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무선 인터넷 서비스를 위한 HTTP 기반의 응용 계층 보안 프로토콜
이동근(Donggeun Lee),김기조(Kijo Kim),임경식(Kyungshik Lim) 한국정보과학회 2003 정보과학회논문지 : 정보통신 Vol.30 No.3
현재 무선 인터넷에서 안전한 서비스를 제공하기 위하여 Secure HyperText Transfer Protocol(S-HTTP), Secure/Multipurpose Internet Mail Extensions(S/MIME), Secure Sockets Layer(SSL)/Transport Layer Security(TLS)와 Wireless TLS(WTLS) 등의 여러 가지 보안 프로토콜이 사용되고 있다. 그러나 S-HTTP와 S/MIME은 특정 응용에 한정적으로 사용 가능하며 SSL/TLS와 WTLS는 채널 보안으로 인하여 자원 낭비가 심할 뿐만 아니라 전자 서명 기능 또한 제공하지 못한다. 본 논문에서는 S-HTTP와 SSL/TLS의 장점을 수용하고 HTTP 기반에서 TLS 보안 메커니즘을 이용한 새로운 형태의 응용 계층 보안 프로토콜인 Application Layer Security(ALS)를 제안한다. ALS는 HTTP 기반에서 동작하므로 다양한 하부 전송망에 독립적이고, 보안을 필요로 하는 응용에 대하여 보안 인터페이스를 제공하는 방법을 통하여 특정 응용에 종속적이지 않는 특성을 가진다. 또한, TLS의 검증된 보안 메커니즘을 적용하여 안전성을 확보하였고, 인증, 기밀성, 무결성, 전자 서명 서비스 및 부분 암호화를 지원함으로써 응용에서 요구하는 다양한 서비스를 제공할 수 있다. 마지막으로 본 논문에서는 ALS를 이용한 Wireless Application Protocol의 단대단 보안 구현 내용을 기술한다. In this paper, we present an application layer protocol to support secure wireless Internet services, called Application Layer Security(ALS). The drawbacks of the two traditional approaches to secure wireless applications motivated the development of ALS. One is that in the conventional application-specific security protocol such as Secure HyperText Transfer Protocol(S-HTTP), security mechanism is included in the application itself. This gives a disadvantage that the security services are available only to that particular application. The other is that a separate protocol layer is inserted between the application and transport layers, as in the Secure Sockets Layer(SSL)/Transport Layer Security(TLS). In this case, all channel data are encrypted regardless of the specific application's requirements, resulting in much waste of network resources. To overcome these problems, ALS is proposed to be implemented on top of HTTP so that it is independent of the various transport layer protocols, and provides a common security interface with security applications so that it greatly improves the portability of security applications. In addition, since ALS takes advantages of well-known TLS mechanism, it eliminates the danger of malicious attack and provides applications with various security services such as authentication, confidentiality, integrity and digital signature, and partial encryption. We conclude this paper with an example of applying ALS to the solution of end-to-end security in a present commercial wireless protocol stack, Wireless Application Protocol.
KIM, Hyo-Shin,SUL, Donggeun,LIM, Ji-Youn,LEE, Dongho,JOO, Seong Soo,HWANG, Kwang Woo,PARK, So-Young Japan Society for Bioscience, Biotechnology, and A 2009 Bioscience, Biotechnology, and Biochemistry Vol.73 No.7
<P>Beta-amyloid (Aβ) has been suggested to induce neurotoxicity in Alzheimer’s disease. We evaluated the neuroprotective effects of delphinidin, an anthocyanidin commonly present in pigmented fruits and vegetables, against Aβ-induced toxicity. Aβ (25–35) significantly decreased the viability of PC12 cells, and this was accompanied by an increase in intracellular calcium levels and tau phosphorylation. However, treatment with delphinidin rescued PC12 cells from Aβ by attenuating the elevation of intracellular calcium levels and tau phosphorylation. Taken together, these results suggest that delphinidin protects PC12 cells against Aβ-induced toxicity by attenuating intracellular calcium influx and tau hyperphosphorylation.</P>
Lee, Jae Min,Lim, Ji-Youn,Kim, Yoonjin,Kim, Ye Ji,Choi, Hyuk Soon,Kim, Eun Sun,Keum, Bora,Seo, Yeon Seok,Jeen, Yoon Tae,Lee, Hong Sik,Um, Soon Ho,Kim, Chang Duck,Ryu, Ho Sang,Sul, Donggeun,Hong, Jungh D.A. Spandidos 2016 Experimental and therapeutic medicine Vol.12 No.2
<P>The present study investigated benexate hydrochloride betadex (BHB)-mediated ulcer healing, and changes to microcirculation modulated through nitric oxide synthase (NOS) and anti-inflammatory activity. A rat model of gastric mucosal injury was established through injection of a 60% acetic acid solution into the stomach. Following ulcer induction, the rats were administered BHB orally for 5 days at doses of 0, 100, 300 or 1,000 mg/kg. The highest dose of BHB was also administered with or without L-<I>NG</I>-nitroarginine methyl ester (L-NAME). The area of gastric ulcers was determined by planimetry, and expression of cyclooxygenases (COX), cytokines and NOS in stomach tissues were measured using western blotting. Compared with the control group, gastric ulcer size was significantly decreased in the 1,000 mg/kg BHB-treated group (P<0.05). Administration of BHB led to a significant increase in endothelial (e)NOS expression (P<0.05). Although acetic acid co-treatment with L-NAME induced more severe mucosal damage, BHB decreased COX expression and tumor necrosis factor-α levels when administered with the nitric oxide inhibitor, L-NAME (P<0.05). BHB exhibited protective effects in a rat model of gastric ulcers, which were associated with a decrease in pro-inflammatory cytokine levels and the activation of eNOS.</P>
Lee, Min,Kim, Byung-Jo,Lim, Eun Jeong,Back, Seung Keun,Lee, Ju-Han,Yu, Sung-Wook,Hong, Sung-Ha,Kim, Joo Han,Lee, Sang-Heon,Jung, Woon-Won,Sul, Donggeun,Na, Heung Sik Ovid Technologies (Wolters Kluwer) - AnesthesiaAna 2009 Anesthesia and analgesia Vol.109 No.4
<P>BACKGROUND: Although numerous animal models for low back pain associated with intervertebral disk (IVD) degeneration have been proposed, insufficient data have been provided to make any conclusions regarding pain. Our aim in this study was to determine the reliability of complete Freund's adjuvant (CFA) injection into the rat spine as an animal model representing human discogenic pain. METHODS: We studied IVD degenerative changes with pain development after a 10-microL CFA injection into the L5-6 IVD of adult rats using behavioral, histologic, and biochemical studies. Serial histologic changes were analyzed to detect degenerative changes. Expression of calcitonin gene-related peptide (CGRP), prostaglandin E (PGE), and inducible nitric oxide synthase (iNOS) were determined using immunohistochemistry or real-time polymerase chain reaction as support data for pain development. In addition, CGRP immunoreactivity (ir) at the IVD was considered indirect evidence of neural ingrowth into the IVD. RESULTS: There was a significant increase of the hindpaw withdrawal response in the CFA group until 7 wk postoperatively (P < 0.05). Histologic analyses revealed progressive degenerative changes of the disks without any damage in adjacent structures, including nerve roots. In the CGRP-ir staining study, the bilateral dorsal horns and IVD had positive ir after intradiscal CFA injection. CGRP mRNA expression was increased in the dorsal root ganglion (DRG) at 2 and 4 wk, whereas PGE and iNOS mRNAs were markedly increased at 2 wk. The increment of CGRP expression was higher in allodynic rats compared with nonallodynic rats. CONCLUSION: Intradiscal CFA injection led to chronic disk degeneration with allodynia, which was suggested by pain behavior and expression of pain-related mediators. The increment of CGRP, PGE, and iNOS also suggest pain-related signal processing between the IVD and the neural pathway in this animal model. This animal model may be useful for future research related to the pathophysiology and development of novel treatment for spine-related pain.</P>