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The Great Power of Illusion: The Influence of European Union Membership on Poland`s Security
( Anna Antczak-barzan ) 한국국방연구원 2016 The Korean Journal of Defense Analysis Vol.28 No.3
The article investigates the extent to which the European Union has contributed to the enhancement of Poland``s security. It achieves this through the analysis of various instruments of the EU within the Common Foreign and Security Policy in order to determine how (or if) they influenced Poland``s security policy. After the accession to NATO and the EU, Poland could not find its next strategic objective and was gradually losing its position as a leader in the region. Without national interests clearly defined, it is difficult to benefit from the opportunities the EU is presenting to its members, even if these opportunities are limited. The greatest problem is going from one extremum to the other with the shift of power between the two main opposing political parties-Civic Platform and Law and Justice.
Jerzy Antczak,Mateusz Szczygiełda,Krystyna Prochaska 한국공업화학회 2019 Journal of Industrial and Engineering Chemistry Vol.77 No.-
In this study a pilot-scale setup was used in the nanofiltration (NF) process for recovery of succinic acidfrom model solutions and the actual post-fermentation broth left after the bioconversion of glycerol. Theeffects of composition and pH of feed solutions, initial concentration of components and the magnitudeof applied transmembrane pressure (TMP) on the separation efficiency, the value of the permeatefluxand retention ratio of individual components present in the feed solutions, were investigated. In order toestimate the mechanism of membrane layer fouling formation, the experimental data obtained werecompared to the Hermia model.
Inhibition of the prostaglandin-degrading enzyme 15-PGDH potentiates tissue regeneration
Zhang, Yongyou,Desai, Amar,Yang, Sung Yeun,Bae, Ki Beom,Antczak, Monika I.,Fink, Stephen P.,Tiwari, Shruti,Willis, Joseph E.,Williams, Noelle S.,Dawson, Dawn M.,Wald, David,Chen, Wei-Dong,Wang, Zhengh American Association for the Advancement of Scienc 2015 Science Vol.348 No.6240
<P><B>A shot in the arm for damaged tissue</B></P><P>Tissue damage can be caused by injury, disease, and even certain medical treatments. There is great interest in identifying drugs that accelerate tissue regeneration and recovery, especially drugs that might benefit multiple organ systems. Zhang <I>et al.</I> describe a compound with this desired activity, at least in mice (see the Perspective by FitzGerald). SW033291 promotes recovery of the hematopoietic system after bone marrow transplantation, prevents the development of ulcerative colitis in the intestine, and accelerates liver regeneration after hepatic surgery. It acts by inhibiting an enzyme that degrades prostaglandins, lipid signaling molecules that have been implicated in tissue stem cell maintenance.</P><P><I>Science</I>, this issue 10.1126/science.aaa2340; see also p. 1208</P><P>Agents that promote tissue regeneration could be beneficial in a variety of clinical settings, such as stimulating recovery of the hematopoietic system after bone marrow transplantation. Prostaglandin PGE2, a lipid signaling molecule that supports expansion of several types of tissue stem cells, is a candidate therapeutic target for promoting tissue regeneration in vivo. Here, we show that inhibition of 15-hydroxyprostaglandin dehydrogenase (15-PGDH), a prostaglandin-degrading enzyme, potentiates tissue regeneration in multiple organs in mice. In a chemical screen, we identify a small-molecule inhibitor of 15-PGDH (SW033291) that increases prostaglandin PGE2 levels in bone marrow and other tissues. SW033291 accelerates hematopoietic recovery in mice receiving a bone marrow transplant. The same compound also promotes tissue regeneration in mouse models of colon and liver injury. Tissues from 15-PGDH knockout mice demonstrate similar increased regenerative capacity. Thus, 15-PGDH inhibition may be a valuable therapeutic strategy for tissue regeneration in diverse clinical contexts.</P>