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RISS 인기검색어
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- 저자 : Jiliang Lin (검색결과 3 건)
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Leg-Balancing Control of the DC-link Voltage for Modular Multilevel Converters
Du, Sixing,Liu, Jinjun,Lin, Jiliang The Korean Institute of Power Electronics 2012 JOURNAL OF POWER ELECTRONICS Vol.12 No.5
This paper applies carrier phase shifted pulse-width modulation (CPS-PWM) to transformerless modular multilevel converters (MMC) to improve the output spectrum. Because the MMC topology is characterized by the double-star connection of six legs consisting of cascaded modular chopper cells with floating capacitors, the balance control of the DC-link capacitor voltage is essential for safe operation. This paper presents a leg-balancing control strategy to achieve DC-link voltage balance under all operating conditions. This strategy based on circulating current decoupling control focused on DC-link balancing between the upper and lower legs in each phase pair by considering the six legs as three independent phase-pairs. Experiments are implemented on a 100-V 3-kVA downscaled prototype. The experimental results show that the proposed leg-balancing control is both effective and practical.
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Leg-Balancing Control of the DC-link Voltage for Modular Multilevel Converters
Sixing Du,Jinjun Liu,Jiliang Lin 전력전자학회 2012 JOURNAL OF POWER ELECTRONICS Vol.12 No.5
This paper applies carrier phase shifted pulse-width modulation (CPS-PWM) to transformerless modular multilevel converters (MMC) to improve the output spectrum. Because the MMC topology is characterized by the double-star connection of six legs consisting of cascaded modular chopper cells with floating capacitors, the balance control of the DC-link capacitor voltage is essential for safe operation. This paper presents a leg-balancing control strategy to achieve DC-link voltage balance under all operating conditions. This strategy based on circulating current decoupling control focused on DC-link balancing between the upper and lower legs in each phase pair by considering the six legs as three independent phase-pairs. Experiments are implemented on a 100-V 3-kVA downscaled prototype. The experimental results show that the proposed leg-balancing control is both effective and practical.
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Yi Li,Qing'an Zeng,Jiliang Qiu,Ting Pang,Fenglian Ye,Lin Huang,Xuexia Zhang 한국유방암학회 2020 Journal of breast cancer Vol.23 No.4
Purpose: Four and a half LIM protein 1 (FHL1) is involved in breast cancer (BC) development, but the regulatory mechanism involved remain unclear. In the present study, we examined the role of FHL1 in BC development. Methods: The expression of FHL1, miR-183-5p, and miR-96-5p in BC tissues was analyzed using StarBase analysis. FHL1 expression in BC tissues, a normal human breast epithelial cell line, and BC cell lines was detected using quantitative reverse transcription polymerase chain reaction (qRT-PCR). The relationship between FHL1 and miR-183-5p/miR-96-5p was analyzed via Pearson's rank correlation, TargetScan, and a dual-luciferase reporter assay. BT549 and MDA-MB-231 cells were transfected with either FHL1 and miR-183-5p mimics, or siFHL1 and a miR-183-5p inhibitor, respectively. The viability, colony number, migration, invasion, and tube length of BT549 and MDA-MB-231 cells were examined using cell counting kit-8, colony formation, wound-healing, Transwell, and tube formation assays, respectively. The levels of FHL1, vascular endothelial growth factor (VEGF), p53, E-cadherin, N-cadherin, and vimentin were quantified using western blotting and qRT-PCR. Results: FHL1 expression was downregulated in BC tissues and cells, whereas miR-183-5p and miR-96-5p were upregulated in BC tissues (negative correlation with FHL1 expression). FHL1 overexpression inhibited the viability, colony number, migration, and invasion of BC cells and the expression of VEGF, N-cadherin, and vimentin, and increased the expression of FHL1, p53, and E-cadherin in BT549 cells. Furthermore, a miR-183-5p mimic reversed these effects of FHL1 overexpression, whereas FHL1 silencing caused opposite results to those observed in MDA-MB-231 cells; however, this was reversed by a miR-183-5p inhibitor. Conclusion: Our study suggests that miR-183-5p promotes cell proliferation, metastasis, and angiogenesis by negatively regulating FHL1 in BC.
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