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Kihoon Jeon,Sanghwa Chung,Donghwa Yoo 한국정보과학회 2020 Journal of Computing Science and Engineering Vol.14 No.2
Owing to the development of the Internet of Things (IoT) paradigm, the energy consumption of devices and the reliability of communication have become important issues. Enhanced TSCH technology introduces a technique to select high-quality channels by using energy detection in the TSCH protocol to improve the reliability of communication in a dynamic environment where interference changes. However, it is difficult to apply ETSCH technology to a multi-hop network because the node that performs energy detection consumes more energy than the node that does not. In this article, we propose an adaptive channel-quality estimation (ACE), which flexibly adjusts the duty cycle of energy detection according to whether interference dynamically changes or not. ACEs are generally applicable regardless of the degree of change of interference, which improves energy efficiency. Also, we present ACE-blacklisting based TSCH (ACEBTSCH) that uses ACE and local channel blacklisting to blacklist the wireless channel based on energy detection in a multi-hop network. Experimental results show that ACEB-TSCH has a performance improvement of 15.94% over TSCH and 8.59% over PDR-blacklisting based TSCH.
Effect of Exercise Intensity on Unfolded Protein Response in Skeletal Muscle of Rat
Kihoon Kim,Yun-Hye Kim,Sung-Hye Lee,Man-Joong Jeon,So-Young Park,Kyung-Oh Doh 대한생리학회-대한약리학회 2014 The Korean Journal of Physiology & Pharmacology Vol.18 No.3
Endoplasmic reticulum (ER) stress, unfolded protein response (UPR), and mitochondrial biogenesis were assessed following varying intensities of exercise training. The animals were randomly assigned to receive either low- (LIT, n=7) or high intensity training (HIT, n=7), or were assigned to a control group (n=7). Over 5 weeks, the animals in the LIT were exercised on a treadmill with a 10° incline for 60 min at a speed of 20 m/min group, and in the HIT group at a speed of 34 m/min for 5 days a week. No statistically significant differences were found in the body weight, plasma triglyceride, and total cholesterol levels across the three groups, but fasting glucose and insulin levels were significantly lower in the exercise-trained groups. Additionally, no statistically significant differences were observed in the levels of PERK phosphorylation in skeletal muscles between the three groups. However, compared to the control and LIT groups, the level of BiP was lower in the HIT group. Compared to the control group, the levels of ATF4 in skeletal muscles and CHOP were significantly lower in the HIT group. The HIT group also showed increased PGC-1α mRNA expression in comparison with the control group. Furthermore, both of the trained groups showed higher levels of mitochondrial UCP3 than the control group. In summary, we found that a 5-week high-intensity exercise training routine resulted in increased mitochondrial biogenesis and decreased ER stress and apoptotic signaling in the skeletal muscle tissue of rats.
Effect of Exercise Intensity on Unfolded Protein Response in Skeletal Muscle of Rat
Kim, Kihoon,Kim, Yun-Hye,Lee, Sung-Hye,Jeon, Man-Joong,Park, So-Young,Doh, Kyung-Oh The Korean Society of Pharmacology 2014 The Korean Journal of Physiology & Pharmacology Vol.18 No.3
Endoplasmic reticulum (ER) stress, unfolded protein response (UPR), and mitochondrial biogenesis were assessed following varying intensities of exercise training. The animals were randomly assigned to receive either low- (LIT, n=7) or high intensity training (HIT, n=7), or were assigned to a control group (n=7). Over 5 weeks, the animals in the LIT were exercised on a treadmill with a $10^{\circ}$ incline for 60 min at a speed of 20 m/min group, and in the HIT group at a speed of 34 m/min for 5 days a week. No statistically significant differences were found in the body weight, plasma triglyceride, and total cholesterol levels across the three groups, but fasting glucose and insulin levels were significantly lower in the exercise-trained groups. Additionally, no statistically significant differences were observed in the levels of PERK phosphorylation in skeletal muscles between the three groups. However, compared to the control and LIT groups, the level of BiP was lower in the HIT group. Compared to the control group, the levels of ATF4 in skeletal muscles and CHOP were significantly lower in the HIT group. The HIT group also showed increased PGC-$1{\alpha}$ mRNA expression in comparison with the control group. Furthermore, both of the trained groups showed higher levels of mitochondrial UCP3 than the control group. In summary, we found that a 5-week high-intensity exercise training routine resulted in increased mitochondrial biogenesis and decreased ER stress and apoptotic signaling in the skeletal muscle tissue of rats.