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Fei Fan,Xunxin Wu,Jiaxiang Wen,Gengyu Lin,Jiawen Lin,Qiuting Yin 한국섬유공학회 2023 Fibers and polymers Vol.24 No.10
This paper explores the effects of a dielectric barrier discharge atmospheric pressure air plasma and citric acid (CA) pretreatment on dyeing gray mulberry silk plain fabrics with a cortex phellodendri plant powder dyestuff. The atmospheric air plasma treatment formed new hydrophilic groups, such as C-O, O-C=O, and C-NH2, on the surface of the fabric. The relative contents of the C-O (C-N) and O-C=O groups indicated that the efficiency of bonding the hydrophilic groups to the fabric increased in the order of citric acid alone < collaborative treatment in different orders < original < atmospheric air plasma alone. X-ray photoelectron spectroscopy (XPS) showed that the relative content of C-O (C-N)/C=O was the highest after 9 treatments at 360 W. Erosion pits and grooves with widths of 0.5-3 µm appeared on the surface after plasma treatment. The surface of the silk fabric was negatively charged, and it interacted electrostatically, adsorbed and combined with the cationic dye. The samples were dyed at 60 °C, with a pH 8, a 60% (owf) dye concentration, a bath ratio of 1:40, and a 60 min dyeing time. After soaping, the K/S value had increased from 8.05 to 13.51, and the washing color fastness rating was 4-5.
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Wang Kailing,Liu Fan,Muchu Budumu,Deng Jiawen,Peng Jing,Xu Yan,Li Fujun,Ouyang Miao 대한약학회 2024 Archives of Pharmacal Research Vol.47 No.7
SMARCA5, a protein in the SWI/SNF family, has been previously implicated in the development of ulcerative colitis (UC) through methylation. However, the specifi c molecular mechanisms by which SMARCA5 contributes to colonic infl ammation and the imbalance between Th17 and Treg cells remain unclear. This study was designed to explore these molecular mechanisms. A UC mouse model was established using dextran sulfate sodium induction, followed by measurements of mouse weight, disease activity index (DAI) score, colon length, pathological changes in the colon, and FITC-dextran concentration. The levels of IL-17a, IFN-γ, IL-6, TNF-α, TGF-β, and IL-10 were measured, along with the protein expression of ZO-1 and Occludin. Flow cytometry was used to assess the presence of IL-17 + CD4 + (Th17 +) cells and FOXP3 + CD25 + CD4 + (Treg +) cells in the spleen and mesenteric lymph nodes of UC mice. We observed that SMARCA5 and RNF180 were increased, while ALKBH5 was downregulated in UC mouse colon tissue. SMARCA5 or RNF180 knockdown or ALKBH5 overexpression ameliorated the colon infl ammation and Th17/Treg cell imbalance in UC mice, shown by increased body weight, colon length, FOXP3 + CD25 + CD4 + T cells, and the levels of ZO-1, Occludin, TGF-β, IL-10, and FOXP3. It decreased DAI scores, IL-17 + CD4 + T cells, and levels of IL-17a, IFN-γ, IL-6, TNF-α, and ROR-γt. ALKBH5 inhibited SMARCA5 expression via m6A modifi cation, while RNF180 reduced ALKBH5 expression via ubiquitination. Our fi ndings indicate that RNF180 aggravated the colon infl ammation and Th17/Treg cell imbalance in UC mice by regulating the ALKBH5/SMARCA5 axis.
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Rui Zhu,Xueqin Yang,Jianjun Zhang,Danwen Xu,Jiawen Fan,Huoying Shi,Shifeng Wang,Xiufan Liu 대한수의학회 2018 Journal of Veterinary Science Vol.19 No.3
The subtype H9N2 avian influenza virus greatly threatens the Chinese poultry industry, even with annual vaccination. Waterfowl can be asymptomatically infected with the H9N2 virus. In this study, three H9N2 virus strains, designated A/Goose/Jiangsu/YZ527/2011 (H9N2, Gs/JS/YZ527/11), A/Goose/Jiangsu/SQ119/2012 (H9N2, Gs/JS/SQ119/12), and A/Goose/Jiangsu/JD564/2012 (H9N2, Gs/JS/JD564/12), were isolated from domestic geese. Molecular characterization of the three isolates showed that the Gs/JS/YZ527/11 virus is a double-reassortant virus, combining genes of A/Quail/Hong Kong/G1/97 (H9N2, G1/97)-like and A/Chicken/Shanghai/F/98 (H9N2, F/98)-like; the Gs/JS/SQ119/12 virus is a triple-reassortant virus combining genes of G1/97-like, F/98-like, and A/Duck/Shantou/163/2004 (H9N2, ST/163/04)-like. The sequences of Gs/JS/JD564/12 share high homology with those of the F/98 virus, except for the neuraminidase gene, whereas the internal genes of Gs/JS/YZ527/11 and Gs/JS/SQ119/12 are closely related to those of the H7N9 viruses. An infectivity analysis of the three isolates showed that Gs/JS/SQ119/12 and Gs/JS/YZ527/11 replicated well, with seroconversion, in geese and chickens, the Gs/JS/JD564/12 did not infect well in geese or chickens, and the F/98 virus only infected chickens, with seroconversion. Emergence of these new reassortant H9N2 avian influenza viruses indicates that these viruses can infect both chicken and goose and can produce different types of lesions in each species.
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