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從親密到冷淡: 朝鮮媒體中的中國形象分析 -以2009-2017年 ≪勞動新聞≫涉華報道爲例-
서옥란 ( Xu Yu-lan ),이상만 ( Lee Sang-man ) 한중사회과학학회 2018 한중사회과학연구 Vol.16 No.4
進入2018年習近平主席與金正恩委員長三次會晤, 中朝關系步入 “新型的中朝友好合作關系”, 也重申了中朝同爲社會主義國家雙邊關系戰略意義。中朝兩國從1949年建交以來關系時而緩和, 始終跌岩起伏。本硏究主要利用內容分析法, 考察金正日到金正恩執政時期朝鮮官方媒體 ≪勞動新聞≫中的中國形象認知, 旨在探究朝鮮媒體建構中國國家形象的深層邏輯。從2009年到2017年朝鮮媒體中的涉華報道從深層的、多角度的轉變爲淺顯的、簡短的;開始關注中國國內政治、社會事件以及災難性問題; “中朝友誼”、“中朝親善”等詞匯逐步減少等現象。深層分析朝鮮官方的中國形象認知變遷, 可歸納爲從親密到疏遠、以中國爲鑒到走自主的路、從沈默到公開對峙等特点。中國與朝鮮各自基于自己的國家和民族利益、爲應對薩德等朝鮮半島危机展開着多層次的交流。在沖突語境下, 基于各種變遷與轉變, 中國在實施睦隣政策時也應适當調整思路與措施, 一方面克制民族主義情緖, 避免造成局勢緊張的誤導性言行, 另一方面是主動突破机制困境, 積極打開幷參與朝鮮半島无核化解決的對話協商。第三, 國內輿論應注意對西方國家特別是韓國媒體的政治介入及其輿論影響, 擺脫美日韓的 “朝鮮敍述”而建立中國立場的 “朝鮮敍述”。 Three meetings between Xi Jinping and Kim Jong-un in the first half of 2018 mark China and DPRK entered new partnership relations and reiterate the strategic importance of such relations. China-DPRK relations have been volatile since their establishment in 1949. This research studied the changes of China’s images described by RodongSinmun in Kim Jong-il administration and Kim Jong-un administration respectively and studied the root reasons for such changes through content analysis. During 2009 and 2017, the changes were as following: news reports about China focused on negatives aspects of China, and a decrease of using words like “friendship” and “closeness”. The root reasons could be concluded as two countries relations changed from close to distant and from silent struggle to confrontation. Based on the changes, China should adjust its foreign policy towards DPRK. First, China should stop the spreading of nationalism in the country to avoid behavior that might worsen two countries’ relations. Second, China should initiate talks with DPRK to denuclearize Korean Peninsula. Third, Chinese media should stay away from ROK media’s “DPRK narratives” in public opinion and establish China’s own “DPRK narratives”.
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Lu, Feng-Bin,Chen, Da-Zhi,Chen, Lu,Hu, En-De,Wu, Jin-Lu,Li, Hui,Gong, Yue-Wen,Lin, Zhuo,Wang, Xiao-Dong,Li, Ji,Jin, Xiao-Ya,Xu, Lan-Man,Chen, Yong-Ping Korean Society for Molecular and Cellular Biology 2019 Molecules and cells Vol.42 No.12
MicroRNA-223-3p (miR-223-3p) is one of the potential microRNAs that have been shown to alleviate inflammatory responses in pre-clinical investigations and is highly encased in exosomes derived from bone mesenchymal stem cells (MSC-exosomes). MSC-exosomes are able to function as carriers to deliver microRNAs into cells. Autoimmune hepatitis is one of the challenging liver diseases with no effective treatment other than steroid hormones. Here, we examined whether MSC-exosomes can transfer miR-223-3p to treat autoimmune hepatitis in an experimental model. We found that MSC-exosomes were successfully incorporated with miR-223-3p and delivered miR-223-3p into macrophages. Moreover, there was no toxic effect of exosomes on the macrophages. Furthermore, treatments of either exosomes or exosomes with miR-223-3p successfully attenuated inflammatory responses in the liver of autoimmune hepatitis and inflammatory cytokine release in both the liver and macrophages. The mechanism may be related to the regulation of miR-223-3p level and STAT3 expression in the liver and macrophages. These results suggest that MSC-exosomes can be used to deliver miR-223-3p for the treatment of autoimmune hepatitis.
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Yong-Ping Chen,Feng-Bin Lu,Da-Zhi Chen,Lu Chen,En-De Hu,Jin-Lu Wu,Hui Li,Yue-Wen Gong,Zhuo Lin,Xiao-Dong Wang,Ji Li,Xiao-Ya Jin,Lan-Man Xu 한국분자세포생물학회 2019 Molecules and cells Vol.42 No.12
MicroRNA-223-3p (miR-223-3p) is one of the potential microRNAs that have been shown to alleviate inflammatory responses in pre-clinical investigations and is highly encased in exosomes derived from bone mesenchymal stem cells (MSC-exosomes). MSC-exosomes are able to function as carriers to deliver microRNAs into cells. Autoimmune hepatitis is one of the challenging liver diseases with no effective treatment other than steroid hormones. Here, we examined whether MSC-exosomes can transfer miR-223-3p to treat autoimmune hepatitis in an experimental model. We found that MSC-exosomes were successfully incorporated with miR-223-3p and delivered miR-223-3p into macrophages. Moreover, there was no toxic effect of exosomes on the macrophages. Furthermore, treatments of either exosomes or exosomes with miR-223-3p successfully attenuated inflammatory responses in the liver of autoimmune hepatitis and inflammatory cytokine release in both the liver and macrophages. The mechanism may be related to the regulation of miR-223-3p level and STAT3 expression in the liver and macrophages. These results suggest that MSC-exosomes can be used to deliver miR-223-3p for the treatment of autoimmune hepatitis.
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