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The development of Taiwan Fracture Liaison Service network
Lo-Yu Chang,Keh-Sung Tsai,Jen-Kuei Peng,Chung-Hwan Chen,Gau-Tyan Lin,Chin-Hsueh Lin,Shih-Te Tu,I-Chieh Mao,Yih-Lan Gau,Hsusan-Chih Liu,Chi-Chien Niu,Min-Hong Hsieh,Jui-Teng Chien,Wei-Chieh Hung,Rong-S 대한골다공증학회 2018 Osteoporosis and Sarcopenia Vol.4 No.2
Osteoporosis and its associated fragility fractures are becoming a severe burden in the healthcare system globally. In the Asian-Pacific (AP) region, the rapidly increasing in aging population is the main reason accounting for the burden. Moreover, the paucity of quality care for osteoporosis continues to be an ongoing challenge. The Fracture Liaison Service (FLS) is a program promoted by International Osteoporosis Foundation (IOF) with a goal to improve quality of postfracture care and prevention of secondary fractures. In this review article, we would like to introduce the Taiwan FLS network. The first 2 programs were initiated in 2014 at the National Taiwan University Hospital and its affiliated Bei-Hu branch. Since then, the Taiwan FLS program has continued to grow exponentially. Through FLS workshops promoted by the Taiwanese Osteoporosis Association (TOA), program mentors have been able to share their valuable knowledge and clinical experience in order to promote establishments of additional programs. With 22 FLS sites including 11 successfully accredited on the best practice map, Taiwan remains as one of the highest FLS coverage countries in the AP region, and was also granted the IOF Best Secondary Fracture Prevention Promotion award in 2017. Despite challenges faced by the TOA, we strive to promote more FLS sites in Taiwan with a main goal of ameliorating further health burden in managing osteoporotic patients.
A Group Decision Model for Selecting Facility Layout Alternatives
Lin, Shui-Shun,Chiou, Wen-Chih,Lee, Ron-Hua,Perng, Chyung,Tsai, Jen-Teng Korean Institute of Industrial Engineers 2005 Industrial Engineeering & Management Systems Vol.4 No.1
Facility layout problems (FLP) are usually treated as design problems. Lack of systematic and objective tools to compare design alternatives results in decision-making to be dominated by the experiences or preferences of designers or managers. To increase objectivity and effectiveness of decision-making in facility layout selections, a decision support model is necessary. We proposed a decision model, which regards the FLP as a multi-attribute decision making (MADM) problem. We identify sets of attributes crucial to layout selections, quantitative indices for attributes, and methods of ranking alternatives. For a requested facility layout design, many alternatives could be developed. The enormous alternatives, various attributes, and comparison of assigned qualitative values to each attribute, form a complicated decision problem. To treat facility layout selection problems as a MADM problem, we used the linear assignment method to rank before selecting those high ranks as candidates. We modelled the application of the Nemawashi process to simulate the group decision-making procedure and help efficiently achieve agreement. The electronics manufacturing service (EMS) industry has frequent and costly facility layout modifications. Our models are helpful to them. We use an electronics manufacturing service company to illustrate the decision-making process of our models.
Pen Hua Su,Jia Yuh Chen,Ju Shan Yu,Suh Jen Chen,Teng Fu Tsao,Shih Jei Tsai 한국유전학회 2008 Genes & Genomics Vol.30 No.4
Cleidocranial dysplasia (CCD, #119600) is a rare, autosomal dominant bone disease characterized by hypoplastic or aplastic clavicles, wide cranial sutures, supernumerary teeth, short stature, and other skeletal disorders. This disease gene has been mapped to chromosome 6p21 within a region containing CBFA1, a member of the runt family of transcription factors (RUNX2). We report one Taiwanese girl with CCD, with multiple wormian bones, persistent synchondrosis, supernumerary teeth, hypoplasia of clavicles, delayed ossification of pubic bone, and short stature. She also has partial fusion of the left 4th and 5th ribs, and posterior cerebral artery malformation. We performed sequence analysis of the RUNX2 gene, and detected a heterozygous C to G transition mutation at nucleotide 1115 in exon 7, leading to P372R mutation. This is an unreported missense mutation in exon 7 which effected the trausactivation domain of RUNX2.