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십이지장 궤양에 있어 혈청 Pepsinogen 1 치의 의의 - 궤양 발생의 유전적 또는 후천적 요인으로서의 의미 -
차수강(Su Gang Cha),김태호(Tae Ho Kim),이준성(June Sung Lee),김재준(Jae Jun Kim),김용태(Yong Tae Kim),윤용범(Yong Bum Yoon),송인성(In Sung Song),최규완(Kyoo Wan Choi),김정룡(Chung Yong Kim) 대한내과학회 1993 대한내과학회지 Vol.45 No.2
Background: Peptic ulcer is a heterogenous disease which has genetic or acquired factors in the development of the disease. Serum pepsinogen I (PG I) was reported as a marker of the genetic factors. Rut it is less reported about the relations between serum PG I and acquired factors in the development of the disease. The aim of this study was to evaluate serum PG I levels as the genetic or acquired factors in the development of duodenal ulcer. Methods: Fasting serum PG I levels were tested in 85 patients with duodenal ulcer, 64 their family members, and 52 normal controls. Results: 1) Serum PG I levels in duodenal ulcer patients (104±44ng/ml) and their family members (74±29ng/ml) were significantly higher than that of the control subjects (57±24ng/ml)(p<0.05). 2) The PG I levels of the family in their family members (78±30ng/ml) who had duodenal ulcer patient with elevated PGI levels in their family were somewhat higher than that of the family members (67±19ng/ml) who had the duodenal ulcer patients with normal PG I levels, but the difference did not acquire statistical significance (p<0.05). 3) The mean serum PG I level of smoker (111±47ng/ml) was higher than that of non-smoker (84±35ng/ml). 4) The mean serum PG I levels did not change in duodenal ulcer patients even after the treatment with H2-receptor blocker. Conclusion: These results suggested that serum PG I levels may be useful as a genetic marker in duodenal ulcer disease, but might be affected by acquired risk factor like smoking as well.
영웅석 ( Ung Suk Yang ),강대환 ( Dae Hwan Kang ),김상현 ( Sang Hyun Kim ),배상문 ( Sang Moon Bae ),차수강 ( Su Gang Cha ),종철수 ( Chul Soo Song ),송근암 ( Geun Am Song ),조몽 ( Mong Cho ) 대한췌장담도학회 1999 대한췌담도학회지 Vol.4 No.1
Endoscopic biilary drainage (EBD) for malignant obstructive jaundice is performed mostly with EST. But EST carries early complications such as bleeding, perforation and pancreatitis. We evaluated safety and usefulness of EBD using endoscopic papillary balloon dilation (EPBD) without endoscopic sphincterotomy (EST). Fifteen patients were treated from Sep. 1998 to Feb. 1999 in Pusan National University Hospital due to malignant obstructive jaundice. EBD was performed using EPBD. They were investigated to estimate the usefulness and early complications of EBD using EPBD. Effective drainage was achieved in 13 patients (87%). Procedure related complications developed in two patients; acute pancreatitis in two(minimal or mild, 13%), hemorrhage in three (clinically insignificant bleeding) EBD using EPBD is a safe and effective treatment method for malignant obstructive jaundice. However, prospective study is thought to be need in large patients group especially as to stent diameter and papilla size.