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Effect of Contact Pressure on Reciprocating Wear Behavior of PEEK, PTFE, and UHMWPE
Liu Lian,Duan Haitao,Jia Dan,Tu Jiesong,Zhan Shengpeng,Li Yinhua,Luo Xiaoshuang,Zhan Wen,Xiong Wei,Li Jian 한국고분자학회 2020 폴리머 Vol.44 No.6
Engineering plastics are macromolecular compounds composed of covalently bonded macromolecules, which have been widely used in sliding wear-resistance materials in isolation bearings. In this study, an MFT-5000 reciprocating friction testing machine was used to compare the friction and wear performance of polyetheretherketone (PEEK), polytetrafluoroethylene (PTFE), and ultra-high molecular weight polyethylene (UHMWPE) under heavy load conditions in dry friction condition. The results show that load has a significant effect on the friction coefficient, wear rate, and wear mechanism of three materials. The instant friction coefficient of PTFE fluctuates under high load, the wear rate clearly increases with the increase in load. Therefore, the application under high load conditions is limited. The wear rate of UHMWPE is the least affected by the load among the three materials. Even when the load exceeds the yield strength, the wear resistance is still good. The friction coefficient of PEEK decreases with the increase in load but maintains a high value that restricts its application in sliding friction pair materials to some degree.
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Hongming Li,Yumei Qiu,Mengdie Xie,Changsheng Ouyang,Xiaoyun Ding,Hao Zhang,Wei Dong,Yinhua Xiong,Xilan Tang 대한약리학회 2023 The Korean Journal of Physiology & Pharmacology Vol.27 No.1
This study aimed to observe the protective effect of momordicine I, a triterpenoid compound extracted from momordica charantia L., on isoproterenol (ISO)-induced hypertrophy in rat H9c2 cardiomyocytes and investigate its potential mechanism. Treatment with 10 μM ISO induced cardiomyocyte hypertrophy as evidenced by increased cell surface area and protein content as well as pronounced upregulation of fetal genes including atrial natriuretic peptide, β-myosin heavy chain, and α-skeletal actin; however, those responses were markedly attenuated by treatment with 12.5 μg/ml momordicine I. Transcriptome experiment results showed that there were 381 and 447 differentially expressed genes expressed in comparisons of model/control and momordicine I intervention/model, respectively. GO enrichment analysis suggested that the anti-cardiomyocyte hypertrophic effect of momordicine I may be mainly associated with the regulation of metabolic processes. Based on our transcriptome experiment results as well as literature reports, we selected glycerophospholipid metabolizing enzymes group VI phospholipase A2 (PLA2G6) and diacylglycerol kinase ζ (DGK-ζ) as targets to further explore the potential mechanism through which momordicine I inhibited ISO-induced cardiomyocyte hypertrophy. Our results demonstrated that momordicine I inhibited ISO-induced upregulations of mRNA levels and protein expressions of PLA2G6 and DGK-ζ. Collectively, momordicine I alleviated ISO-induced cardiomyocyte hypertrophy, which may be related to its inhibition of the expression of glycerophospholipid metabolizing enzymes PLA2G6 and DGK-ζ.
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