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- 저자 : Yamei Han (검색결과 3 건)
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Yiqiang HAN,Yamei GAO,Ying Shi,Jidao Du,Dianfeng Zheng,Guifeng Liu 한국식물학회 2017 Journal of Plant Biology Vol.60 No.4
Uniconazole, a plant growth retardant, possessesthe ability to improve quality and increase tolerance of plant. However, it is known little about the effects of uniconazoleon root. In this study, uniconazole treatments that were appliedthrough seed soaking, promoted soybean root development,and the microstructure of root showed increase of corticalthickness and cortex cell width. Meanwhile, the endogenoushormone content also altered in root after uniconazoletreatments. To obtain the molecular mechanism underlyingthe effects of uniconazole on root, we performed an RNAseqof roots harvested 3 days after uniconazole treatment. Through analyses of phytohormone-associated genes forendogenous hormones changes, we found that not only GAbiosynthesis pathway but also the regulation genes of thepathway were affected. Above all, the dominant pathwayplant hormone signal transduction may be the main factor ofthe cambium proliferation, in especial ethylene/ERF signalingpathway. Moreover, the transcriptome demonstrated differentiallyexpressed genes that determined cell division and cell wallmodification may be regulators of root growth. CLE signalingand receptor-like kinases may play a crucial role in the rootelongation. Besides, 177 transcription factors (TFs) wereinvolved in response to uniconazole. Taken together, allthese findings provide insights into the complex molecularmechanisms associated with root development after uniconazoletreatment.
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( Liaoyuan Wu ),( Yamei Wang ),( Jianghong Han ),( Wenqiang Chen ),( Lusheng Wang ) 한국인터넷정보학회 2016 KSII Transactions on Internet and Information Syst Vol.10 No.5
Successive interference cancellation (SIC) is considered to be a promising technique to mitigate multi-user interference and achieve concurrent uplink transmissions, but the optimal power allocation (PA) issue for SIC users is not well addressed. In this article, we focus on the optimization of the PA ratio of users on an SIC channel and analytically obtain the optimal PA ratio with regard to the signal-to-interference-plus-noise ratio (SINR) threshold for successful demodulation and the sustainable demodulation error rate. Then, we design an efficient resource allocation (RA) scheme using the obtained optimal PA ratio. Finally, we compare the proposal with the near-optimum RA obtained by a simulated annealing search and the RA scheme with random PA. Simulation results show that our proposal achieves a performance close to the near-optimum and much higher performance than the random scheme in terms of total utility and Jain`s fairness index. To demonstrate the applicability of our proposal, we also simulate the proposal in various network paradigms, including wireless local area network, body area network, and vehicular ad hoc network.
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Ze-Hua Zhao,Feng-Zhi Xin,Yaqian Xue,Zhimin Hu,Yamei Han,Fengguang Ma,Da Zhou,Xiao-Lin Liu,Aoyuan Cui,Zhengshuai Liu,Yuxiao Liu,Jing Gao,Qin Pan,Yu Li,Jian-Gao Fan 생화학분자생물학회 2019 Experimental and molecular medicine Vol.51 No.-
Microbial metabolites have emerged as critical components that mediate the metabolic effects of the gut microbiota. Here, we show that indole-3-propionic acid (IPA), a tryptophan metabolite produced by gut bacteria, is a potent anti-non-alcoholic steatohepatitis (NASH) microbial metabolite. Here, we demonstrate that administration of IPA modulates the microbiota composition in the gut and inhibits microbial dysbiosis in rats fed a high-fat diet. IPA induces the expression of tight junction proteins, such as ZO-1 and Occludin, and maintains intestinal epithelium homeostasis, leading to a reduction in plasma endotoxin levels. Interestingly, IPA inhibits NF-κB signaling and reduces the levels of proinflammatory cytokines, such as TNFα, IL-1β, and IL-6, in response to endotoxin in macrophages to repress hepatic inflammation and liver injury. Moreover, IPA is sufficient to inhibit the expression of fibrogenic and collagen genes and attenuate diet-induced NASH phenotypes. The beneficial effects of IPA on the liver are likely mediated through inhibiting the production of endotoxin in the gut. These findings suggest a protective role of IPA in the control of metabolism and uncover the gut microbiome and liver cross-talk in regulating the intestinal microenvironment and liver pathology via a novel dietary nutrient metabolite. IPA may provide a new therapeutic strategy for treating NASH.
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