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( Wang Kuner ),( Huang Yushun ) 성균관대학교 유교문화연구소 2011 儒敎文化硏究(中文版) Vol.0 No.15
本文是對中國詩學基本范疇“比興”與朱熹《詩集傳》的專題硏究, 然而意在儒學本身的本源觀念的澄淸。朱熹的詩學思想其實是儒學的一種形而上學建構, 這種精致的哲學建構將儒家形而上學推向了歷史的高峰, 但同時也蔽塞了孔子“比興”觀念對于儒學的本源意義。本文旨在通過對朱熹詩學的重新認識和批評, 還原儒家詩學的本眞面目, 從而回到孔子儒學的本源觀念。 The thesis is the research about the basic category of Chinese poetics-“analogy(比) and rise(兴)” and Zhu Xi’s Variorum of the Book of Songs. However, the gist of the thesis is to clear the original ideas of Confucianism itself. Zhu Xi’s thought of poetics is actually a construction of Confucian metaphysics, this exquisite construction has push the Confucian metaphysics to the climax of history, while on the other hand, blocking the original sense of “analogy and rise” of Confucius to Confucianism. The thesis aims at reduction of Confucian poetics, thus coming back to the original idea of Confucianism of Confucius, through recognizing and criticizing Zhu Xi’s poetics. [Article in Chinese]
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Progressive NKCC1-Dependent Neuronal Chloride Accumulation during Neonatal Seizures.
Dzhala, Volodymyr I,Kuchibhotla, Kishore V,Glykys, Joseph C,Kahle, Kristopher T,Swiercz, Waldemar B,Feng, Guoping,Kuner, Thomas,Augustine, George J,Bacskai, Brian J,Staley, Kevin J The Society 2010 The Journal of neuroscience Vol.30 No.35
<P>Seizures induce excitatory shifts in the reversal potential for GABA(A)-receptor-mediated responses, which may contribute to the intractability of electro-encephalographic seizures and preclude the efficacy of widely used GABAergic anticonvulsants such as phenobarbital. We now report that, in intact hippocampi prepared from neonatal rats and transgenic mice expressing Clomeleon, recurrent seizures progressively increase the intracellular chloride concentration ([Cl(-)](i)) assayed by Clomeleon imaging and invert the net effect of GABA(A) receptor activation from inhibition to excitation assayed by the frequency of action potentials and intracellular Ca(2+) transients. These changes correlate with increasing frequency of seizure-like events and reduction in phenobarbital efficacy. The Na(+)-K(+)-2Cl(-) (NKCC1) cotransporter blocker bumetanide inhibited seizure-induced neuronal Cl(-) accumulation and the consequent facilitation of recurrent seizures. Our results demonstrate a novel mechanism by which seizure activity leads to [Cl(-)](i) accumulation, thereby increasing the probability of subsequent seizures. This provides a potential mechanism for the early crescendo phase of neonatal seizures.</P>
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