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- 저자 : Juan Solé (검색결과 3 건)
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Interest Rate Defenses of Currency Pegs
Juan Solé,김수용 한국국제경제학회 2011 International Economic Journal Vol.25 No.3
This paper studies a policy often used to defend currency pegs: raising short-term interest rates to stem demand for foreign reserves. Yet, this mechanism is absent from most monetary models. This paper develops a model with asset market frictions where this policy can be effective. The friction I emphasize is as in Lucas (199017. Lucas , R. E. 1990 . Liquidity and interest rates . Journal of Economic Theory , 50 : 237 – 264 . [CrossRef], [Web of Science ®]View all references): the need of liquidity for asset transactions. When the government raises domestic interest rates, agents increase their domestic currency holdings in order to acquire interest-bearing domestic assets, instead of increasing their demand for the central bank's reserves, and thus the peg survives. The model shows that, while interest rate defenses can be successful, they may impose great costs for domestic agents; hence governments’ reluctance to sustain this policy for long periods. Finally, the general equilibrium nature of the model allows computing the welfare cost of this policy.
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Activation of Neuronal Ensembles via Controlled Synchronization
Gualberto Solís-Perales,Juan Gonzalo Barajas-Ramírez 제어·로봇·시스템학회 2017 International Journal of Control, Automation, and Vol.15 No.1
In this contribution we present the activation of neuronal ensembles of Hindmarsh-Rose neurons bycontrolled synchronization. The main problem consists in to impose a particular spiking-bursting behavior in allthe neurons of the network. We consider a network where the neurons are in its resting state, it is desired that theneurons change their resting state to a particular behavior of activation, dictated by a neuron called the referenceneuron. The goal is reached by controlling some neurons in the network controlling only the membrane potential(electrical synapse). The key feature of the present contribution is that by controlling a small number of neuronsin the network a desired behavior is induced in all the neurons in the network despite its network topology. Theimportant parameters are the control gain and the coupling strength, thus the activation of the network lays downon a compromise between the control gain and the coupling strength.
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Homozygous LPL p.Gly188Glu Mutation in a Mexican Girl With Lipoprotein Lipase Deficiency
Ana Gabriela Colima Fausto,Juan Ramón González García,Teresita De Jesús Hernández Flores,Norma Alejandra Vázquez Cárdenas,Nery Eduardo Solís Perales,María Teresa Magaña Torres 대한진단검사의학회 2017 Annals of Laboratory Medicine Vol.37 No.4
Dear Editor, Lipoprotein lipase (LPL) deficiency is a rare disease characterized by severe hypertriglyceridemia due to mutations in the LPL gene. It shows an autosomal recessive inheritance pattern and has a general prevalence of 1:1,000,000 [1]. Patients typically present abdominal pain, hemorrhage, failure to thrive, jaundice, eruptive xanthomas, lipemia retinalis, neurological complications, hepatosplenomegaly, and life-threatening pancreatitis [2]. The LPL gene is located on chromosomal band 8p22, contains 10 exons, and codes for a 475-amino acid protein that is active only in its dimeric form. The LPL protein has two functions in lipid metabolism: it hydrolyzes triglycerides and functions as a ligand [3]. The aim of this work is to describe the biochemical, clinical, and molecular features of a Mexican girl with LPL deficiency.
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