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Pino Cidad,Eugenia González,Mónica Asencio,Jesús García 대한안과학회 2015 Korean Journal of Ophthalmology Vol.29 No.5
Purpose: To study the retinal pigment epithelium (RPE) and retinal alterations in chronic central serous chorioretinopathy treated with photodynamic therapy, and its correlation with functional parameters such as best-corrected visual acuity (BCVA) and contrast sensitivity (CS). Methods: Retrospective, noncomparative, consecutive evaluation by optical coherence tomography and its correlation with BCVA and CS in 31 eyes of 26 patients. Results: In all affected patients, 88.5% were male with a mean age of 42.9 years. The right eye was involved in 64.5% of cases, bilateral in 19% and 73.9% were hyperopic (spherical refraction between 0 and +5.0 diopters). Of these cases, 51.5% had peri-RPE abnormalities, 17.3% hyperreflective substances at RPE, 19.4% RPE atrophy, 55.3% foveolar atrophy, 3.1% pigment epithelial detachment, 5.2% subretinal fluid persistence, 8.3% fibrin deposits, 68.4% photoreceptor inner and outer segment line interruption and 31.1% external limiting membrane interruption. Conclusions: Time evolution and number of outbreaks were related to the decrease in foveal and chorodial thickness and in those with worse BCVA and CS. RPE abnormalities and atrophy were related to the age of onset of symptoms. Photoreceptor elongation has been correlated with poor BCVA and inner and outer segment line destructuring and interruption with poor CS.
Unilateral Punctate Keratitis Secondary to Wallenberg Syndrome
Pino Cidad,Ana Boto,Almudena Del Hierro,Maria Capote,Susana Noval,Amanda Garcia,Susana Santiago 대한안과학회 2014 Korean Journal of Ophthalmology Vol.28 No.3
We studied three patients who developed left unilateral punctate keratitis after suffering left-sidedWallenberg Syndrome. A complex evolution occurred in two of them. In all cases, neurophysiologicalstudies showed damage in the trigeminal sensory component at the bulbar level. Corneal involvementsecondary to Wallenberg syndrome is a rare cause of unilateral superficial punctate keratitis. The lossof corneal sensitivity caused by trigeminal neuropathy leads to epithelial erosions that are frequentlyunobserved by the patient, resulting in a high risk of corneal-ulcer development with the possibility ofsuperinfection. Neurophysiological studies can help to locate the anatomical level of damage at theophthalmic branch of the trigeminal nerve, confirming the suspected etiology of stroke, and demonstratingthat prior vascular involvement coincides with the location of trigeminal nerve damage. In some of thesepatients, oculofacial pain is a distinctive feature.