented words play a role in detecting left clause boundary. These findings might suggest that prosodic influences may be observed in unaccented word sequences even in silent readings.
REWIEW : Role of the CXC12-CXCR4 Axis and CXCL16 in Inflammatory Bowel Disease
( Hiroshi Nakase ),( Minoru Matsuura ),( Sakae Mikami ),( Norimitsu Uza ),( Tsutomu Chiba ) 대한장연구학회 2012 Intestinal Research Vol.10 No.2
Numerous studies of colitis in IBD (inflammatory bowel diseases) patients and in animal models have demonstrated that both inflammatory cytokines and chemokines are up-regulated in settings of active inflammation. Blockade or absence of various cytokines and chemokines attenuates the disease in murine models of IBD. Therefore, identifying cytokines and chemokines involved in intestinal inflammation provide promising targets for the development of new drugs in the treatment of IBD. In general, chemokines have been implicated in many fundamental immune processes including lymphoid organogenesis, immune cell differentiation, development and positioning. Many chemokines are markedly increased in intestinal tissue from patients with IBD. In this study, we focused on the role of CXCL12-CXCR4 and CXCL16. CXCL12-CXCR4 axis plays a crucial role in the pathophysiology of IBD, especially UC, while SR-PSOX/CXCL16 plays a significant role in the pathophysiology of CD. Our present data suggest new insights into the etiology of IBD and we hope that the manipulation of these chemokines may have therapeutic value. (Intest Res 2012;10:125-133)